nitrates has been researched along with Hyperplasia in 37 studies
Nitrates: Inorganic or organic salts and esters of nitric acid. These compounds contain the NO3- radical.
Hyperplasia: An increase in the number of cells in a tissue or organ without tumor formation. It differs from HYPERTROPHY, which is an increase in bulk without an increase in the number of cells.
| Excerpt | Relevance | Reference |
|---|---|---|
| " Treatment with simvastatin attenuated goblet cell hyperplasia, arginase-1 protein expression, and total arginase enzyme activity, but it did not alter airway hydroxyproline content or transforming growth factor-β1." | 7.76 | Simvastatin inhibits goblet cell hyperplasia and lung arginase in a mouse model of allergic asthma: a novel treatment for airway remodeling? ( Bratt, JM; Kenyon, NJ; Last, JA; Rabowsky, M; Zeki, AA, 2010) |
| "Sexual dimorphism exists in the response of rats to lead nitrate, liver hyperplasia occuring earlier and being more pronounced in males." | 7.70 | Cell cholesterol esters and high-density lipoprotein plasma levels during liver hyperplasia in choline-fed male and female rats. ( Batetta, B; Dessi, S; Marengo, B; Mulas, MF; Tessitore, L; Vizio, B, 2000) |
| "In the present study the three dimeric molecular forms of rat liver glucose-6-phosphate dehydrogenase were investigated during liver hyperplasia induced by lead nitrate." | 7.68 | Multiple molecular forms of rat liver glucose-6-phosphate dehydrogenase during liver hyperplasia induced by lead nitrate. ( Batetta, B; Carrucciu, A; Dessì, S; Pani, P; Pulisci, D, 1990) |
| "Wistar rats treated with lead nitrate were used in these experiments to provide evidence of the possible correlation between hyperplasia, induced cholesterol synthesis and the levels of glucose-6-phosphate dehydrogenase (G-6-PD) in the liver." | 7.67 | Hepatic cholesterol in lead nitrate induced liver hyperplasia. ( Batetta, B; Dessì, S; Ennas, C; Laconi, E; Pani, P, 1984) |
| "Liver hyperplasia was accompanied by an increase in cholesterol synthesis, an accumulation of cholesterol esters and by a stimulation of hexose-monophosphate (HMP) shunt enzyme activities." | 5.28 | Modifying influence of fasting on liver hyperplasia induced by lead nitrate. ( Batetta, B; Carrucciu, A; Dessì, S; Ferreli, A; Mura, E; Pani, P; Pulisci, D, 1990) |
| " Treatment with simvastatin attenuated goblet cell hyperplasia, arginase-1 protein expression, and total arginase enzyme activity, but it did not alter airway hydroxyproline content or transforming growth factor-β1." | 3.76 | Simvastatin inhibits goblet cell hyperplasia and lung arginase in a mouse model of allergic asthma: a novel treatment for airway remodeling? ( Bratt, JM; Kenyon, NJ; Last, JA; Rabowsky, M; Zeki, AA, 2010) |
| "GABAergic alterations in hypothalamus during compensatory hyperplasia after partial hepatectomy (PH), lead nitrate (LN) induced direct hyperplasia and N-nitrosodiethylamine (NDEA) induced neoplasia in liver were investigated." | 3.71 | Hypothalamic GABA receptor functional regulation and liver cell proliferation. ( Biju, MP; Paulose, CS; Pyroja, S; Rajeshkumar, NV, 2001) |
