Page last updated: 2024-11-01

ns 1619 and Vascular Calcification

ns 1619 has been researched along with Vascular Calcification in 1 studies

NS 1619: structure given in first source
NS 1619 : A member of the class of benzimidazoles that is 1,3-dihydro-2H-benzimidazol-2-one in which the hydrogens at positions 1 and 5 are replaced are replaced by 2-hydroxy-5-(trifluoromethyl)phenyl and trifluoromethyl groups, respectively. It is an opener/activator of the large-conductance calcium-activated potassium channel (Bkca).

Vascular Calcification: Deposition of calcium into the blood vessel structures. Excessive calcification of the vessels are associated with ATHEROSCLEROTIC PLAQUES formation particularly after MYOCARDIAL INFARCTION (see MONCKEBERG MEDIAL CALCIFIC SCLEROSIS) and chronic kidney diseases which in turn increase VASCULAR STIFFNESS.

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's1 (100.00)2.80

Authors

AuthorsStudies
Ning, FL1
Tao, J1
Li, DD1
Tian, LL1
Wang, ML1
Reilly, S1
Liu, C1
Cai, H1
Xin, H1
Zhang, XM1

Other Studies

1 other study available for ns 1619 and Vascular Calcification

ArticleYear
Activating BK channels ameliorates vascular smooth muscle calcification through Akt signaling.
    Acta pharmacologica Sinica, 2022, Volume: 43, Issue:3

    Topics: Alkaline Phosphatase; Animals; Aorta, Thoracic; Benzimidazoles; Cholecalciferol; Disease Models, Ani

2022