nickel has been researched along with Alveolitis, Fibrosing in 23 studies
Nickel: A trace element with the atomic symbol Ni, atomic number 28, and atomic weight 58.69. It is a cofactor of the enzyme UREASE.
nickel ion : A nickel atom having a net electric charge.
nickel atom : Chemical element (nickel group element atom) with atomic number 28.
Excerpt | Relevance | Reference |
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"Nickel oxide nanoparticles (NiO NPs) causes pulmonary fibrosis via activating transforming growth factor-β1 (TGF-β1) in rats, but its upstream regulatory mechanisms are unknown." | 8.02 | LncRNA MEG3 mediates nickel oxide nanoparticles-induced pulmonary fibrosis via suppressing TGF-β1 expression and epithelial-mesenchymal transition process. ( Chang, X; Gao, Q; Li, C; Li, S; Liu, H; Sun, Y; Wang, X; Yang, M; Zhan, H, 2021) |
"Animal studies have shown that nickel compounds may induce pulmonary fibrosis, but so far only limited documentation in humans has been available." | 7.72 | Radiographic evidence of pulmonary fibrosis and possible etiologic factors at a nickel refinery in Norway. ( Berge, SR; Skyberg, K, 2003) |
"To identify pulmonary fibrosis, Masson trichrome staining, hydroxyproline content, and collagen protein expression were performed." | 5.46 | Nickel oxide nanoparticles induced pulmonary fibrosis via TGF- β1 activation in rats. ( Chang, XH; Han, AJ; Li, J; Li, S; Liu, FF; Liu, SK; Su, L; Sun, YB; Sun, YF; Sun, YY; Zhou, HH; Zhu, A; Zou, LY, 2017) |
"Nickel oxide nanoparticles (NiO NPs) causes pulmonary fibrosis via activating transforming growth factor-β1 (TGF-β1) in rats, but its upstream regulatory mechanisms are unknown." | 4.02 | LncRNA MEG3 mediates nickel oxide nanoparticles-induced pulmonary fibrosis via suppressing TGF-β1 expression and epithelial-mesenchymal transition process. ( Chang, X; Gao, Q; Li, C; Li, S; Liu, H; Sun, Y; Wang, X; Yang, M; Zhan, H, 2021) |
"Nickel oxide nanoparticles (Nano NiO) could induce pulmonary fibrosis, however, the mechanisms are still unknown." | 3.91 | TGF-β1 mediated MAPK signaling pathway promotes collagen formation induced by Nano NiO in A549 cells. ( Chang, X; Li, C; Li, S; Sun, Y; Tian, M; Zhang, Q, 2019) |
"Animal studies have shown that nickel compounds may induce pulmonary fibrosis, but so far only limited documentation in humans has been available." | 3.72 | Radiographic evidence of pulmonary fibrosis and possible etiologic factors at a nickel refinery in Norway. ( Berge, SR; Skyberg, K, 2003) |
"To identify pulmonary fibrosis, Masson trichrome staining, hydroxyproline content, and collagen protein expression were performed." | 1.46 | Nickel oxide nanoparticles induced pulmonary fibrosis via TGF- β1 activation in rats. ( Chang, XH; Han, AJ; Li, J; Li, S; Liu, FF; Liu, SK; Su, L; Sun, YB; Sun, YF; Sun, YY; Zhou, HH; Zhu, A; Zou, LY, 2017) |
"One cause of debilitating pulmonary fibrosis is inhalation of insoluble metals." | 1.31 | Nickel-induced plasminogen activator inhibitor-1 expression inhibits the fibrinolytic activity of human airway epithelial cells. ( Andrew, A; Barchowsky, A, 2000) |
"Experiments on acute and chronic inhalation of copper and nickel sulfide ore dust proved that the dust of rich ore, if compared with that of copper-bearing and impregnated ore, induces more marked toxic effects." | 1.30 | [Fibrogenic and general toxic effects of copper and nickel sulfide ore dust (data for a hygienic evaluation)]. ( Borisenkova, RV; Gvozdeva, LL; Ivanova, LG; Lutsenko, LA; Skriabin, SIu, 1997) |