| "Sexual dimorphism exists in the response of rats to lead nitrate, liver hyperplasia occuring earlier and being more pronounced in males." | 3.70 | Cell cholesterol esters and high-density lipoprotein plasma levels during liver hyperplasia in choline-fed male and female rats. ( Batetta, B; Dessi, S; Marengo, B; Mulas, MF; Tessitore, L; Vizio, B, 2000) |
| "A single intravenous injection of lead nitrate (LN) to rats induces liver cell proliferation without causing cell necrosis (direct hyperplasia)." | 3.69 | Possible roles of nonparenchymal cells in hepatocyte proliferation induced by lead nitrate and by tumor necrosis factor alpha. ( Columbano, A; Katyal, SL; Ledda-Columbano, GM; Ohmura, T; Shinozuka, H; Zedda, AI, 1996) |
| "Apoptosis plays a major role in the regression of mitogen (lead nitrate)-induced hepatic hyperplasia." | 3.69 | Identification of apoptotic hepatocytes in situ in rat liver after lead nitrate administration. ( Deguchi, T; Hikita, H; Kagawa, K; Kakusui, M; Katagishi, T; Kimura, H; Nakajima, T; Ohkawara, T; Okanoue, T; Ueda, K, 1995) |
| "The steady-state levels of c-fos, c-jun and c-myc messenger RNA were investigated in rat liver tissue after proliferative stimuli of different nature-namely, compensatory regeneration induced by partial hepatectomy or carbon tetrachloride administration-and direct hyperplasia induced by four different hepatomitogens: lead nitrate, ethylene dibromide, cyproterone acetate and nafenopin." | 3.68 | Differences in the steady-state levels of c-fos, c-jun and c-myc messenger RNA during mitogen-induced liver growth and compensatory regeneration. ( Columbano, A; Coni, P; de Prati, AC; Ledda-Columbano, GM; Menegazzi, M; Sarma, DS; Simbula, G; Suzuki, H, 1993) |
| " The concentration of the isodipeptide was significantly increased in both when high rate of apoptosis with phagocytosis of dying hepatocytes was produced either by epidermal growth factor in the culture or by lead nitrate-induced hyperplasia with subsequent involution in rats." | 3.68 | Degradation of cells dying by apoptosis leads to accumulation of epsilon(gamma-glutamyl)lysine isodipeptide in culture fluid and blood. ( Autuori, F; Fesus, L; Kedei, N; Piacentini, M; Tarcsa, E, 1991) |
| " Animals were given a single dose of diethylnitrosamine (100 mg/kg body weight)." | 3.68 | Cell proliferation and promotion of rat liver carcinogenesis: different effect of hepatic regeneration and mitogen induced hyperplasia on the development of enzyme-altered foci. ( Chelo, A; Columbano, A; Curto, M; Ennas, MG; Ledda-Columbano, GM; Pani, P, 1990) |
| "In the present study the three dimeric molecular forms of rat liver glucose-6-phosphate dehydrogenase were investigated during liver hyperplasia induced by lead nitrate." | 3.68 | Multiple molecular forms of rat liver glucose-6-phosphate dehydrogenase during liver hyperplasia induced by lead nitrate. ( Batetta, B; Carrucciu, A; Dessì, S; Pani, P; Pulisci, D, 1990) |
| "Wistar rats treated with lead nitrate were used in these experiments to provide evidence of the possible correlation between hyperplasia, induced cholesterol synthesis and the levels of glucose-6-phosphate dehydrogenase (G-6-PD) in the liver." | 3.67 | Hepatic cholesterol in lead nitrate induced liver hyperplasia. ( Batetta, B; Dessì, S; Ennas, C; Laconi, E; Pani, P, 1984) |
