niacinamide has been researched along with Protein Aggregation, Pathological in 2 studies
nicotinamide : A pyridinecarboxamide that is pyridine in which the hydrogen at position 3 is replaced by a carboxamide group.
Protein Aggregation, Pathological: A biochemical phenomenon in which misfolded proteins aggregate either intra- or extracellularly. Triggered by factors such as MUTATION; POST-TRANSLATIONAL MODIFICATIONS, and environmental stress, it is generally associated with ALZHEIMER DISEASE; PARKINSON DISEASE; HUNTINGTON DISEASE; and TYPE 2 DIABETES MELLITUS.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Alzheimer's disease is a common and devastating disease characterized by aggregation of the amyloid-β peptide." | 1.46 | Enhancing mitochondrial proteostasis reduces amyloid-β proteotoxicity. ( Auwerx, J; Beck, JS; Counts, SE; D'Amico, D; Mouchiroud, L; Moullan, N; Potenza, F; Rietsch, S; Romani, M; Schmid, AW; Sorrentino, V; Zhang, H, 2017) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 1 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Hosseini, L | 1 |
| Mahmoudi, J | 1 |
| Pashazadeh, F | 1 |
| Salehi-Pourmehr, H | 1 |
| Sadigh-Eteghad, S | 1 |
| Sorrentino, V | 1 |
| Romani, M | 1 |
| Mouchiroud, L | 1 |
| Beck, JS | 1 |
| Zhang, H | 1 |
| D'Amico, D | 1 |
| Moullan, N | 1 |
| Potenza, F | 1 |
| Schmid, AW | 1 |
| Rietsch, S | 1 |
| Counts, SE | 1 |
| Auwerx, J | 1 |
1 review available for niacinamide and Protein Aggregation, Pathological
| Article | Year |
|---|---|
Protective Effects of Nicotinamide Adenine Dinucleotide and Related Precursors in Alzheimer's Disease: A Systematic Review of Preclinical Studies.
Topics: Alzheimer Disease; Amyloid beta-Peptides; Animals; Anti-Inflammatory Agents; Apoptosis; Behavior, An | 2021 |
1 other study available for niacinamide and Protein Aggregation, Pathological
| Article | Year |
|---|---|
Enhancing mitochondrial proteostasis reduces amyloid-β proteotoxicity.
Topics: Alzheimer Disease; Amyloid beta-Peptides; Animals; Caenorhabditis elegans; Disease Models, Animal; H | 2017 |