nitrites has been researched along with Hyperemia in 27 studies
Nitrites: Salts of nitrous acid or compounds containing the group NO2-. The inorganic nitrites of the type MNO2 (where M=metal) are all insoluble, except the alkali nitrites. The organic nitrites may be isomeric, but not identical with the corresponding nitro compounds. (Grant & Hackh's Chemical Dictionary, 5th ed)
Hyperemia: The presence of an increased amount of blood in a body part or an organ leading to congestion or engorgement of blood vessels. Hyperemia can be due to increase of blood flow into the area (active or arterial), or due to obstruction of outflow of blood from the area (passive or venous).
| Excerpt | Relevance | Reference |
|---|---|---|
| " The aim of this study was to analyse the effects of a single session of resistance exercise on blood flow, reactive hyperemia, plasma nitrite, and plasma malondialdehyde in patients with peripheral artery disease." | 5.20 | A session of resistance exercise increases vasodilation in intermittent claudication patients. ( Brasileiro-Santos, M; Correia, M; Forjaz, CL; Lima, A; Miranda, A; Ritti-Dias, R; Santos, A; Silva, A; Sobral Filho, D, 2015) |
| " Forearm blood flow (FBF) during reactive hyperemia and after sublingual nitroglycerin (NTG) administration was measured by strain-gauge plethysmography." | 3.71 | Hormone replacement effects on endothelial function measured in the forearm resistance artery in normocholesterolemic and hypercholesterolemic postmenopausal women. ( Chayama, K; Higashi, Y; Kimura, M; Kodama, I; Nakagawa, K; Ohama, K; Sanada, M; Tsuda, M, 2002) |
| " The response of the resistance bed in the forearm after release of inflow occlusion (reactive hyperemia), to hand exercise, and to local heating and the response of the calf resistance vessels to arterial occlusion and intra-arterial sodium nitrite and phentolamine were studied in 23 patients with congestive heart failure and 21 normal subjects." | 3.64 | A comparison of the effects of vasodilator stimuli on peripheral resistance vessels in normal subjects and in patients with congestive heart failure. ( Braunwald, E; Mason, DT; Zelis, R, 1968) |
| "Blood withdrawal and reactive hyperemia index measurements were performed before and 10 min, 1 h, 2 h, and 4 h after transfusion." | 2.77 | Transfusion of stored autologous blood does not alter reactive hyperemia index in healthy volunteers. ( Berra, L; Bloch, KD; Coppadoro, A; Dzik, WH; Feelisch, M; Fernandez, BO; Lei, C; Lin, T; Sammy, FY; Spagnolli, E; Steinbicker, AU; Stowell, CP; Warren, HS; Yu, B; Zapol, WM, 2012) |
| "Moreover, reactive hyperemia evaluated by strain-gauge plethysmography after 6 months (1." | 2.70 | Low-dose doxazosin improved aortic stiffness and endothelial dysfunction as measured by noninvasive evaluation. ( Higaki, J; Jinno, T; Katsuya, T; Komai, N; Matsumoto, K; Moriguchi, A; Ogihara, T; Ohishi, M; Rakugi, H; Yanagitani, Y, 2002) |
| "We measured ischemic reactive hyperemia (IRH), plasma levels of soluble vascular cell adhesion molecule-1 (sVCAM-1), total nitrite, lipid peroxidation products (LPO), hydrogen peroxide (H2O2), superoxide dismutase (SOD) and glutathione peroxidase (GPx) plasma activities." | 1.39 | Oxidative stress is associated with the number of components of metabolic syndrome: LIPGENE study. ( Delgado-Lista, J; Fuentes, F; Lopez-Miranda, J; Marin, C; Peña-Orihuela, P; Perez-Jimenez, F; Perez-Martinez, P; Roche, HM; Tinahones, FJ; Tunez, I; Yubero-Serrano, EM, 2013) |