| "Regression of renal hyperplasia after withdrawal of the mitogenic stimulus induced by a single injection of lead nitrate was studied in male Wistar rats." | 3.67 | Cell deletion by apoptosis during regression of renal hyperplasia. ( Columbano, A; Coni, P; Faa, G; Ledda-Columbano, GM; Pani, P, 1989) |
| "During the involution of lead nitrate-induced hyperplasia in rat liver a significant increase of transglutaminase activity, enzyme concentration, transglutaminase messenger RNA and protein-bound epsilon-(gamma-glutamyl)lysine (product of transglutaminase action) coincided with programmed death (apoptosis) of hepatocytes." | 3.67 | Induction and activation of tissue transglutaminase during programmed cell death. ( Falus, A; Fesus, L; Thomazy, V, 1987) |
| " To investigate short-term toxic effects and establish exposure parameters for chronic inhalation studies, F344/N rats and B6C3F1 mice were exposed to IBN vapors on a 6 hr/day + t90, 5 days/week schedule." | 1.28 | Prechronic inhalation toxicity studies of isobutyl nitrite. ( Abdo, KM; Aranyi, C; Gaworski, CL; Hall, A; Jackson, CD; Levine, BS, 1992) |
| "Liver hyperplasia was accompanied by an increase in cholesterol synthesis, an accumulation of cholesterol esters and by a stimulation of hexose-monophosphate (HMP) shunt enzyme activities." | 1.28 | Modifying influence of fasting on liver hyperplasia induced by lead nitrate. ( Batetta, B; Carrucciu, A; Dessì, S; Ferreli, A; Mura, E; Pani, P; Pulisci, D, 1990) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 10 (27.03) | 18.7374 |
| 1990's | 15 (40.54) | 18.2507 |
| 2000's | 6 (16.22) | 29.6817 |
| 2010's | 6 (16.22) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Lopez, CA | 1 |
| Miller, BM | 1 |
| Rivera-Chávez, F | 1 |
| Velazquez, EM | 1 |
| Byndloss, MX | 1 |
| Chávez-Arroyo, A | 1 |
| Lokken, KL | 1 |
| Tsolis, RM | 1 |
| Winter, SE | 1 |
| Bäumler, AJ | 1 |
| Zeki, AA | 1 |
| Bratt, JM | 1 |
| Rabowsky, M | 1 |
| Last, JA | 1 |
| Kenyon, NJ | 1 |
| Cooke, JP | 2 |
| Ghebremariam, YT | 1 |
| Vavra, AK | 1 |
| Havelka, GE | 1 |
| Martinez, J | 1 |
| Lee, VR | 1 |
| Fu, B | 1 |
| Jiang, Q | 1 |
| Keefer, LK | 1 |
| Kibbe, MR | 1 |
| Alef, MJ | 1 |
| Tzeng, E | 1 |
| Zuckerbraun, BS | 1 |
| Nemoto, K | 3 |
| Ikeda, A | 1 |
| Hikida, T | 1 |
| Kojima, M | 2 |
| Degawa, M | 3 |
| Murai, U | 2 |
| Yoshimura, N | 1 |
| Ayabe, Y | 1 |
| Nolan, BP | 1 |
| Senechal, P | 1 |
| Waqar, S | 1 |
| Myers, J | 1 |
| Standley, CA | 1 |
| Standley, PR | 1 |
| TOTTEN, RS | 2 |
| MORAN, TJ | 1 |
| PIERCE, GB | 1 |
| Columbano, A | 8 |
| Ledda-Columbano, GM | 8 |
| Coni, PP | 2 |
| Vargiu, M | 1 |
| Faa, G | 4 |
| Pani, P | 8 |
| Dessì, S | 4 |
| Batetta, B | 4 |
| Laconi, E | 2 |
| Ennas, C | 1 |
| Dini, L | 1 |
| Falasca, L | 1 |
| Lentini, A | 1 |
| Mattioli, P | 1 |
| Piacentini, M | 2 |
| Piredda, L | 1 |
| Autuori, F | 2 |
| Rijhsinghani, K | 1 |
| Choi, HS | 1 |
| Burton, LA | 1 |
| Paronetto, F | 1 |
| Tavoloni, N | 1 |
