nimodipine has been researched along with Hyperglycemia, Postprandial in 3 studies
Nimodipine: A calcium channel blockader with preferential cerebrovascular activity. It has marked cerebrovascular dilating effects and lowers blood pressure.
nimodipine : A dihydropyridine that is 1,4-dihydropyridine which is substituted by methyl groups at positions 2 and 6, a (2-methoxyethoxy)carbonyl group at position 3, a m-nitrophenyl group at position 4, and an isopropoxycarbonyl group at position 5. An L-type calcium channel blocker, it acts particularly on cerebral circulation, and is used both orally and intravenously for the prevention and treatment of subarachnoid hemorrhage from ruptured intracranial aneurysm.
Hyperglycemia, Postprandial: Abnormally high BLOOD GLUCOSE level after a meal.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Preischemic hyperglycemia is recognized to increase ischemic tissue acidosis and the present studies were undertaken to correlate depolarization and tissue acidosis during acute focal cerebral ischemia and hyperglycemia." | 5.30 | In vivo uptake of [3H]nimodipine in focal cerebral ischemia: modulation by hyperglycemia. ( Hogan, MJ; Osuga, S, 1997) |
| "Nimodipine-treated rats received the drug as an intravenous infusion of 0." | 5.28 | Nimodipine prevents hyperglycemia-induced cerebral acidosis in middle cerebral artery occluded rats. ( Berger, L; Hakim, AM, 1989) |
| "Preischemic hyperglycemia is recognized to increase ischemic tissue acidosis and the present studies were undertaken to correlate depolarization and tissue acidosis during acute focal cerebral ischemia and hyperglycemia." | 1.30 | In vivo uptake of [3H]nimodipine in focal cerebral ischemia: modulation by hyperglycemia. ( Hogan, MJ; Osuga, S, 1997) |
| "Nimodipine-treated rats received the drug as an intravenous infusion of 0." | 1.28 | Nimodipine prevents hyperglycemia-induced cerebral acidosis in middle cerebral artery occluded rats. ( Berger, L; Hakim, AM, 1989) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (33.33) | 18.7374 |
| 1990's | 1 (33.33) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Wang, H | 1 |
| James, ML | 1 |
| Venkatraman, TN | 1 |
| Wilson, LJ | 1 |
| Lyuboslavsky, P | 1 |
| Myers, SJ | 1 |
| Lascola, CD | 1 |
| Laskowitz, DT | 1 |
| Osuga, S | 1 |
| Hogan, MJ | 1 |
| Berger, L | 1 |
| Hakim, AM | 1 |
3 other studies available for nimodipine and Hyperglycemia, Postprandial
| Article | Year |
|---|---|
pH-sensitive NMDA inhibitors improve outcome in a murine model of SAH.
Topics: Animals; Behavior, Animal; Calcium Channel Blockers; Disease Models, Animal; Hydrogen-Ion Concentrat | 2014 |
In vivo uptake of [3H]nimodipine in focal cerebral ischemia: modulation by hyperglycemia.
Topics: Animals; Autoradiography; Blood Flow Velocity; Calcium Channel Blockers; Cerebral Cortex; Corpus Str | 1997 |
Nimodipine prevents hyperglycemia-induced cerebral acidosis in middle cerebral artery occluded rats.
Topics: Acidosis; Animals; Brain Diseases; Brain Ischemia; Cerebrovascular Circulation; Hydrogen-Ion Concent | 1989 |