Page last updated: 2024-11-01

ns 1619 and Heart Disease, Ischemic

ns 1619 has been researched along with Heart Disease, Ischemic in 2 studies

NS 1619: structure given in first source
NS 1619 : A member of the class of benzimidazoles that is 1,3-dihydro-2H-benzimidazol-2-one in which the hydrogens at positions 1 and 5 are replaced are replaced by 2-hydroxy-5-(trifluoromethyl)phenyl and trifluoromethyl groups, respectively. It is an opener/activator of the large-conductance calcium-activated potassium channel (Bkca).

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (100.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Xu, W1
Liu, Y1
Wang, S1
McDonald, T1
Van Eyk, JE1
Sidor, A1
O'Rourke, B1
Wang, X1
Yin, C1
Xi, L1
Kukreja, RC1

Other Studies

2 other studies available for ns 1619 and Heart Disease, Ischemic

ArticleYear
Cytoprotective role of Ca2+- activated K+ channels in the cardiac inner mitochondrial membrane.
    Science (New York, N.Y.), 2002, Nov-01, Volume: 298, Issue:5595

    Topics: Adenosine Triphosphate; Animals; Benzimidazoles; Calcium; Charybdotoxin; Cytoprotection; Guinea Pigs

2002
Opening of Ca2+-activated K+ channels triggers early and delayed preconditioning against I/R injury independent of NOS in mice.
    American journal of physiology. Heart and circulatory physiology, 2004, Volume: 287, Issue:5

    Topics: Animals; Animals, Outbred Strains; Benzimidazoles; Cardiotonic Agents; Ischemic Preconditioning, Myo

2004