| Coni, P | 3 |
| Simbula, G | 1 |
| de Prati, AC | 1 |
| Menegazzi, M | 2 |
| Suzuki, H | 2 |
| Sarma, DS | 2 |
| Szabó, I | 1 |
| Kapp, P | 1 |
| Shinozuka, H | 2 |
| Ohmura, T | 1 |
| Katyal, SL | 1 |
| Zedda, AI | 1 |
| Nakajima, T | 1 |
| Deguchi, T | 1 |
| Kagawa, K | 1 |
| Hikita, H | 1 |
| Ueda, K | 1 |
| Katagishi, T | 1 |
| Ohkawara, T | 1 |
| Kakusui, M | 1 |
| Kimura, H | 1 |
| Okanoue, T | 1 |
| Carcereri-De Prati, A | 1 |
| Pibiri, M | 1 |
| Piga, R | 1 |
| Laconi, S | 1 |
| Pillai, S | 1 |
| Scintu, F | 1 |
| Curreli, F | 1 |
| Shafritz, DA | 1 |
| Vasdev, S | 1 |
| Ford, CA | 1 |
| Longerich, L | 1 |
| Parai, S | 1 |
| Gadag, V | 1 |
| Wadhawan, S | 1 |
| Miyata, S | 1 |
| Nemoto, F | 1 |
| Yasumoto, T | 1 |
| Kageyama, H | 1 |
| Tessitore, L | 1 |
| Vizio, B | 1 |
| Mulas, MF | 1 |
| Marengo, B | 1 |
| Uemura, S | 1 |
| Fathman, CG | 1 |
| Rothbard, JB | 1 |
| Biju, MP | 1 |
| Pyroja, S | 1 |
| Rajeshkumar, NV | 1 |
| Paulose, CS | 1 |
| Curto, M | 2 |
| Giacomini, L | 1 |
| Gaworski, CL | 1 |
| Aranyi, C | 1 |
| Hall, A | 1 |
| Levine, BS | 1 |
| Jackson, CD | 1 |
| Abdo, KM | 1 |
| Fesus, L | 2 |
| Tarcsa, E | 1 |
| Kedei, N | 1 |
| Ennas, MG | 1 |
| Chelo, A | 1 |
| Pulisci, D | 2 |
| Carrucciu, A | 2 |
| Mura, E | 1 |
| Ferreli, A | 1 |
| Thomazy, V | 1 |
| Falus, A | 1 |
| Liguori, C | 1 |
| Santa Cruz, G | 1 |
| Greenberg, SD | 1 |
| Györkey, F | 1 |
| Jenkins, DE | 1 |
| Györkey, P | 1 |
| Barnes, FW | 1 |
| Seip, WF | 1 |
| Burch, CC | 1 |
| Dungworth, DL | 1 |
| Clarke, GL | 1 |
| Plata, RL | 1 |
1 review available for nitrates and Hyperplasia
| Article | Year |
|---|---|
Nitric oxide and nitrite-based therapeutic opportunities in intimal hyperplasia.
Topics: Animals; Humans; Hyperplasia; Nitrates; Nitric Oxide; Nitrites; Signal Transduction; Tunica Intima; | 2012 |
36 other studies available for nitrates and Hyperplasia
| Article | Year |
|---|---|
Virulence factors enhance Citrobacter rodentium expansion through aerobic respiration.
Topics: Aerobiosis; Amyloid Precursor Protein Secretases; Animals; Citrobacter rodentium; Colitis; Colon; Cy | 2016 |
Simvastatin inhibits goblet cell hyperplasia and lung arginase in a mouse model of allergic asthma: a novel treatment for airway remodeling?
Topics: Aerosols; Animals; Arginase; Arginine; Asthma; Blotting, Western; Coloring Agents; Disease Models, A | 2010 |
Dietary nitrate, nitric oxide, and restenosis.
Topics: Animals; Coronary Restenosis; Diet; Endothelium, Vascular; Homeostasis; Hyperplasia; Mice; Models, B | 2011 |
Insights into the effect of nitric oxide and its metabolites nitrite and nitrate at inhibiting neointimal hyperplasia.
Topics: Animals; Cell Proliferation; Cells, Cultured; Coronary Artery Disease; Disease Models, Animal; Hyper | 2011 |
Induced expression of hepatic N-methyl-D-aspartate receptor 2C subunit gene during liver enlargement induced by lead nitrate, a hepatocellular mitogen.
Topics: Animals; Gene Expression Regulation; Hyperplasia; Lead; Liver; Male; Mitogens; Nitrates; Rats; Rats, | 2013 |
Altered gene expression of hepatic lanosterol 14alpha-demethylase (CYP51) in lead nitrate-treated rats.
