ns 1619 has been researched along with Acute Brain Injuries in 6 studies
NS 1619: structure given in first source
NS 1619 : A member of the class of benzimidazoles that is 1,3-dihydro-2H-benzimidazol-2-one in which the hydrogens at positions 1 and 5 are replaced are replaced by 2-hydroxy-5-(trifluoromethyl)phenyl and trifluoromethyl groups, respectively. It is an opener/activator of the large-conductance calcium-activated potassium channel (Bkca).
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 6 (100.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Armstead, WM | 6 |
Hecker, JG | 1 |
Salvucci, A | 1 |
6 other studies available for ns 1619 and Acute Brain Injuries
Article | Year |
---|---|
Heat shock protein modulation of KATP and KCa channel cerebrovasodilation after brain injury.
Topics: Animals; Animals, Newborn; Benzimidazoles; Blood Pressure; Brain Injuries; Calcitonin Gene-Related P | 2005 |
Vasopressin impairs K(ATP) and K(ca) channel function after brain injury.
Topics: Animals; Animals, Newborn; Benzimidazoles; Brain Injuries; Cerebral Arteries; Cerebral Veins; Cromak | 2000 |
Vasopressin-induced protein kinase C-dependent superoxide generation contributes to atp-sensitive potassium channel but not calcium-sensitive potassium channel function impairment after brain injury.
Topics: Adenosine Triphosphate; Animals; Animals, Newborn; Arteries; Benzimidazoles; Brain Injuries; Brain I | 2001 |
Vasopressin induced cyclooxygenase dependent superoxide generation contributes to K(+) channel function impairment after brain injury.
Topics: Animals; Animals, Newborn; Anti-Inflammatory Agents, Non-Steroidal; Arginine Vasopressin; Benzimidaz | 2001 |
Endothelin-Induced cyclooxygenase-dependent superoxide generation contributes to K+ channel functional impairment after brain injury.
Topics: Animals; Animals, Newborn; ATP-Binding Cassette Transporters; Benzimidazoles; Brain Injuries; Calcit | 2001 |
Protein tyrosine kinase and mitogen-activated protein kinase activation contribute to K(ATP) and K(ca) channel impairment after brain injury.
Topics: Animals; Animals, Newborn; Benzimidazoles; Brain; Brain Injuries; Calcitonin Gene-Related Peptide; C | 2002 |