o,o-diethyl o-3,5,6-trichloro-2-pyridyl phosphate has been researched along with Encephalopathy, Toxic in 8 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 3 (37.50) | 29.6817 |
2010's | 4 (50.00) | 24.3611 |
2020's | 1 (12.50) | 2.80 |
Authors | Studies |
---|---|
Aschner, M; Colle, D; da Rocha, JBT; Dafré, AL; Farina, M; Leal, RB; Lopes, MW; Naime, AA; Suñol, C | 1 |
Lockridge, O; Schopfer, LM | 1 |
Liu, J; Pope, C | 1 |
Grigoryan, H; Lockridge, O | 1 |
Duysen, EG; Hansen, H; Jiang, W; Lockridge, O; Schopfer, LM; Shlyakhtenko, L | 1 |
Flaskos, J | 1 |
Costa, LG; Honegger, P; Monnet-Tschudi, F; Schilter, B; Zurich, MG | 1 |
Darvesh, S; Duysen, EG; Li, B; Lockridge, O | 1 |
1 review(s) available for o,o-diethyl o-3,5,6-trichloro-2-pyridyl phosphate and Encephalopathy, Toxic
Article | Year |
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The developmental neurotoxicity of organophosphorus insecticides: a direct role for the oxon metabolites.
Topics: Animals; Brain; Chlorpyrifos; Cholinesterase Inhibitors; Female; Humans; Insecticides; Neurotoxicity Syndromes; Organophosphorus Compounds; Pregnancy | 2012 |
7 other study(ies) available for o,o-diethyl o-3,5,6-trichloro-2-pyridyl phosphate and Encephalopathy, Toxic
Article | Year |
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Glutathione in Chlorpyrifos-and Chlorpyrifos-Oxon-Induced Toxicity: a Comparative Study Focused on Non-cholinergic Toxicity in HT22 Cells.
Topics: Acetylcholine; Acetylcholinesterase; Animals; Atropine; Cell Survival; Chlorpyrifos; Cholinesterase Inhibitors; Glutathione; Neurons; Neurotoxicity Syndromes | 2020 |
Chlorpyrifos oxon promotes tubulin aggregation via isopeptide cross-linking between diethoxyphospho-Lys and Glu or Asp: Implications for neurotoxicity.
Topics: Acetylcholinesterase; Amino Acid Sequence; Animals; Aspartic Acid; Chlorpyrifos; Cholinesterase Inhibitors; Cross-Linking Reagents; Crystallography, X-Ray; Glutamic Acid; Lysine; Models, Molecular; Neurotoxicity Syndromes; Peptides; Phosphorylation; Protein Aggregates; Swine; Tubulin; Tyrosine | 2018 |
The cannabinoid receptor antagonist AM251 increases paraoxon and chlorpyrifos oxon toxicity in rats.
Topics: Amidohydrolases; Analysis of Variance; Animals; Arachidonic Acids; Cannabinoid Receptor Agonists; Cannabinoid Receptor Antagonists; Chlorpyrifos; Cholinesterase Inhibitors; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Synergism; Endocannabinoids; Insecticides; Male; Monoacylglycerol Lipases; Neurotoxicity Syndromes; Paraoxon; Piperidines; Polyunsaturated Alkamides; Pyrazoles; Rats; Rats, Sprague-Dawley; Time Factors; Tritium | 2015 |
Nanoimages show disruption of tubulin polymerization by chlorpyrifos oxon: implications for neurotoxicity.
Topics: Animals; Cattle; Chlorpyrifos; Cholinesterase Inhibitors; Dose-Response Relationship, Drug; Guanosine Triphosphate; Microscopy, Atomic Force; Nanostructures; Neurotoxicity Syndromes; Protein Multimerization; Spectrometry, Mass, Electrospray Ionization; Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization; Tandem Mass Spectrometry; Tubulin; Tubulin Modulators; Tyrosine | 2009 |
Mice treated with chlorpyrifos or chlorpyrifos oxon have organophosphorylated tubulin in the brain and disrupted microtubule structures, suggesting a role for tubulin in neurotoxicity associated with exposure to organophosphorus agents.
Topics: Animals; Biotinylation; Body Temperature; Body Weight; Brain; Brain Chemistry; Chlorpyrifos; Cholinesterases; Female; Insecticides; Mass Spectrometry; Mice; Mice, Inbred Strains; Microtubule-Associated Proteins; Microtubules; Neurotoxicity Syndromes; Organophosphates; Peptide Mapping; Spectrometry, Mass, Electrospray Ionization; Tubulin; Tubulin Modulators | 2010 |
Involvement of glial cells in the neurotoxicity of parathion and chlorpyrifos.
Topics: Acetylcholinesterase; Animals; Astrocytes; Brain; Cells, Cultured; Chlorpyrifos; Choline O-Acetyltransferase; Cholinesterase Inhibitors; Glutamate Decarboxylase; Glutamate-Ammonia Ligase; Immunohistochemistry; Insecticides; L-Lactate Dehydrogenase; Neuroglia; Neurotoxicity Syndromes; Paraoxon; Parathion; Protein Binding; Rats | 2004 |
Sensitivity of butyrylcholinesterase knockout mice to (--)-huperzine A and donepezil suggests humans with butyrylcholinesterase deficiency may not tolerate these Alzheimer's disease drugs and indicates butyrylcholinesterase function in neurotransmission.
Topics: Acetylcholine; Acetylcholinesterase; Alkaloids; Alzheimer Disease; Animals; Butyrylcholinesterase; Chlorpyrifos; Cholinesterase Inhibitors; Disease Models, Animal; Donepezil; Dose-Response Relationship, Drug; Female; Humans; Indans; Male; Mice; Mice, Knockout; Neurotoxicity Syndromes; Piperidines; Sesquiterpenes; Synaptic Transmission | 2007 |