Page last updated: 2024-08-25

nobiletin and Cardiovascular Stroke

nobiletin has been researched along with Cardiovascular Stroke in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (66.67)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Gao, Z; Liang, Z; Liu, Z; Wu, X; Zeng, L; Zheng, D1
Liang, Z; Liu, Z; Qin, Y; Wu, X; Zeng, L; Zhang, G; Zheng, D; Zhu, X1
Liang, X; Lu, Y; Mao, Q; Wu, Y1

Other Studies

3 other study(ies) available for nobiletin and Cardiovascular Stroke

ArticleYear
Nobiletin ameliorates cardiac impairment and alleviates cardiac remodeling after acute myocardial infarction in rats via JNK regulation.
    Pharmacology research & perspectives, 2021, Volume: 9, Issue:2

    Topics: Animals; Apoptosis; Cardiotonic Agents; Cell Line; Disease Models, Animal; Dose-Response Relationship, Drug; Flavones; Humans; JNK Mitogen-Activated Protein Kinases; Male; MAP Kinase Signaling System; Myoblasts; Myocardial Infarction; Myocardium; Rats; Ventricular Remodeling

2021
Nobiletin attenuates adverse cardiac remodeling after acute myocardial infarction in rats via restoring autophagy flux.
    Biochemical and biophysical research communications, 2017, 10-14, Volume: 492, Issue:2

    Topics: Animals; Antioxidants; Autophagy; Cardiotonic Agents; Cell Line; Flavones; Glucose; Heart; Male; Myocardial Infarction; Myocardial Ischemia; Myocardium; Myocytes, Cardiac; Oxygen; Rats, Sprague-Dawley

2017
Nobiletin protects against myocardial injury and myocardial apoptosis following coronary microembolization via activating PI3K/Akt pathway in rats.
    Naunyn-Schmiedeberg's archives of pharmacology, 2019, Volume: 392, Issue:9

    Topics: Animals; Apoptosis; Cardiotonic Agents; Embolization, Therapeutic; Flavones; Heart; Male; Malondialdehyde; Myocardial Infarction; Myocardium; Oxidative Stress; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Rats, Sprague-Dawley; Signal Transduction; Superoxide Dismutase; Troponin C; Ventricular Function, Left

2019