ns 1619 has been researched along with Reperfusion Injury in 3 studies
NS 1619: structure given in first source
NS 1619 : A member of the class of benzimidazoles that is 1,3-dihydro-2H-benzimidazol-2-one in which the hydrogens at positions 1 and 5 are replaced are replaced by 2-hydroxy-5-(trifluoromethyl)phenyl and trifluoromethyl groups, respectively. It is an opener/activator of the large-conductance calcium-activated potassium channel (Bkca).
Reperfusion Injury: Adverse functional, metabolic, or structural changes in tissues that result from the restoration of blood flow to the tissue (REPERFUSION) following ISCHEMIA.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Wang, Q | 2 |
| Kalogeris, TJ | 1 |
| Wang, M | 2 |
| Jones, AW | 1 |
| Korthuis, RJ | 2 |
| Liu, Y | 1 |
| Kalogeris, T | 1 |
| Zuidema, MY | 1 |
| Dai, H | 1 |
| Davis, MJ | 1 |
| Hill, MA | 1 |
| Domoki, F | 1 |
3 other studies available for ns 1619 and Reperfusion Injury
| Article | Year |
|---|---|
Antecedent ethanol attenuates cerebral ischemia/reperfusion-induced leukocyte-endothelial adhesive interactions and delayed neuronal death: role of large conductance, Ca2+-activated K+ channels.
Topics: Animals; Benzimidazoles; Blood-Brain Barrier; Brain Ischemia; Cell Adhesion; Cell Death; Cerebrovasc | 2010 |
Hydrogen sulfide preconditioning or neutrophil depletion attenuates ischemia-reperfusion-induced mitochondrial dysfunction in rat small intestine.
Topics: Animals; Benzimidazoles; Cytochromes c; Hydrogen Sulfide; Intestinal Diseases; Intestine, Small; Isc | 2012 |
[Changes in the post-ischemic glutaminic vascular reactions in newborn swine].
Topics: Animals; Animals, Newborn; Benzimidazoles; Blood Vessels; Brain Ischemia; Cycloheximide; Potassium C | 2002 |