"This report presents the results of noncancer dose-response modeling for inhalation and oral exposures to nickel compounds using the NOAEL/LOAEL and benchmark dose (BMD) approaches." | 1.30 | Non-cancer risk assessment for nickel compounds: issues associated with dose-response modeling of inhalation and oral exposures. ( Allen, BC; Haber, LT; Kimmel, CA, 1998) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 9 (39.13) | 18.7374 |
1990's | 4 (17.39) | 18.2507 |
2000's | 2 (8.70) | 29.6817 |
2010's | 6 (26.09) | 24.3611 |
2020's | 2 (8.70) | 2.80 |
Authors | Studies |
---|---|
Mo, Y | 1 |
Zhang, Y | 1 |
Wan, R | 1 |
Jiang, M | 1 |
Xu, Y | 1 |
Zhang, Q | 2 |
Zhan, H | 1 |
Chang, X | 2 |
Wang, X | 1 |
Yang, M | 1 |
Gao, Q | 1 |
Liu, H | 1 |
Li, C | 3 |
Li, S | 3 |
Sun, Y | 2 |
Wu, CH | 2 |
Hsiao, YM | 1 |
Yeh, KT | 1 |
Tsou, TC | 1 |
Chen, CY | 1 |
Wu, MF | 1 |
Ko, JL | 2 |
Yang, L | 1 |
Lin, Z | 1 |
Wang, Y | 1 |
Xu, W | 1 |
Li, Q | 1 |
Yao, W | 1 |
Song, Z | 1 |
Liu, G | 1 |
Tian, M | 1 |
Glista-Baker, EE | 1 |
Taylor, AJ | 1 |
Sayers, BC | 1 |
Thompson, EA | 1 |
Bonner, JC | 1 |
Chang, XH | 1 |
Zhu, A | 1 |
Liu, FF | 1 |
Zou, LY | 1 |
Su, L | 1 |
Liu, SK | 1 |
Zhou, HH | 1 |
Sun, YY | 1 |
Han, AJ | 1 |
Sun, YF | 1 |
Li, J | 1 |
Sun, YB | 1 |
Tang, SC | 1 |
Wang, PH | 1 |
Lee, H | 1 |
Berge, SR | 1 |
Skyberg, K | 1 |
Woźniak, H | 1 |
Muir, DC | 1 |
Julian, J | 1 |
Jadon, N | 1 |
Roberts, R | 1 |
Roos, J | 1 |
Chan, J | 1 |
Maehle, W | 1 |
Morgan, WK | 1 |
Benson, JM | 1 |
Chang, IY | 1 |
Cheng, YS | 1 |
Hahn, FF | 1 |
Kennedy, CH | 1 |
Barr, EB | 1 |
Maples, KR | 1 |
Snipes, MB | 1 |
Lutsenko, LA | 1 |
Borisenkova, RV | 1 |
Gvozdeva, LL | 1 |
Skriabin, SIu | 1 |
Ivanova, LG | 1 |
Haber, LT | 1 |
Allen, BC | 1 |
Kimmel, CA | 1 |
Andrew, A | 1 |
Barchowsky, A | 1 |
Stettler, LE | 2 |
Groth, DH | 1 |
MacKay, GR | 1 |
Proctor, JE | 1 |
Platek, SF | 1 |
Carolan, RJ | 1 |
Smith, RJ | 1 |
Donaldson, HM | 1 |
Schneider, WD | 1 |
Konetzke, GW | 1 |
Siegesmund, KA | 1 |
Funahashi, A | 1 |
Pintar, K | 1 |
Cralley, LJ | 1 |
Lainhart, WS | 1 |
Jones, JG | 1 |
Warner, CG | 1 |
Kosova, LV | 1 |
Kurennaia, SS | 1 |
Latushkina, VB | 1 |
Kochetkova, TA | 1 |
Kireev, VI | 1 |
Tolgskaia, MS | 1 |
23 other studies available for nickel and Alveolitis, Fibrosing
Article | Year |
---|---|
miR-21 mediates nickel nanoparticle-induced pulmonary injury and fibrosis.
Topics: Animals; Cytokines; Lung; Lung Injury; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; MicroRNAs; Na | 2020 |
LncRNA MEG3 mediates nickel oxide nanoparticles-induced pulmonary fibrosis via suppressing TGF-β1 expression and epithelial-mesenchymal transition process.
Topics: Animals; Epithelial-Mesenchymal Transition; Male; Nanoparticles; Nickel; Pulmonary Fibrosis; Rats; R | 2021 |
Upregulation of microRNA-4417 and Its Target Genes Contribute to Nickel Chloride-promoted Lung Epithelial Cell Fibrogenesis and Tumorigenesis.
Topics: Cell Line, Transformed; Cell Transformation, Neoplastic; Epithelial Cells; Humans; Lung; Lung Neopla | 2017 |
Nickle(II) ions exacerbate bleomycin-induced pulmonary inflammation and fibrosis by activating the ROS/Akt signaling pathway.
Topics: A549 Cells; Animals; Bleomycin; Cell Survival; Disease Models, Animal; Humans; Mice; Mice, Inbred C5 | 2018 |
TGF-β1 mediated MAPK signaling pathway promotes collagen formation induced by Nano NiO in A549 cells.
