Page last updated: 2024-11-01

nilvadipine and Innate Inflammatory Response

nilvadipine has been researched along with Innate Inflammatory Response in 3 studies

Research Excerpts

ExcerptRelevanceReference
" The aim of the present study was to examine whether the CCBs nilvadipine and diltiazem reduce ocular inflammation in endotoxin-induced uveitis (EIU)."7.76Calcium channel blocker nilvadipine, but not diltiazem, inhibits ocular inflammation in endotoxin-induced uveitis. ( Ishida, S; Koto, T; Nagai, N; Oike, Y, 2010)
" The aim of the present study was to examine whether the CCBs nilvadipine and diltiazem reduce ocular inflammation in endotoxin-induced uveitis (EIU)."3.76Calcium channel blocker nilvadipine, but not diltiazem, inhibits ocular inflammation in endotoxin-induced uveitis. ( Ishida, S; Koto, T; Nagai, N; Oike, Y, 2010)

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19901 (33.33)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (33.33)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Morin, A1
Mouzon, B1
Ferguson, S1
Paris, D1
Browning, M1
Stewart, W1
Mullan, M1
Crawford, F1
Ishida, S1
Koto, T1
Nagai, N1
Oike, Y1
Nomoto, A1
Mutoh, S1
Hagihara, H1
Yamaguchi, I1

Other Studies

3 other studies available for nilvadipine and Innate Inflammatory Response

ArticleYear
Nilvadipine suppresses inflammation via inhibition of P-SYK and restores spatial memory deficits in a mouse model of repetitive mild TBI.
    Acta neuropathologica communications, 2020, 10-19, Volume: 8, Issue:1

    Topics: Animals; Antigens, CD; Antigens, Differentiation, Myelomonocytic; Brain Concussion; Calcium Channel

2020
Calcium channel blocker nilvadipine, but not diltiazem, inhibits ocular inflammation in endotoxin-induced uveitis.
    Japanese journal of ophthalmology, 2010, Volume: 54, Issue:6

    Topics: Animals; Blood-Aqueous Barrier; Calcium Channel Blockers; Cell Adhesion; Chemokine CCL2; Diltiazem;

2010
Smooth muscle cell migration induced by inflammatory cell products and its inhibition by a potent calcium antagonist, nilvadipine.
    Atherosclerosis, 1988, Volume: 72, Issue:2-3

    Topics: Animals; Aorta, Thoracic; Calcium; Calcium Channel Blockers; Cells, Cultured; Chemotaxis; Exudates a

1988