nicotine has been researched along with Cleft Palate in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Chen, J; Du, J; Li, T; Liu, Y; Peng, X; Wang, X; Wang, Y; Yao, Y | 1 |
Canturk, KM; Nawshad, A; Otu, HH; Ozturk, F; Sharma, J; Sheldon, E | 1 |
Baroni, T; Bellucci, C; Bodo, M; Carinci, F; Lilli, C; Lumare, E; Palmieri, A; Pezzetti, F; Stabellini, G | 1 |
Kang, P; Svoboda, KK | 1 |
4 other study(ies) available for nicotine and Cleft Palate
Article | Year |
---|---|
Autophagy triggered by the ROS/ERK signaling pathway protects mouse embryonic palatal cells from apoptosis induced by nicotine.
Topics: Acetylcysteine; Animals; Apoptosis; Autophagy; Chloroquine; Cleft Lip; Cleft Palate; Electronic Nicotine Delivery Systems; MAP Kinase Signaling System; Mice; Microtubule-Associated Proteins; Nicotine; Osteogenesis; Reactive Oxygen Species; Signal Transduction; Teratogens | 2022 |
Nicotine Exposure During Pregnancy Results in Persistent Midline Epithelial Seam With Improper Palatal Fusion.
Topics: Animals; Cleft Palate; Female; Fetal Development; Humans; Infant, Low Birth Weight; Infant, Newborn; Mice, Inbred BALB C; Models, Animal; Nicotine; Palate; Pregnancy; Pregnancy Outcome; Prenatal Exposure Delayed Effects; Smoking; Stillbirth | 2016 |
Human cleft lip and palate fibroblasts and normal nicotine-treated fibroblasts show altered in vitro expressions of genes related to molecular signaling pathways and extracellular matrix metabolism.
Topics: Case-Control Studies; Cell Shape; Cell Survival; Cells, Cultured; Child, Preschool; Cleft Lip; Cleft Palate; Extracellular Matrix; Fibroblasts; Gene Expression Profiling; Gene Expression Regulation; Genotype; Humans; Male; Nicotine; Nicotinic Agonists; Phenotype; Reverse Transcriptase Polymerase Chain Reaction; Signal Transduction | 2010 |
Nicotine inhibits palatal fusion and modulates nicotinic receptors and the PI-3 kinase pathway in medial edge epithelia.
Topics: Animals; Bungarotoxins; Cleft Palate; DNA-Binding Proteins; Epithelium; Immunohistochemistry; Mesoderm; Mice; Mice, Inbred Strains; Microscopy, Confocal; Nicotine; Nicotinic Agonists; Organ Culture Techniques; Palate, Hard; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Receptors, Nicotinic; Signal Transduction; Smad2 Protein; Trans-Activators; Transforming Growth Factor beta; Transforming Growth Factor beta3 | 2003 |