ns 1619 has been researched along with Diabetes Mellitus, Type 1 in 2 studies
NS 1619: structure given in first source
NS 1619 : A member of the class of benzimidazoles that is 1,3-dihydro-2H-benzimidazol-2-one in which the hydrogens at positions 1 and 5 are replaced are replaced by 2-hydroxy-5-(trifluoromethyl)phenyl and trifluoromethyl groups, respectively. It is an opener/activator of the large-conductance calcium-activated potassium channel (Bkca).
Diabetes Mellitus, Type 1: A subtype of DIABETES MELLITUS that is characterized by INSULIN deficiency. It is manifested by the sudden onset of severe HYPERGLYCEMIA, rapid progression to DIABETIC KETOACIDOSIS, and DEATH unless treated with insulin. The disease may occur at any age, but is most common in childhood or adolescence.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Vetri, F | 2 |
Qi, M | 1 |
Xu, H | 2 |
Oberholzer, J | 1 |
Paisansathan, C | 2 |
Pelligrino, DA | 1 |
2 other studies available for ns 1619 and Diabetes Mellitus, Type 1
Article | Year |
---|---|
Impairment of neurovascular coupling in Type 1 Diabetes Mellitus in rats is prevented by pancreatic islet transplantation and reversed by a semi-selective PKC inhibitor.
Topics: Animals; Arterioles; Benzimidazoles; Cardiovascular Agents; Diabetes Mellitus, Experimental; Diabete | 2017 |
Impairment of neurovascular coupling in type 1 diabetes mellitus in rats is linked to PKC modulation of BK(Ca) and Kir channels.
Topics: Animals; Arterioles; Benzimidazoles; Cerebrovascular Circulation; Diabetes Complications; Diabetes M | 2012 |