| "Both uncontrolled hypertension and type II diabetes mellitus, but not smoking, are associated with impaired vascular smooth-muscle reactivity induced by NO donors." | 1.32 | Uncontrolled hypertension, uncompensated type II diabetes, and smoking have different patterns of vascular dysfunction. ( Cittadino, M; Moreno, H; Sabha, M; Sousa, MG; Tácito, LH; Tanus-Santos, JE; Yugar-Toledo, JC, 2004) |
| "In reactive hyperemia in the hindlimb, the peak flow did not differ; however, both the repayment flow and the duration were significantly larger in pulsatile flow." | 1.31 | Pulsatile flow enhances endothelium-derived nitric oxide release in the peripheral vasculature. ( Nagano, I; Nakano, T; Okabe, H; Tominaga, R; Yasui, H, 2000) |
| "Portal hypertension is characterized by splanchnic hyperemia due to a reduction in mesenteric vascular resistance." | 1.29 | Increased endothelial nitric oxide synthase activity in the hyperemic vessels of portal hypertensive rats. ( Cahill, PA; Hodges, R; Redmond, EM; Sitzmann, JV; Zhang, S, 1996) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (7.41) | 18.7374 |
| 1990's | 2 (7.41) | 18.2507 |
| 2000's | 9 (33.33) | 29.6817 |
| 2010's | 13 (48.15) | 24.3611 |
| 2020's | 1 (3.70) | 2.80 |
| Authors | Studies |
|---|---|
| Malishevskaya, TN | 1 |
| Filippova, YE | 1 |
| Rodrigues-Krause, J | 1 |
| Krause, M | 1 |
| Rocha, IMGD | 1 |
| Umpierre, D | 1 |
| Fayh, APT | 1 |
| Nyberg, M | 1 |
| Al-Khazraji, BK | 1 |
| Mortensen, SP | 1 |
| Jackson, DN | 1 |
| Ellis, CG | 1 |
| Hellsten, Y | 1 |
| Yubero-Serrano, EM | 1 |
| Delgado-Lista, J | 1 |
| Peña-Orihuela, P | 1 |
| Perez-Martinez, P | 1 |
| Fuentes, F | 1 |
| Marin, C | 1 |
| Tunez, I | 1 |
| Tinahones, FJ | 1 |
| Perez-Jimenez, F | 1 |
| Roche, HM | 1 |
| Lopez-Miranda, J | 1 |
| Rassaf, T | 1 |
| Totzeck, M | 1 |
| Hendgen-Cotta, UB | 1 |
| Shiva, S | 1 |
| Heusch, G | 1 |
| Kelm, M | 1 |
| Lima, A | 1 |
| Ritti-Dias, R | 1 |
| Forjaz, CL | 1 |
| Correia, M | 1 |
| Miranda, A | 1 |
| Brasileiro-Santos, M | 1 |
| Santos, A | 1 |
| Sobral Filho, D | 1 |
| Silva, A | 1 |
| Pettit, AP | 1 |
| Kipen, H | 1 |
| Laumbach, R | 1 |
| Ohman-Strickland, P | 1 |
| Kelly-McNeill, K | 1 |
| Cepeda, C | 1 |
| Fan, ZH | 1 |
| Amorosa, L | 1 |
| Lubitz, S | 1 |
| Schneider, S | 1 |
| Gow, A | 1 |
| Piknova, B | 1 |
| Park, JW | 1 |
| Kwan Jeff Lam, K | 1 |
| Schechter, AN | 1 |
| Djurica, D | 1 |
| Holt, RR | 1 |
| Ren, J | 1 |
| Shindel, AW | 1 |
| Hackman, RM | 1 |
| Keen, CL | 1 |
| Lima, AHRA | 1 |
| Correia, MA | 1 |
| Soares, AHG | 1 |
| Farah, BQ | 1 |
| Forjaz, CLM | 1 |
| Silva, AS | 1 |
| Brasileiro-Santos, MS | 1 |
| Santos, AC | 1 |
| Ritti-Dias, RM | 1 |
| Schwarz, A | 1 |
| Modun, D | 1 |
| Heusser, K | 1 |
| Tank, J | 1 |
| Gutzki, FM | 1 |
| Mitschke, A | 1 |
| Jordan, J | 1 |
| Tsikas, D | 1 |
| Kapil, V | 1 |
| Milsom, AB | 1 |
| Okorie, M | 1 |
| Maleki-Toyserkani, S | 1 |
| Akram, F | 1 |
| Rehman, F | 1 |
| Arghandawi, S | 1 |
| Pearl, V | 1 |
| Benjamin, N | 1 |
| Loukogeorgakis, S | 1 |
| Macallister, R | 1 |
| Hobbs, AJ | 1 |
| Webb, AJ | 1 |
| Ahluwalia, A | 1 |
| García, RG | 1 |
| Zarruk, JG | 1 |
| Barrera, C | 1 |
| Pinzón, A | 1 |
| Trillos, E | 1 |
| Arenas, WD | 1 |
| Luengas, C | 1 |
| Tomaz, C | 1 |
| López-Jaramillo, P | 1 |
| Berra, L | 1 |
| Coppadoro, A | 1 |
| Yu, B | 1 |
| Lei, C | 1 |
| Spagnolli, E | 1 |
| Steinbicker, AU | 1 |
| Bloch, KD | 1 |
| Lin, T | 1 |
| Sammy, FY | 1 |
| Warren, HS | 1 |
| Fernandez, BO | 1 |
| Feelisch, M | 1 |
| Dzik, WH | 1 |
| Stowell, CP | 1 |
| Zapol, WM | 1 |
| Sanada, M | 2 |
| Higashi, Y | 2 |
| Nakagawa, K | 2 |
| Tsuda, M | 2 |
| Kodama, I | 2 |
| Kimura, M | 1 |
| Chayama, K | 1 |
| Ohama, K | 2 |
| Yugar-Toledo, JC | 1 |
| Tanus-Santos, JE | 1 |
| Sabha, M | 1 |
| Sousa, MG | 1 |
| Cittadino, M | 1 |
| Tácito, LH | 1 |
| Moreno, H | 1 |
| Bustamante, SA | 1 |
