nitrites has been researched along with Myocarditis in 4 studies
Nitrites: Salts of nitrous acid or compounds containing the group NO2-. The inorganic nitrites of the type MNO2 (where M=metal) are all insoluble, except the alkali nitrites. The organic nitrites may be isomeric, but not identical with the corresponding nitro compounds. (Grant & Hackh's Chemical Dictionary, 5th ed)
Myocarditis: Inflammatory processes of the muscular walls of the heart (MYOCARDIUM) which result in injury to the cardiac muscle cells (MYOCYTES, CARDIAC). Manifestations range from subclinical to sudden death (DEATH, SUDDEN). Myocarditis in association with cardiac dysfunction is classified as inflammatory CARDIOMYOPATHY usually caused by INFECTION, autoimmune diseases, or responses to toxic substances. Myocarditis is also a common cause of DILATED CARDIOMYOPATHY and other cardiomyopathies.
| Excerpt | Relevance | Reference |
|---|---|---|
| " The reduced bioavailability of endothelium-derived nitric oxide (NO) may play a role in endothelial vasodilator dysfunction and the structural changes that are characteristic of GCAD." | 5.33 | Dimethylarginine dimethylaminohydrolase overexpression suppresses graft coronary artery disease. ( Cooke, JP; Gunawan, F; Jacobi, J; Robbins, RC; Sydow, K; Tanaka, M; Tsao, PS, 2005) |
| " Our data showed that immunization with cardiac myosin combined with IFA led to inflammatory response in the rat myocardium." | 1.38 | [Dynamic of myocarditis development in rats after injection of cardiac myosine combined with IFA]. ( Chentsov, IuS; Gavrilova, SA; Morozova, MP; Pogodina, LS; Postnikov, AB; Zemtsova, LV, 2012) |
| " The reduced bioavailability of endothelium-derived nitric oxide (NO) may play a role in endothelial vasodilator dysfunction and the structural changes that are characteristic of GCAD." | 1.33 | Dimethylarginine dimethylaminohydrolase overexpression suppresses graft coronary artery disease. ( Cooke, JP; Gunawan, F; Jacobi, J; Robbins, RC; Sydow, K; Tanaka, M; Tsao, PS, 2005) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (25.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 2 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bongartz, LG | 1 |
| Joles, JA | 1 |
| Verhaar, MC | 1 |
| Cramer, MJ | 1 |
| Goldschmeding, R | 1 |
| Tilburgs, C | 1 |
| Gaillard, CA | 1 |
| Doevendans, PA | 1 |
| Braam, B | 1 |
| Morozova, MP | 1 |
| Gavrilova, SA | 1 |
| Zemtsova, LV | 1 |
| Pogodina, LS | 1 |
| Postnikov, AB | 1 |
| Chentsov, IuS | 1 |
| Tanaka, M | 1 |
| Sydow, K | 1 |
| Gunawan, F | 1 |
| Jacobi, J | 1 |
| Tsao, PS | 1 |
| Robbins, RC | 1 |
| Cooke, JP | 1 |
| Wang, WZ | 1 |
| Matsumori, A | 1 |
| Matoba, Y | 1 |
| Matsui, S | 1 |
| Sato, Y | 1 |
| Hirozane, T | 1 |
| Shioi, T | 1 |
| Sasayama, S | 1 |
4 other studies available for nitrites and Myocarditis
| Article | Year |
|---|---|
Subtotal nephrectomy plus coronary ligation leads to more pronounced damage in both organs than either nephrectomy or coronary ligation.
Topics: Animals; Blood Pressure; Cardio-Renal Syndrome; Coronary Stenosis; Disease Models, Animal; Gene Expr | 2012 |
[Dynamic of myocarditis development in rats after injection of cardiac myosine combined with IFA].
Topics: Animals; Apoptosis; Autoimmune Diseases; Cardiac Myosins; Cytokines; Freund's Adjuvant; Inflammation | 2012 |
Dimethylarginine dimethylaminohydrolase overexpression suppresses graft coronary artery disease.
Topics: Amidohydrolases; Animals; Arginine; Coronary Artery Disease; Cytokines; Enzyme Inhibitors; Female; H | 2005 |
Protective effects of Mu-Fang-Ji-Tang against myocardial injury in a murine model of congestive heart failure induced by viral myocarditis.
Topics: Animals; Body Weight; Cell Line; Disease Models, Animal; Drugs, Chinese Herbal; Heart Failure; Macro | 1998 |