Topics: A549 Cells; Collagen Type I; Humans; Hydroxyproline; MAP Kinase Signaling System; Mitogen-Activated | 2019 |
Nickel nanoparticles cause exaggerated lung and airway remodeling in mice lacking the T-box transcription factor, TBX21 (T-bet).
Topics: Airway Remodeling; Animals; Antibodies, Blocking; Antibodies, Monoclonal; Bronchoalveolar Lavage Flu | 2014 |
Nickel oxide nanoparticles induced pulmonary fibrosis via TGF- β1 activation in rats.
Topics: Animals; Gene Expression Regulation; Male; Metal Nanoparticles; Nickel; Pulmonary Fibrosis; Rats; Ra | 2017 |
Nickel-induced epithelial-mesenchymal transition by reactive oxygen species generation and E-cadherin promoter hypermethylation.
Topics: Acetylcysteine; Antigens, Differentiation; Azacitidine; Bronchi; Cadherins; Cell Line, Transformed; | 2012 |
Radiographic evidence of pulmonary fibrosis and possible etiologic factors at a nickel refinery in Norway.
Topics: Adult; Age Factors; Aged; Aged, 80 and over; Humans; Male; Middle Aged; Nickel; Norway; Occupational | 2003 |
[Polish asbestos and its fibrogenic effect. I. Fibrous minerals in nickel ore deposits].
Topics: Animals; Asbestos; Asbestosis; Chromium; Copper; Crystallization; Iron; Mining; Nickel; Particle Siz | 1984 |
Prevalence of small opacities in chest radiographs of nickel sinter plant workers.
Topics: Adult; Aged; Carcinogens; Dust; Humans; Lung; Male; Metallurgy; Middle Aged; Nickel; Occupational Di | 1993 |
Particle clearance and histopathology in lungs of F344/N rats and B6C3F1 mice inhaling nickel oxide or nickel sulfate.
Topics: Administration, Inhalation; Animals; Hyperplasia; Lung; Lung Diseases, Interstitial; Macrophages, Al | 1995 |
[Fibrogenic and general toxic effects of copper and nickel sulfide ore dust (data for a hygienic evaluation)].
Topics: Animals; Copper; Dust; Male; Maximum Allowable Concentration; Mining; Nickel; Pneumoconiosis; Pulmon | 1997 |
Non-cancer risk assessment for nickel compounds: issues associated with dose-response modeling of inhalation and oral exposures.
Topics: Administration, Oral; Animals; Dose-Response Relationship, Drug; Humans; Inhalation Exposure; Lung; | 1998 |
Nickel-induced plasminogen activator inhibitor-1 expression inhibits the fibrinolytic activity of human airway epithelial cells.
Topics: Bronchi; Epithelial Cells; Fibrinolysis; Humans; Nickel; Plasminogen Activator Inhibitor 1; Pulmonar | 2000 |
Identification of stainless steel welding fume particulates in human lung and environmental samples using electron probe microanalysis.
Topics: Air Pollutants, Occupational; Chromium; Electron Probe Microanalysis; Environmental Exposure; Filtra | 1977 |
Fibrogenicity and carcinogenic potential of smelter slags used as abrasive blasting substitutes.
Topics: Air Pollutants, Occupational; Animals; Copper; Dust; Environmental Exposure; Granuloma; Lung Neoplas | 1988 |
[Comment on the contribution by B. Wiemann and L. Bergmann. "Development of bronchial cancer at the site of siderofibrosis"].
Topics: Air Pollutants, Occupational; Carcinoma, Bronchogenic; Chromium; Humans; Lung Neoplasms; Nickel; Occ | 1989 |
Identification of metals in lung from a patient with interstitial pneumonia.
Topics: Chromium; Cobalt; Cytological Techniques; Humans; Iron; Lung; Macrophages; Male; Metals; Methods; Mi | 1974 |
Are trace metals associated with asbestos fibers responsible for the biologic effects attributed to asbestos?
Topics: Aluminum; Animals; Asbestos; Asbestosis; Chromium; Dust; Environmental Exposure; Fibrosarcoma; Human | 1973 |
Chronic exposure to iron oxide, chromium oxide, and nickel oxide fumes of metal dressers in a steelworks.
Topics: Adolescent; Adult; Alloys; Chromium; Environmental Exposure; Humans; Iron; Male; Metallurgy; Middle | 1972 |
[On the effect of oxyfers on the lung in experiments].
Topics: Animals; Bronchitis; Dust; Iron; Lung; Male; Nickel; Pneumoconiosis; Pulmonary Fibrosis; Rats; Zinc | 1967 |
[A study of the fibrogenic action of dust formed in the processing of pig iron and nickel alloys].
Topics: Animals; Dust; Iron; Metallurgy; Nickel; Pulmonary Fibrosis; Rats; Technology | 1969 |