| Sanches, N | 1 |
| Crosier, J | 1 |
| Miranda, D | 1 |
| Colombo, G | 1 |
| Miller, MJ | 1 |
| Cahill, PA | 1 |
| Redmond, EM | 1 |
| Hodges, R | 1 |
| Zhang, S | 1 |
| Sitzmann, JV | 1 |
| Nakano, T | 1 |
| Tominaga, R | 1 |
| Nagano, I | 1 |
| Okabe, H | 1 |
| Yasui, H | 1 |
| Lind, L | 1 |
| Hall, J | 1 |
| Larsson, A | 1 |
| Annuk, M | 1 |
| Fellström, B | 1 |
| Lithell, H | 1 |
| Najarian, T | 2 |
| Marrache, AM | 1 |
| Dumont, I | 1 |
| Hardy, P | 2 |
| Beauchamp, MH | 2 |
| Hou, X | 2 |
| Peri, K | 1 |
| Gobeil, F | 2 |
| Varma, DR | 2 |
| Chemtob, S | 2 |
| Sasaki, S | 1 |
| Sakashita, T | 1 |
| Egan, CG | 1 |
| Lockhart, JC | 1 |
| Ferrell, WR | 1 |
| Day, SM | 1 |
| McLean, JS | 1 |
| Komai, N | 1 |
| Ohishi, M | 1 |
| Moriguchi, A | 1 |
| Yanagitani, Y | 1 |
| Jinno, T | 1 |
| Matsumoto, K | 1 |
| Katsuya, T | 1 |
| Rakugi, H | 1 |
| Higaki, J | 1 |
| Ogihara, T | 1 |
| Checchin, D | 1 |
| Abran, D | 1 |
| Bernier, SG | 1 |
| Quiniou, C | 1 |
| Uzoukwu, M | 1 |
| Sleight, SD | 1 |
| Zelis, R | 1 |
| Mason, DT | 1 |
| Braunwald, E | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| A Randomised Controlled Trial of the Effect of Remote Ischaemic Conditioning on Coronary Endothelial Function in Patients With Angina.[NCT02666235] | Phase 2 | 60 participants (Actual) | Interventional | 2011-07-31 | Completed | ||
| The Impact of Dietary Cocoa Flavanols on Cardio-pulmonary Exercise Capacity in Human.[NCT01259739] | 13 participants (Actual) | Interventional | 2010-11-30 | Completed | |||
| Investigation of the Sex Differences in Oral Microbiota and Their Effect on Circulating Nitrite Levels[NCT01583803] | 24 participants (Anticipated) | Observational | 2012-06-30 | Active, not recruiting | |||
| The Acute Effect of Beetroot Juice on Circulating Nitrate and Nitrite Levels and Blood Pressure in Normotensives and Hypertensives[NCT01236872] | 15 participants (Actual) | Interventional | 2009-12-31 | Completed | |||
| Red Cell Storage Duration Study[NCT00991341] | Phase 3 | 1,481 participants (Actual) | Interventional | 2010-01-31 | Completed | ||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
Subjects were randomized for RECESS no earlier than one calendar day before the planned date of surgery, and were followed for all-cause mortality until post-operative Day 28, death, or study withdrawal, whichever occurred first. In some cases the surgery was postponed after randomization had already occurred. If surgery did not occur within 30 days after randomization, the subject ended the study and was not considered evaluable. If surgery did occur within 30 days after randomization, and the subject received at least one RBC transfusion between randomization and 96 hours after the end of surgery, the subject was considered evaluable. Therefore, in a few evaluable subjects, post-operative Day 28 could be nearly two months after the date of randomization. The times in the time-to-event analysis started at randomization. (NCT00991341)
Timeframe: 28 days post-surgery
| Intervention | participants with event (Number) |
|---|---|
| Shorter-storage Red Blood Cell Units | 23 |
| Longer-storage Red Blood Cell Units | 29 |
(NCT00991341)
Timeframe: 48 hours post-operation through day 28, hospital discharge, or death, whichever occurs first
| Intervention | participants with event (Number) |
|---|---|
| Shorter-storage Red Blood Cell Units | 68 |
| Longer-storage Red Blood Cell Units | 80 |
(NCT00991341)
Timeframe: Through post-operative day 7, hospital discharge, or death, whichever occurs first
| Intervention | mg/dL (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 0.85 |
| Longer-storage Red Blood Cell Units | 1.49 |
