nisoldipine has been researched along with Acidosis in 2 studies
Nisoldipine: A dihydropyridine calcium channel antagonist that acts as a potent arterial vasodilator and antihypertensive agent. It is also effective in patients with cardiac failure and angina.
nisoldipine : A racemate consisting of equimolar amounts of (R)- and (S)-nisoldipine. A calcium channel blocker, it is used in the treatment of hypertension and angina pectoris.
methyl 2-methylpropyl 2,6-dimethyl-4-(2-nitrophenyl)-1,4-dihydropyridine-3,5-dicarboxylate : A dihydropyridine that is 1,4-dihydropyridine which is substituted by methyl groups at positions 2 and 6, a methoxycarbonyl group at position 3, an o-nitrophenyl group at position 4, and an isobutoxycarbonyl group at position 5. The racemate, a calcium channel blocker, is used in the treatment of hypertension and angina pectoris.
Acidosis: A pathologic condition of acid accumulation or depletion of base in the body. The two main types are RESPIRATORY ACIDOSIS and metabolic acidosis, due to metabolic acid build up.
| Excerpt | Relevance | Reference |
|---|---|---|
| "We examined whether nisoldipine, a calcium (Ca) channel blocker, increases coronary blood flow (CBF) without decreasing aortic blood pressure (AoP) with ischemic and nonischemic hearts, and whether the presence of cellular acidosis in ischemic myocardium contributes to the augmentation of coronary vasodilation due to nisoldipine." | 7.70 | Nisoldipine selectively induces coronary vasodilation and improves mild myocardial ischemia in dogs: a potential role of cellular acidosis. ( Funaya, H; Hori, M; Kitakaze, M; Komamura, K; Kuzuya, T; Minamino, T; Mori, H; Node, K; Takeda, H, 1998) |
| "We examined whether nisoldipine, a calcium (Ca) channel blocker, increases coronary blood flow (CBF) without decreasing aortic blood pressure (AoP) with ischemic and nonischemic hearts, and whether the presence of cellular acidosis in ischemic myocardium contributes to the augmentation of coronary vasodilation due to nisoldipine." | 3.70 | Nisoldipine selectively induces coronary vasodilation and improves mild myocardial ischemia in dogs: a potential role of cellular acidosis. ( Funaya, H; Hori, M; Kitakaze, M; Komamura, K; Kuzuya, T; Minamino, T; Mori, H; Node, K; Takeda, H, 1998) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (100.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Stengl, M | 1 |
| Carmeliet, E | 1 |
| Mubagwa, K | 1 |
| Flameng, W | 1 |
| Kitakaze, M | 1 |
| Funaya, H | 1 |
| Komamura, K | 1 |
| Node, K | 1 |
| Minamino, T | 1 |
| Mori, H | 1 |
| Takeda, H | 1 |
| Kuzuya, T | 1 |
| Hori, M | 1 |
2 other studies available for nisoldipine and Acidosis
| Article | Year |
|---|---|
Modulation of transient outward current by extracellular protons and Cd2+ in rat and human ventricular myocytes.
Topics: Acidosis; Acids; Animals; Binding, Competitive; Cadmium; Calcium Channel Blockers; Calcium Channels; | 1998 |
Nisoldipine selectively induces coronary vasodilation and improves mild myocardial ischemia in dogs: a potential role of cellular acidosis.
Topics: Acidosis; Amiloride; Animals; Bicarbonates; Blood Pressure; Buffers; Coronary Circulation; Diuretics | 1998 |