nicotine has been researched along with Conus Medullaris Syndrome in 1 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Flora, G; Geddes, JW; Hennig, B; Ravikumar, R; Toborek, M | 1 |
1 other study(ies) available for nicotine and Conus Medullaris Syndrome
| Article | Year |
|---|---|
Nicotine attenuates oxidative stress, activation of redox-regulated transcription factors and induction of proinflammatory genes in compressive spinal cord trauma.
Topics: Animals; Binding, Competitive; Chemokine CCL2; Cyclic AMP Response Element-Binding Protein; Disease Models, Animal; Dose-Response Relationship, Drug; Gene Expression Regulation; Inflammation; Inflammation Mediators; Male; NF-kappa B; Nicotine; Nicotinic Antagonists; Oxidation-Reduction; Oxidative Stress; Rats; Rats, Long-Evans; Receptors, Nicotinic; RNA, Messenger; Spinal Cord Compression; Transcription Factor AP-1; Transcription Factors; Treatment Outcome; Tumor Necrosis Factor-alpha; Up-Regulation | 2004 |