The arterial lactate levels were adjusted to make them comparable to venous lactate levels. (NCT00991341)
Timeframe: Through post-operative day 7, hospital discharge, or death, whichever occurs first
| Intervention | mmol/L (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 2.30 |
| Longer-storage Red Blood Cell Units | 2.92 |
The follow-up MODS used to calculate 28-day ΔMODS from pre-op baseline was based on the worst value of each component of MODS observed through post-op day 28, hospital discharge, or death, whichever occurred first, even if a subject's worst values for different components occurred on different dates. Subjects who died during this time period were assigned the worst possible follow-up MODS score, 24 points, and each component of MODS was set at 4, which is the worst score. If a subject did not die during this time period but had at least one day where the Glasgow Coma Score couldn't be scored[subject sedated; neurologic function not normal by pre-op history (prior stroke, tumor or trauma sequelae, cognitively challenged, behavioral disorder, etc.) or intra-op history, but currently unable to assess because of sedation], then a post-op MODS score was set to missing and a 28-day ΔMODS was not computed. The total MODS score ranges from 0 (best possible) to 24 points (worst possible). (NCT00991341)
Timeframe: Through 28 days post-surgery, hospital discharge, or death, whichever occurs first
| Intervention | MOD score points (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 8.74 |
| Longer-storage Red Blood Cell Units | 9.07 |
(NCT00991341)
Timeframe: Through post-operative day 7, hospital discharge, or death, whichever occurs first
| Intervention | mg/dL (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 0.35 |
| Longer-storage Red Blood Cell Units | 0.35 |
(NCT00991341)
Timeframe: Through post-operative day 7, hospital discharge, or death, whichever occurs first
| Intervention | ng/mL (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 15.82 |
| Longer-storage Red Blood Cell Units | 14.06 |
(NCT00991341)
Timeframe: Through post-operative day 7, hospital discharge, or death, whichever occurs first
| Intervention | participants with event (Number) |
|---|---|
| Shorter-storage Red Blood Cell Units | 206 |
| Longer-storage Red Blood Cell Units | 230 |
(NCT00991341)
Timeframe: Through post-operative day 7, hospital discharge, or death, whichever occurs first
| Intervention | participants with event (Number) |
|---|---|
| Shorter-storage Red Blood Cell Units | 91 |
| Longer-storage Red Blood Cell Units | 87 |
(NCT00991341)
Timeframe: Through post-operative day 7, hospital discharge, or death, whichever occurs first
| Intervention | participants with event (Number) |
|---|---|
| Shorter-storage Red Blood Cell Units | 62 |
| Longer-storage Red Blood Cell Units | 75 |
(NCT00991341)
Timeframe: Through post-op day 28
| Intervention | days (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 25.38 |
| Longer-storage Red Blood Cell Units | 25.17 |
Subjects were randomized for RECESS no earlier than one calendar day before the planned date of surgery, and were followed until post-operative Day 28, death, or study withdrawal, whichever occurred first. In some cases the surgery was postponed after randomization had already occurred. If surgery did not occur within 30 days after randomization, the subject ended the study and was not considered evaluable. If surgery did occur within 30 days after randomization, and the subject received at least one RBC transfusion between randomization and 96 hours after the end of surgery, the subject was considered evaluable. Therefore, in a few evaluable subjects, post-operative Day 28 could be nearly two months after the date of randomization. The times in the time-to-event analyses are from randomization to first post-operative bowel movement. (NCT00991341)