Topics: Animals; CCAAT-Enhancer-Binding Proteins; Cytochrome P-450 Enzyme System; DNA-Binding Proteins; Gene | 2002 |
Altered insulin-like growth factor-1 and nitric oxide sensitivities in hypertension contribute to vascular hyperplasia.
Topics: Animals; Aorta, Thoracic; Biomarkers; Blood Pressure; Cell Count; Cell Survival; Cyclic GMP; Diastol | 2003 |
Cortisone and atypical pulmonary "epithelial" hyperplasia. Effects of pretreatment with cortisone on repair of chemically damaged rabbit lungs.
Topics: Animals; Cortisone; Hyperplasia; Lung; Lung Neoplasms; Nitrates; Precancerous Conditions; Rabbits; W | 1961 |
CORTISONE AND ATYPICAL PULMONARY "EPITHELIAL" HYPERPLASIA: FURTHER STUDIES INCLUDING ELECTRON MICROSCOPY, TISSUE CULTURE, ANIMAL TRANSPLANTATION AND LONG TERM OBSERVATIONS.
Topics: Cortisone; Cricetinae; Electrons; Epithelium; Hyperplasia; Lung; Lung Diseases; Microscopy; Microsco | 1964 |
Liver hyperplasia and regression after lead nitrate administration.
Topics: Animals; Cadmium; Cadmium Compounds; DNA; Female; Hyperplasia; Injections, Intravenous; Lead; Liver; | 1984 |
Hepatic cholesterol in lead nitrate induced liver hyperplasia.
Topics: Animals; Cholesterol; Cholesterol Esters; Glucosephosphate Dehydrogenase; Hyperplasia; Lead; Liver; | 1984 |
Galactose-specific receptor modulation related to the onset of apoptosis in rat liver.
Topics: Animals; Apoptosis; Asialoglycoprotein Receptor; Carbohydrates; Coated Pits, Cell-Membrane; Endocyto | 1993 |
Immunoelectron microscopy identification of early proliferating cells in rat liver tissue during hyperplasia induced by lead nitrate.
Topics: Animals; Antibodies, Monoclonal; Bromodeoxyuridine; Cell Division; Cell Nucleus; DNA; DNA Replicatio | 1993 |
Differences in the steady-state levels of c-fos, c-jun and c-myc messenger RNA during mitogen-induced liver growth and compensatory regeneration.
Topics: Animals; Carbon Tetrachloride; Cell Division; Cyproterone Acetate; Ethylene Dibromide; Gene Expressi | 1993 |
[The effect of carbamide feeding over a long period of time on thyroid gland activity in bulls].
Topics: Animal Feed; Animals; Cattle; Hyperplasia; In Vitro Techniques; Male; Microscopy, Electron; Nitrates | 1995 |
Possible roles of nonparenchymal cells in hepatocyte proliferation induced by lead nitrate and by tumor necrosis factor alpha.
Topics: Animals; Bromodeoxyuridine; Cell Division; DNA; Humans; Hyperplasia; Immunohistochemistry; Lead; Liv | 1996 |
Identification of apoptotic hepatocytes in situ in rat liver after lead nitrate administration.
Topics: Animals; Apoptosis; Coloring Agents; Deoxyuracil Nucleotides; DNA Damage; Eosine Yellowish-(YS); Hem | 1995 |
Liver cell proliferation induced by nafenopin and cyproterone acetate is not associated with increases in activation of transcription factors NF-kappaB and AP-1 or with expression of tumor necrosis factor alpha.
Topics: Animals; Binding, Competitive; Carbon Tetrachloride; Cell Division; Cyproterone Acetate; Hepatectomy | 1997 |
Direct hyperplasia does not enhance the kinetics of liver repopulation in a new model of hepatocyte transplantation in the rat.
Topics: Animals; Antineoplastic Agents, Phytogenic; Cell Transplantation; Dipeptidyl Peptidase 4; Disease Mo | 1999 |
Aldehyde induced hypertension in rats: prevention by N-acetyl cysteine.
Topics: Acetylcysteine; Aldehydes; Animals; Arterioles; Blood Platelets; Blood Pressure; Body Weight; Calciu | 1998 |
Gene expression of neurotrophins and their receptors in lead nitrate-induced rat liver hyperplasia.
