niacinamide has been researched along with Ischemic Attack, Transient in 14 studies
nicotinamide : A pyridinecarboxamide that is pyridine in which the hydrogen at position 3 is replaced by a carboxamide group.
Ischemic Attack, Transient: Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)
| Excerpt | Relevance | Reference |
|---|---|---|
| "Focal cerebral ischemia was induced by middle cerebral artery occlusion (MCAO)." | 1.37 | Proteomic identification of proteins differentially expressed by nicotinamide in focal cerebral ischemic injury. ( Kim, MO; Koh, PO; Sung, JH, 2011) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (14.29) | 18.7374 |
| 1990's | 5 (35.71) | 18.2507 |
| 2000's | 5 (35.71) | 29.6817 |
| 2010's | 2 (14.29) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Sung, JH | 1 |
| Kim, MO | 1 |
| Koh, PO | 1 |
| Chen, TY | 1 |
| Lin, MH | 1 |
| Lee, WT | 1 |
| Huang, SY | 1 |
| Chen, YH | 1 |
| Lee, AC | 1 |
| Lin, HW | 1 |
| Lee, EJ | 2 |
| Yang, J | 1 |
| Klaidman, LK | 1 |
| Nalbandian, A | 1 |
| Oliver, J | 1 |
| Chang, ML | 1 |
| Chan, PH | 1 |
| Adams, JD | 1 |
| Zhang, H | 1 |
| Cook, D | 1 |
| Matsui, T | 1 |
| Nagafuji, T | 1 |
| Tsutsumi, K | 1 |
| Uchida, H | 1 |
| Miyauchi, T | 1 |
| Asano, T | 1 |
| Kurihara, J | 1 |
| Ochiai, N | 1 |
| Kato, H | 1 |
| Germanò, A | 1 |
| Imperatore, C | 1 |
| d'Avella, D | 1 |
| Costa, G | 1 |
| Tomasello, F | 1 |
| Sun, AY | 1 |
| Cheng, JS | 1 |
| Sakakibara, Y | 2 |
| Mitha, AP | 1 |
| Ogilvy, CS | 2 |
| Maynard, KI | 4 |
| Mokudai, T | 1 |
| Ayoub, IA | 3 |
| Shen, CC | 1 |
| Harder, DR | 1 |
| Dernbach, P | 1 |
| Waters, A | 1 |
| Clerens, J | 1 |
1 review available for niacinamide and Ischemic Attack, Transient
| Article | Year |
|---|---|
Cerebral vascular smooth muscle potassium channels and their possible role in the management of vasospasm.
Topics: Adenosine Triphosphate; Animals; Benzopyrans; Calcium; Cromakalim; Electrophysiology; Guanidines; Is | 1994 |
13 other studies available for niacinamide and Ischemic Attack, Transient
| Article | Year |
|---|---|
Proteomic identification of proteins differentially expressed by nicotinamide in focal cerebral ischemic injury.
Topics: Animals; Cerebral Cortex; Chaperonin 60; Electrophoresis, Gel, Two-Dimensional; Infarction, Middle C | 2011 |
Nicotinamide inhibits nuclear factor-kappa B translocation after transient focal cerebral ischemia.
Topics: Animals; Behavior, Animal; Confidence Intervals; Disease Models, Animal; Electrophoresis, Gel, Two-D | 2012 |
The effects of nicotinamide on energy metabolism following transient focal cerebral ischemia in Wistar rats.
Topics: Adenosine Triphosphate; Animals; Brain; Cerebral Cortex; Corpus Striatum; Energy Metabolism; Ischemi | 2002 |
The effect of Nicorandil on chronic cerebral vasospasm.
Topics: Animals; Basilar Artery; Chronic Disease; Dogs; Female; Ischemic Attack, Transient; Male; Niacinamid | 1994 |
Protection by nicorandil against the dysfunction of the central vagal baroreflex system following transient global cerebral ischaemia in dogs.
Topics: Animals; Aorta, Thoracic; Blood Pressure; Cerebrovascular Circulation; Dogs; Female; Heart Rate; Isc | 1993 |
Antivasospastic and brain-protective effects of a hydroxyl radical scavenger (AVS) after experimental subarachnoid hemorrhage.
Topics: Animals; Antioxidants; Basilar Artery; Behavior, Animal; Blood-Brain Barrier; Body Weight; Brain; Br | 1998 |
Neuroprotective effects of poly (ADP-ribose) polymerase inhibitors in transient focal cerebral ischemia of rats.
Topics: ADP Ribose Transferases; Animals; Benzamides; Brain; Enzyme Inhibitors; Ischemic Attack, Transient; | 1998 |
Post-treatment with nicotinamide (vitamin B(3)) reduces the infarct volume following permanent focal cerebral ischemia in female Sprague-Dawley and Wistar rats.
Topics: Animals; Brain; Enzyme Inhibitors; Female; Injections, Intravenous; Ischemic Attack, Transient; Neur | 2000 |
Delayed treatment with nicotinamide (Vitamin B(3)) improves neurological outcome and reduces infarct volume after transient focal cerebral ischemia in Wistar rats.
Topics: Animals; Brain; Cerebral Infarction; Energy Metabolism; Ischemic Attack, Transient; Male; Motor Acti | 2000 |
Delayed multidose treatment with nicotinamide extends the degree and duration of neuroprotection by reducing infarction and improving behavioral scores up to two weeks following transient focal cerebral ischemia in Wistar rats.
Topics: Animals; Cerebral Infarction; Ischemic Attack, Transient; Male; Motor Activity; Neuroprotective Agen | 2001 |
Therapeutic window for nicotinamide following transient focal cerebral ischemia.
Topics: Animals; Behavior, Animal; Cerebral Infarction; Drug Administration Schedule; Ischemic Attack, Trans | 2002 |
Possible cellular mechanism for cerebral vasospasm after experimental subarachnoid hemorrhage in the dog.
Topics: Action Potentials; Animals; Biomechanical Phenomena; Cerebral Angiography; Dogs; Homeostasis; Ischem | 1987 |
[Problem in psychogeriatrics: quantitative and qualitative cerebral circulation insufficiencies in geriatric medicine].
Topics: Aged; Aging; Cerebrovascular Disorders; Dietary Proteins; Glucose; Hemodynamics; Humans; Infections; | 1971 |