Timeframe: Through post-operative day 28, hospital discharge, or death, whichever occurs first
| Intervention | days (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 5.89 |
| Longer-storage Red Blood Cell Units | 6.62 |
Subjects were randomized for RECESS no earlier than one calendar day before the planned date of surgery, and were followed until post-operative Day 28, death, or study withdrawal, whichever occurred first. In some cases the surgery was postponed after randomization had already occurred. If surgery did not occur within 30 days after randomization, the subject ended the study and was not considered evaluable. If surgery did occur within 30 days after randomization, and the subject received at least one RBC transfusion between randomization and 96 hours after the end of surgery, the subject was considered evaluable. Therefore, in a few evaluable subjects, post-operative Day 28 could be nearly two months after the date of randomization. The times in the time-to-event analyses are from randomization to first post-operative solid food. (NCT00991341)
Timeframe: Through post-operative day 28, hospital discharge, or death, whichever occurs first
| Intervention | days (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 5.73 |
| Longer-storage Red Blood Cell Units | 6.18 |
The follow-up MODS used to calculate 7-day ΔMODS from pre-op baseline was based on the worst value of each component of MODS observed through post-op day 7, hospital discharge, or death, whichever occurred first, even if a subject's worst values for different components occurred on different dates. Subjects who died during this time period were assigned the worst possible follow-up MODS score, 24 points, and each component of MODS was set at 4, which is the worst score. If a subject did not die during this time period but had at least one day where the Glasgow Coma Score couldn't be scored [subject sedated; neurologic function not normal by pre-op history (prior stroke, tumor or trauma sequelae, cognitively challenged, behavioral disorder, etc.) or intra-op history, but currently unable to assess because of sedation], then a post-op MODS score was set to missing and a 7-day ΔMODS was not computed. The total MODS score ranges from 0 (best possible) to 24 points (worst possible). (NCT00991341)
Timeframe: Through post-operative day 7, hospital discharge, or death, whichever occurs first
| Intervention | MOD score points (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 8.49 |
| Longer-storage Red Blood Cell Units | 8.66 |
Because some subjects may experience multiple periods of ventilator use, the total duration that they were on a ventilator was compared between the two groups. (NCT00991341)
Timeframe: Through post-operative day 28, hospital discharge, or death, whichever occurs first
| Intervention | days (Mean) |
|---|---|
| Shorter-storage Red Blood Cell Units | 2.7 |
| Longer-storage Red Blood Cell Units | 2.8 |
1 review available for nitrites and Hyperemia
| Article | Year |
|---|---|
Association of l-Arginine Supplementation with Markers of Endothelial Function in Patients with Cardiovascular or Metabolic Disorders: A Systematic Review and Meta-Analysis.
Topics: Adult; Aged; Arginine; Cardiovascular Diseases; Diabetes Mellitus; Dietary Supplements; Endothelium, | 2018 |
9 trials available for nitrites and Hyperemia
| Article | Year |
|---|---|
Circulating nitrite contributes to cardioprotection by remote ischemic preconditioning.
