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niacinamide and Hypertrophy, Right Ventricular

niacinamide has been researched along with Hypertrophy, Right Ventricular in 4 studies

nicotinamide : A pyridinecarboxamide that is pyridine in which the hydrogen at position 3 is replaced by a carboxamide group.

Hypertrophy, Right Ventricular: Enlargement of the RIGHT VENTRICLE of the heart. This increase in ventricular mass is often attributed to PULMONARY HYPERTENSION and is a contributor to cardiovascular morbidity and mortality.

Research Excerpts

ExcerptRelevanceReference
"The purpose of this paper was to examine the effects of nicotinamide (ND) and L-arginine (L-ARG) on pulmonary vascular and heart changes induced by pulmonary hypertension in rats in a gender-dependent way."7.96Protective effect of nicotinamide and L-arginine against monocrotaline-induced pulmonary hypertension in rats: gender dependence. ( Jankowski, Z; Kocić, I; Sztormowska-Achranowicz, K, 2020)
"Sorafenib treatment (daily gavage, 2."5.35Genomic assessment of a multikinase inhibitor, sorafenib, in a rodent model of pulmonary hypertension. ( Desai, AA; Garcia, JG; Gomberg-Maitland, M; Husain, AN; Lang, RM; Liu, Y; Lussier, YA; Maitland, ML; Moreno-Vinasco, L; Ratain, MJ; Sam, L; Sammani, S; Singleton, PA, 2008)
"The purpose of this paper was to examine the effects of nicotinamide (ND) and L-arginine (L-ARG) on pulmonary vascular and heart changes induced by pulmonary hypertension in rats in a gender-dependent way."3.96Protective effect of nicotinamide and L-arginine against monocrotaline-induced pulmonary hypertension in rats: gender dependence. ( Jankowski, Z; Kocić, I; Sztormowska-Achranowicz, K, 2020)
"Progression of pulmonary hypertension is associated with the activation of the NNMT-MNA pathway in rats and humans."1.43Activation of the nicotinamide N-methyltransferase (NNMT)-1-methylnicotinamide (MNA) pathway in pulmonary hypertension. ( Chlopicki, S; Fedorowicz, A; Jakubowski, A; Kopec, G; Kutryb-Zając, B; Mateuszuk, Ł; Skórka, T; Słomińska, E; Walczak, M; Zakrzewska, A; Łomnicka, M, 2016)
"Sorafenib treatment (daily gavage, 2."1.35Genomic assessment of a multikinase inhibitor, sorafenib, in a rodent model of pulmonary hypertension. ( Desai, AA; Garcia, JG; Gomberg-Maitland, M; Husain, AN; Lang, RM; Liu, Y; Lussier, YA; Maitland, ML; Moreno-Vinasco, L; Ratain, MJ; Sam, L; Sammani, S; Singleton, PA, 2008)

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (25.00)29.6817
2010's2 (50.00)24.3611
2020's1 (25.00)2.80

Authors

AuthorsStudies
Sztormowska-Achranowicz, K1
Jankowski, Z1
Kocić, I1
Leong, ZP1
Hikasa, Y1
Fedorowicz, A1
Mateuszuk, Ł1
Kopec, G1
Skórka, T1
Kutryb-Zając, B1
Zakrzewska, A1
Walczak, M1
Jakubowski, A1
Łomnicka, M1
Słomińska, E1
Chlopicki, S1
Moreno-Vinasco, L1
Gomberg-Maitland, M1
Maitland, ML1
Desai, AA1
Singleton, PA1
Sammani, S1
Sam, L1
Liu, Y1
Husain, AN1
Lang, RM1
Ratain, MJ1
Lussier, YA1
Garcia, JG1

Other Studies

4 other studies available for niacinamide and Hypertrophy, Right Ventricular

ArticleYear
Protective effect of nicotinamide and L-arginine against monocrotaline-induced pulmonary hypertension in rats: gender dependence.
    Pharmacological reports : PR, 2020, Volume: 72, Issue:5

    Topics: Animals; Arginine; Female; Heart Ventricles; Hypertension, Pulmonary; Hypertrophy, Right Ventricular

2020
Effects of toceranib compared with sorafenib on monocrotaline-induced pulmonary arterial hypertension and cardiopulmonary remodeling in rats.
    Vascular pharmacology, 2018, Volume: 110

    Topics: Animals; Antihypertensive Agents; Arterial Pressure; Autophagy; Disease Models, Animal; Dose-Respons

2018
Activation of the nicotinamide N-methyltransferase (NNMT)-1-methylnicotinamide (MNA) pathway in pulmonary hypertension.
    Respiratory research, 2016, 08-31, Volume: 17, Issue:1

    Topics: 6-Ketoprostaglandin F1 alpha; Adult; Animals; Case-Control Studies; Disease Models, Animal; Disease

2016
Genomic assessment of a multikinase inhibitor, sorafenib, in a rodent model of pulmonary hypertension.
    Physiological genomics, 2008, Apr-22, Volume: 33, Issue:2

    Topics: Animals; Apoptosis; Benzenesulfonates; Blotting, Western; Cell Proliferation; Complement C1q; Diseas

2008