oleanolic acid has been researched along with Genetic Predisposition in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Honkura, Y; Katori, Y; Kawase, T; Matsuo, H; Mizutari, K; Morita, I; Motohashi, H; Murakami, S; Sakiyama, M; Shinomiya, N; Shiotani, A; Yamamoto, M | 1 |
Egner, PA; Kaneko, T; Kensler, TW; Mashimo, T; Morita, M; Taguchi, K; Takaku, M; Yamamoto, M | 1 |
Allaway, G; Bhattachayya, M; Biswal, SS; Jones, BM; Korman, B; Lord, G; Marangoni, RG; Tourtellotte, WG; Varga, J; Wei, J; Zhu, H | 1 |
3 other study(ies) available for oleanolic acid and Genetic Predisposition
Article | Year |
---|---|
NRF2 Is a Key Target for Prevention of Noise-Induced Hearing Loss by Reducing Oxidative Damage of Cochlea.
Topics: Animals; Cochlea; Disease Models, Animal; Disease Progression; Gene Expression Regulation; Genetic Predisposition to Disease; Glutathione; Hearing Loss, Noise-Induced; Imidazoles; Lipid Peroxidation; Mice; Mice, Knockout; NF-E2-Related Factor 2; Oleanolic Acid; Oxidative Stress; Polymorphism, Single Nucleotide; Promoter Regions, Genetic; Protective Agents; Reactive Oxygen Species | 2016 |
Generation of a New Model Rat: Nrf2 Knockout Rats Are Sensitive to Aflatoxin B1 Toxicity.
Topics: Aflatoxin B1; Animals; Chemical and Drug Induced Liver Injury; Disease Models, Animal; DNA Adducts; Gene Editing; Gene Expression Profiling; Genetic Predisposition to Disease; Imidazoles; Inactivation, Metabolic; Liver; Male; Mice, Inbred C57BL; Mice, Knockout; Mutation; NF-E2-Related Factor 2; Oleanolic Acid; Oligonucleotide Array Sequence Analysis; Phenotype; Rats, Inbred F344; Rats, Transgenic | 2016 |
Nrf2 exerts cell-autonomous antifibrotic effects: compromised function in systemic sclerosis and therapeutic rescue with a novel heterocyclic chalcone derivative.
Topics: Animals; Bleomycin; Cells, Cultured; Chalcones; Down-Regulation; Fibroblasts; Fibrosis; Gene Expression Regulation; Genetic Predisposition to Disease; Humans; Mice; Mice, Knockout; NF-E2-Related Factor 2; Oleanolic Acid; RNA, Messenger; Scleroderma, Systemic; Transforming Growth Factor beta | 2017 |