Page last updated: 2024-11-01

nomifensine and Parkinson Disease, Secondary

nomifensine has been researched along with Parkinson Disease, Secondary in 5 studies

Nomifensine: An isoquinoline derivative that prevents dopamine reuptake into synaptosomes. The maleate was formerly used in the treatment of depression. It was withdrawn worldwide in 1986 due to the risk of acute hemolytic anemia with intravascular hemolysis resulting from its use. In some cases, renal failure also developed. (From Martindale, The Extra Pharmacopoeia, 30th ed, p266)
nomifensine : An N-methylated tetrahydroisoquinoline carrying phenyl and amino substituents at positions C-4 and C-8, respectively.

Parkinson Disease, Secondary: Conditions which feature clinical manifestations resembling primary Parkinson disease that are caused by a known or suspected condition. Examples include parkinsonism caused by vascular injury, drugs, trauma, toxin exposure, neoplasms, infections and degenerative or hereditary conditions. Clinical features may include bradykinesia, rigidity, parkinsonian gait, and masked facies. In general, tremor is less prominent in secondary parkinsonism than in the primary form. (From Joynt, Clinical Neurology, 1998, Ch38, pp39-42)

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19902 (40.00)18.7374
1990's2 (40.00)18.2507
2000's1 (20.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Sotnikova, TD1
Beaulieu, JM1
Barak, LS1
Wetsel, WC1
Caron, MG1
Gainetdinov, RR1
Sarre, S1
De Klippel, N1
Herregodts, P1
Ebinger, G1
Michotte, Y1
Tedroff, J1
Aquilonius, SM1
Laihinen, A1
Rinne, U1
Hartvig, P1
Anderson, J1
Lundqvist, H1
Haaparanta, M1
Solin, O1
Antoni, G1
Schultz, W2
Scarnati, E2
Sundström, E2
Romo, R1
Tsutsumi, T1
Jonsson, G1

Other Studies

5 other studies available for nomifensine and Parkinson Disease, Secondary

ArticleYear
Dopamine-independent locomotor actions of amphetamines in a novel acute mouse model of Parkinson disease.
    PLoS biology, 2005, Volume: 3, Issue:8

    Topics: alpha-Methyltyrosine; Amphetamines; Animals; Antiparkinson Agents; Basal Ganglia; Blepharoptosis; Di

2005
Biotransformation of locally applied L-dopa in the corpus striatum of the hemi-parkinsonian rat studied with microdialysis.
    Naunyn-Schmiedeberg's archives of pharmacology, 1994, Volume: 350, Issue:1

    Topics: 3,4-Dihydroxyphenylacetic Acid; Animals; Benserazide; Biotransformation; Corpus Striatum; Denervatio

1994
Striatal kinetics of [11C]-(+)-nomifensine and 6-[18F]fluoro-L-dopa in Parkinson's disease measured with positron emission tomography.
    Acta neurologica Scandinavica, 1990, Volume: 81, Issue:1

    Topics: Adult; Aged; Aged, 80 and over; Corpus Striatum; Dopamine; Female; Humans; Male; Middle Aged; Nomife

1990
Protection against 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced parkinsonism by the catecholamine uptake inhibitor nomifensine: behavioral analysis in monkeys with partial striatal dopamine depletions.
    Neuroscience, 1989, Volume: 31, Issue:1

    Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Animals; Catecholamines; Female; Macaca fascicularis;

1989
The catecholamine uptake blocker nomifensine protects against MPTP-induced parkinsonism in monkeys.
    Experimental brain research, 1986, Volume: 63, Issue:1

    Topics: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; 3,4-Dihydroxyphenylacetic Acid; Animals; Basal Ganglia

1986