Page last updated: 2024-11-01

ns 1619 and Electron Transport Chain Deficiencies, Mitochondrial

ns 1619 has been researched along with Electron Transport Chain Deficiencies, Mitochondrial in 1 studies

NS 1619: structure given in first source
NS 1619 : A member of the class of benzimidazoles that is 1,3-dihydro-2H-benzimidazol-2-one in which the hydrogens at positions 1 and 5 are replaced are replaced by 2-hydroxy-5-(trifluoromethyl)phenyl and trifluoromethyl groups, respectively. It is an opener/activator of the large-conductance calcium-activated potassium channel (Bkca).

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Liu, Y1
Kalogeris, T1
Wang, M1
Zuidema, MY1
Wang, Q1
Dai, H1
Davis, MJ1
Hill, MA1
Korthuis, RJ1

Other Studies

1 other study available for ns 1619 and Electron Transport Chain Deficiencies, Mitochondrial

ArticleYear
Hydrogen sulfide preconditioning or neutrophil depletion attenuates ischemia-reperfusion-induced mitochondrial dysfunction in rat small intestine.
    American journal of physiology. Gastrointestinal and liver physiology, 2012, Jan-01, Volume: 302, Issue:1

    Topics: Animals; Benzimidazoles; Cytochromes c; Hydrogen Sulfide; Intestinal Diseases; Intestine, Small; Isc

2012