Topics: Animals; Base Sequence; DNA Primers; Gene Expression; Hyperplasia; Lead; Liver; Male; Nerve Growth F | 2000 |
Cell cholesterol esters and high-density lipoprotein plasma levels during liver hyperplasia in choline-fed male and female rats.
Topics: Animals; Cholesterol Esters; Choline; Female; Hyperplasia; Lead; Lipoproteins, HDL; Liver; Male; Nit | 2000 |
Rapid and efficient vascular transport of arginine polymers inhibits myointimal hyperplasia.
Topics: Animals; Biological Transport, Active; Biotin; Carotid Arteries; Cell Division; Cells, Cultured; Dos | 2000 |
Hypothalamic GABA receptor functional regulation and liver cell proliferation.
Topics: Alkylating Agents; Animals; Cell Division; Chromatography, High Pressure Liquid; Diethylnitrosamine; | 2001 |
Mitogen-induced liver hyperplasia does not substitute for compensatory regeneration during promotion of chemical hepatocarcinogenesis.
Topics: 2-Acetylaminofluorene; Animals; Carbon Tetrachloride; Cell Division; Diethylnitrosamine; gamma-Gluta | 1992 |
Prechronic inhalation toxicity studies of isobutyl nitrite.
Topics: Administration, Inhalation; Animals; Body Weight; Bone Marrow; Butanes; Epithelium; Female; Hematolo | 1992 |
Degradation of cells dying by apoptosis leads to accumulation of epsilon(gamma-glutamyl)lysine isodipeptide in culture fluid and blood.
Topics: Animals; Animals, Newborn; Cell Survival; Cells, Cultured; Dipeptides; Epidermal Growth Factor; Hype | 1991 |
Cell proliferation and promotion of rat liver carcinogenesis: different effect of hepatic regeneration and mitogen induced hyperplasia on the development of enzyme-altered foci.
Topics: Adenosine Triphosphatases; Animals; Body Weight; Carbon Tetrachloride; Cell Division; Diethylnitrosa | 1990 |
Multiple molecular forms of rat liver glucose-6-phosphate dehydrogenase during liver hyperplasia induced by lead nitrate.
Topics: Animals; Electrophoresis, Polyacrylamide Gel; Fasting; Glucosephosphate Dehydrogenase; Hyperplasia; | 1990 |
Modifying influence of fasting on liver hyperplasia induced by lead nitrate.
Topics: Animals; Cholesterol; DNA; Fasting; Glucosephosphate Dehydrogenase; Hyperplasia; Lead; Liver; Male; | 1990 |
Cell deletion by apoptosis during regression of renal hyperplasia.
Topics: Animals; Cell Division; Cell Survival; Hyperplasia; Inclusion Bodies; Injections, Intravenous; Kidne | 1989 |
Induction and activation of tissue transglutaminase during programmed cell death.
Topics: Animals; Cell Survival; Enzyme Activation; Enzyme Induction; Glucocorticoids; Hyperplasia; Lead; Liv | 1987 |
Occurrence of cell death (apoptosis) during the involution of liver hyperplasia.
Topics: Alanine Transaminase; Animals; Cell Survival; Chemical and Drug Induced Liver Injury; DNA; Hyperplas | 1985 |
Alveolar epithelial cells following exposure to nitric acid. Electron microscopic study in rats.
Topics: Animals; Bronchitis; Carbon; Cytoplasm; Epithelium; Hyperplasia; Indicators and Reagents; Intubation | 1971 |
Proliferation and resistance of epidermis in response to harmful stimuli.
Topics: Animals; Fluorides; Fluorine; Guinea Pigs; Hyperplasia; Lanolin; Mice; Nicotiana; Nitrates; Nitroben | 1968 |
Pulmonary lesions produced in A-strain mice by long-term exposure to peroxyacetyl nitrate.
Topics: Air Pollution; Animals; Body Weight; Bronchi; Bronchitis; Bronchopneumonia; Environmental Exposure; | 1969 |