Topics: Animals; Forearm; Hindlimb; Humans; Hyperemia; Ischemic Preconditioning; Ischemic Preconditioning, M | 2014 |
Circulating nitrite contributes to cardioprotection by remote ischemic preconditioning.
Topics: Animals; Forearm; Hindlimb; Humans; Hyperemia; Ischemic Preconditioning; Ischemic Preconditioning, M | 2014 |
Circulating nitrite contributes to cardioprotection by remote ischemic preconditioning.
Topics: Animals; Forearm; Hindlimb; Humans; Hyperemia; Ischemic Preconditioning; Ischemic Preconditioning, M | 2014 |
Circulating nitrite contributes to cardioprotection by remote ischemic preconditioning.
Topics: Animals; Forearm; Hindlimb; Humans; Hyperemia; Ischemic Preconditioning; Ischemic Preconditioning, M | 2014 |
A session of resistance exercise increases vasodilation in intermittent claudication patients.
Topics: Aged; Endothelium, Vascular; Female; Humans; Hyperemia; Hypertension; Intermittent Claudication; Leg | 2015 |
Effects of a dietary strawberry powder on parameters of vascular health in adolescent males.
Topics: Adolescent; Beverages; Biomarkers; Cardiovascular Diseases; Cross-Over Studies; Diet; Double-Blind M | 2016 |
Acute effects of walking and combined exercise on oxidative stress and vascular function in peripheral artery disease.
Topics: Aged; Ankle Brachial Index; Biomarkers; Blood Pressure; Female; Hemodynamics; Humans; Hyperemia; Int | 2018 |
Inorganic nitrate supplementation lowers blood pressure in humans: role for nitrite-derived NO.
Topics: Blood Pressure; Brachial Artery; Cardiovascular Diseases; Cross-Over Studies; Cyclic GMP; Double-Bli | 2010 |
Inorganic nitrate supplementation lowers blood pressure in humans: role for nitrite-derived NO.
Topics: Blood Pressure; Brachial Artery; Cardiovascular Diseases; Cross-Over Studies; Cyclic GMP; Double-Bli | 2010 |
Inorganic nitrate supplementation lowers blood pressure in humans: role for nitrite-derived NO.
Topics: Blood Pressure; Brachial Artery; Cardiovascular Diseases; Cross-Over Studies; Cyclic GMP; Double-Bli | 2010 |
Inorganic nitrate supplementation lowers blood pressure in humans: role for nitrite-derived NO.
Topics: Blood Pressure; Brachial Artery; Cardiovascular Diseases; Cross-Over Studies; Cyclic GMP; Double-Bli | 2010 |
Transfusion of stored autologous blood does not alter reactive hyperemia index in healthy volunteers.
Topics: Adult; Blood Preservation; Blood Transfusion, Autologous; Cross-Over Studies; Female; Hemolysis; Hum | 2012 |
Evaluation of endothelium-dependent vasodilation in the human peripheral circulation.
Topics: Adult; Aged; Brachial Artery; Endothelium, Vascular; Female; Forearm; Humans; Hyperemia; Male; Metha | 2000 |
Estrogen replacement therapy in postmenopausal women augments reactive hyperemia in the forearm by reducing angiotensin converting enzyme activity.
Topics: Blood Flow Velocity; Endothelium, Vascular; Estrogen Replacement Therapy; Estrogens, Conjugated (USP | 2001 |
Low-dose doxazosin improved aortic stiffness and endothelial dysfunction as measured by noninvasive evaluation.
Topics: Adrenergic alpha-Antagonists; Aorta; Arteriosclerosis; Doxazosin; Elasticity; Endothelium, Vascular; | 2002 |
17 other studies available for nitrites and Hyperemia
| Article | Year |
|---|---|
[The effect of antioxidant therapy on some pathogenetic factors of primary open-angle glaucoma].
Topics: Aged; Antioxidants; Cholesterol; Endothelin-1; Glaucoma, Open-Angle; Humans; Hyperemia; Nitrites | 2023 |
Effect of extraluminal ATP application on vascular tone and blood flow in skeletal muscle: implications for exercise hyperemia.
