Page last updated: 2024-10-31

nocodazole and Pancreatic Neoplasms

nocodazole has been researched along with Pancreatic Neoplasms in 3 studies

Pancreatic Neoplasms: Tumors or cancer of the PANCREAS. Depending on the types of ISLET CELLS present in the tumors, various hormones can be secreted: GLUCAGON from PANCREATIC ALPHA CELLS; INSULIN from PANCREATIC BETA CELLS; and SOMATOSTATIN from the SOMATOSTATIN-SECRETING CELLS. Most are malignant except the insulin-producing tumors (INSULINOMA).

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (66.67)18.2507
2000's0 (0.00)29.6817
2010's1 (33.33)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Parsels, LA1
Tanska, DM1
Parsels, JD1
Zabludoff, SD1
Cuneo, KC1
Lawrence, TS1
Maybaum, J1
Morgan, MA1
Farshori, PQ1
Goode, D1
Gansauge, S1
Nussler, AK1
Beger, HG1
Gansauge, F1

Other Studies

3 other studies available for nocodazole and Pancreatic Neoplasms

ArticleYear
Dissociation of gemcitabine chemosensitization by CHK1 inhibition from cell cycle checkpoint abrogation and aberrant mitotic entry.
    Cell cycle (Georgetown, Tex.), 2016, Volume: 15, Issue:5

    Topics: Antimetabolites, Antineoplastic; Cell Cycle Checkpoints; Cell Line, Tumor; Checkpoint Kinase 1; Cycl

2016
Effects of the microtubule depolymerizing and stabilizing agents Nocodazole and taxol on glucose-induced insulin secretion from hamster islet tumor (HIT) cells.
    Journal of submicroscopic cytology and pathology, 1994, Volume: 26, Issue:2

    Topics: Adenoma, Islet Cell; Animals; Colforsin; Cricetinae; Fluorescent Antibody Technique; Glucose; Insuli

1994
Nitric oxide-induced apoptosis in human pancreatic carcinoma cell lines is associated with a G1-arrest and an increase of the cyclin-dependent kinase inhibitor p21WAF1/CIP1.
    Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research, 1998, Volume: 9, Issue:8

    Topics: Apoptosis; Blotting, Western; Cyclin-Dependent Kinase Inhibitor p21; Cyclins; G1 Phase; Humans; Inte

1998