Page last updated: 2024-11-01

niclosamide and Reperfusion Injury

niclosamide has been researched along with Reperfusion Injury in 1 studies

Niclosamide: An antihelmintic that is active against most tapeworms. (From Martindale, The Extra Pharmacopoeia, 30th ed, p48)
niclosamide : A secondary carboxamide resulting from the formal condensation of the carboxy group of 5-chlorosalicylic acid with the amino group of 2-chloro-4-nitroaniline. It is an oral anthelmintic drug approved for use against tapeworm infections.

Reperfusion Injury: Adverse functional, metabolic, or structural changes in tissues that result from the restoration of blood flow to the tissue (REPERFUSION) following ISCHEMIA.

Research Excerpts

ExcerptRelevanceReference
"Treatment with niclosamide was associated with induction of autophagy and an overall improvement in renal function."5.46Niclosamide attenuates inflammatory cytokines via the autophagy pathway leading to improved outcomes in renal ischemia/reperfusion injury. ( Ding, XG; Gao, SL; Sun, DL; Zhang, HM; Zhang, LX; Zhao, HJ, 2017)
"Treatment with niclosamide was associated with induction of autophagy and an overall improvement in renal function."1.46Niclosamide attenuates inflammatory cytokines via the autophagy pathway leading to improved outcomes in renal ischemia/reperfusion injury. ( Ding, XG; Gao, SL; Sun, DL; Zhang, HM; Zhang, LX; Zhao, HJ, 2017)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Zhang, LX1
Zhao, HJ1
Sun, DL1
Gao, SL1
Zhang, HM1
Ding, XG1

Other Studies

1 other study available for niclosamide and Reperfusion Injury

ArticleYear
Niclosamide attenuates inflammatory cytokines via the autophagy pathway leading to improved outcomes in renal ischemia/reperfusion injury.
    Molecular medicine reports, 2017, Volume: 16, Issue:2

    Topics: Animals; Autophagosomes; Autophagy; Beclin-1; Biomarkers; Blood Urea Nitrogen; Creatinine; Cytokines

2017