ns 1619 has been researched along with Insulin Resistance in 1 studies
NS 1619: structure given in first source
NS 1619 : A member of the class of benzimidazoles that is 1,3-dihydro-2H-benzimidazol-2-one in which the hydrogens at positions 1 and 5 are replaced are replaced by 2-hydroxy-5-(trifluoromethyl)phenyl and trifluoromethyl groups, respectively. It is an opener/activator of the large-conductance calcium-activated potassium channel (Bkca).
Insulin Resistance: Diminished effectiveness of INSULIN in lowering blood sugar levels: requiring the use of 200 units or more of insulin per day to prevent HYPERGLYCEMIA or KETOSIS.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Dymkowska, D | 1 |
| Drabarek, B | 1 |
| Jakubczyk, J | 1 |
| Wojciechowska, S | 1 |
| Zabłocki, K | 1 |
1 other study available for ns 1619 and Insulin Resistance
| Article | Year |
|---|---|
Potassium channel openers prevent palmitate-induced insulin resistance in C2C12 myotubes.
Topics: Animals; Benzimidazoles; Cell Line; Insulin Resistance; Ion Channel Gating; Membrane Potential, Mito | 2014 |