Page last updated: 2024-08-26

nicotine and Carcinoma, Transitional Cell

nicotine has been researched along with Carcinoma, Transitional Cell in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chen, RJ; Guo, HR; Ho, YS; Wang, YJ2
Han, X; Li, B; Li, W; Liu, D; Pan, F; Pang, Z; Shi, Y; Zhang, Q1

Other Studies

3 other study(ies) available for nicotine and Carcinoma, Transitional Cell

ArticleYear
Long-term nicotine exposure-induced chemoresistance is mediated by activation of Stat3 and downregulation of ERK1/2 via nAChR and beta-adrenoceptors in human bladder cancer cells.
    Toxicological sciences : an official journal of the Society of Toxicology, 2010, Volume: 115, Issue:1

    Topics: Apoptosis; Carcinoma, Transitional Cell; Cell Cycle; Cell Line, Tumor; Cell Proliferation; Cell Survival; Cyclin D1; Drug Resistance, Neoplasm; Humans; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Nicotine; Nicotinic Agonists; Proliferating Cell Nuclear Antigen; Receptors, Adrenergic, beta; Receptors, Nicotinic; RNA, Small Interfering; Signal Transduction; STAT3 Transcription Factor; Transfection; Urinary Bladder Neoplasms

2010
Intervention of nicotine on MNU-induced bladder cancer in rats.
    Journal of Huazhong University of Science and Technology. Medical sciences = Hua zhong ke ji da xue xue bao. Yi xue Ying De wen ban = Huazhong keji daxue xuebao. Yixue Yingdewen ban, 2011, Volume: 31, Issue:1

    Topics: Animals; Carcinogens; Carcinoma, Transitional Cell; Female; Methylnitrosourea; Mutation; Nicotine; Rats; Rats, Wistar; Tumor Suppressor Protein p53; Urinary Bladder Neoplasms

2011
Rapid activation of Stat3 and ERK1/2 by nicotine modulates cell proliferation in human bladder cancer cells.
    Toxicological sciences : an official journal of the Society of Toxicology, 2008, Volume: 104, Issue:2

    Topics: Carcinoma, Transitional Cell; Cell Line, Tumor; Cell Proliferation; Cyclin D; Cyclins; Dose-Response Relationship, Drug; Epithelium; Humans; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Nicotine; Protein Kinase C; Receptors, Nicotinic; Signal Transduction; STAT3 Transcription Factor; Urinary Bladder Neoplasms

2008