Topics: 6-Ketoprostaglandin F1 alpha; Adenosine Triphosphate; Adult; Animals; Blood Flow Velocity; Cells, Cu | 2013 |
Oxidative stress is associated with the number of components of metabolic syndrome: LIPGENE study.
Topics: Aged; Anthropometry; Antioxidants; Biomarkers; Blood Pressure; Endothelium, Vascular; Female; Glutat | 2013 |
Disrupted Nitric Oxide Metabolism from Type II Diabetes and Acute Exposure to Particulate Air Pollution.
Topics: Aged; Air Filters; Air Pollution; Diabetes Mellitus, Type 2; Endothelium, Vascular; Female; Humans; | 2015 |
Nitrate as a source of nitrite and nitric oxide during exercise hyperemia in rat skeletal muscle.
Topics: Animals; Hyperemia; Male; Muscle, Skeletal; Nitrates; Nitric Oxide; Nitrites; Physical Conditioning, | 2016 |
Stable-isotope dilution GC-MS approach for nitrite quantification in human whole blood, erythrocytes, and plasma using pentafluorobenzyl bromide derivatization: nitrite distribution in human blood.
Topics: Erythrocytes; Fluorobenzenes; Gas Chromatography-Mass Spectrometry; Humans; Hyperemia; Isotope Label | 2011 |
Plasma nitrate levels and flow-mediated vasodilation in untreated major depression.
Topics: Adolescent; Adult; Blood Flow Velocity; Brachial Artery; Cardiovascular Diseases; Case-Control Studi | 2011 |
Hormone replacement effects on endothelial function measured in the forearm resistance artery in normocholesterolemic and hypercholesterolemic postmenopausal women.
Topics: Arteries; Blood Flow Velocity; Cholesterol, HDL; Cholesterol, LDL; Endothelium, Vascular; Estradiol; | 2002 |
Uncontrolled hypertension, uncompensated type II diabetes, and smoking have different patterns of vascular dysfunction.
Topics: Adult; Blood Flow Velocity; Cyclic GMP; Diabetes Mellitus, Type 2; Endothelium, Vascular; Humans; Hy | 2004 |
Dietary nucleotides: effects on the gastrointestinal system in swine.
Topics: Adenosine; Animals; Animals, Newborn; Animals, Suckling; Diet; Digestive System; DNA; Granulocytes; | 1994 |
Increased endothelial nitric oxide synthase activity in the hyperemic vessels of portal hypertensive rats.
Topics: Animals; Blotting, Western; Cyclic GMP; Endothelium, Vascular; Enzyme Induction; Humans; Hyperemia; | 1996 |
Pulsatile flow enhances endothelium-derived nitric oxide release in the peripheral vasculature.
Topics: Angiotensin II; Animals; Blood Pressure; Calcium-Calmodulin-Dependent Protein Kinases; Calmodulin; C | 2000 |
Prolonged hypercapnia-evoked cerebral hyperemia via K(+) channel- and prostaglandin E(2)-dependent endothelial nitric oxide synthase induction.
Topics: Acidosis; Animals; Calcium Signaling; Carbon Dioxide; Dinoprostone; Disease Models, Animal; Dose-Res | 2000 |
Pathophysiological basis of acute inflammatory hyperaemia in the rat knee: roles of cyclo-oxygenase-1 and -2.
Topics: Animals; Arthritis, Experimental; Carrageenan; Cyclooxygenase 1; Cyclooxygenase 2; Dinoprostone; Ede | 2002 |
PGE(2)-mediated eNOS induction in prolonged hypercapnia.
Topics: Animals; Blood Flow Velocity; Calcium; Calcium Channel Blockers; Cyclooxygenase Inhibitors; Dactinom | 2002 |
Lactic and succinic dehydrogenase activity in nitrite toxicosis in the guinea pig.
Topics: Animals; Appetite Depressants; Body Weight; Guinea Pigs; Hemoglobins; Hyperemia; Hypoxia; Kidney; L- | 1970 |
A comparison of the effects of vasodilator stimuli on peripheral resistance vessels in normal subjects and in patients with congestive heart failure.
Topics: Adult; Forearm; Heart Failure; Hot Temperature; Humans; Hyperemia; Leg; Middle Aged; Nitrites; Phent | 1968 |