Page last updated: 2024-08-26

nicotine and Pulmonary Arterial Remodeling

nicotine has been researched along with Pulmonary Arterial Remodeling in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (33.33)24.3611
2020's2 (66.67)2.80

Authors

AuthorsStudies
Chen, L; Du, X; Gao, A; Hao, Y; Hou, X; Li, Q; Qin, X; Shi, Y; Xu, X; Zhao, L1
Fried, ND; Gardner, JD; Gilpin, NW; Lazartigues, E; Lobell, TD; Morris, TM; Oakes, JM; Pearson, CS; Xu, J; Yue, X1
Allawzi, A; Chichger, H; Choudhary, G; Clements, RT; Dudley, SC; Jeong, EM; Kue, N; Lu, Q; O'Connell, K; Rounds, S; Sakhatskyy, P; Vang, A; Zhang, P1

Other Studies

3 other study(ies) available for nicotine and Pulmonary Arterial Remodeling

ArticleYear
Down-regulation of connexin 43 contributes to structure and function of pulmonary artery in nicotine-administered mice.
    Toxicology letters, 2023, Mar-15, Volume: 377

    Topics: Animals; Connexin 43; Connexins; Down-Regulation; Mice; Mice, Inbred C57BL; Nicotine; Pulmonary Artery; RNA, Messenger; Vascular Remodeling

2023
Effects of Chronic Nicotine Inhalation on Systemic and Pulmonary Blood Pressure and Right Ventricular Remodeling in Mice.
    Hypertension (Dallas, Tex. : 1979), 2020, Volume: 75, Issue:5

    Topics: Administration, Inhalation; Angiotensin II; Animals; Atmosphere Exposure Chambers; Blood Pressure; Cardiac Catheterization; Hypertension, Pulmonary; Hypertrophy, Right Ventricular; Male; Mice; Mice, Inbred C57BL; Nicotine; Pulmonary Artery; Vascular Remodeling; Vascular Resistance; Ventricular Remodeling

2020
Effect of α7 nicotinic acetylcholine receptor activation on cardiac fibroblasts: a mechanism underlying RV fibrosis associated with cigarette smoke exposure.
    American journal of physiology. Lung cellular and molecular physiology, 2017, 05-01, Volume: 312, Issue:5

    Topics: alpha7 Nicotinic Acetylcholine Receptor; Animals; Cell Proliferation; Enzyme Activation; Fibroblasts; Heart Ventricles; Hemodynamics; Hypertrophy, Right Ventricular; Male; MAP Kinase Signaling System; Mice, Inbred AKR; Myocardium; Nicotine; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Protein Kinase C-alpha; Protein Kinase C-delta; Rats, Sprague-Dawley; Smoking; Vascular Remodeling; Ventricular Dysfunction, Right

2017