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niacinamide and Cyst

niacinamide has been researched along with Cyst in 1 studies

nicotinamide : A pyridinecarboxamide that is pyridine in which the hydrogen at position 3 is replaced by a carboxamide group.

Research Excerpts

ExcerptRelevanceReference
"In PC2-defective cells, sorafenib inhibits B-Raf but paradoxically activates Raf-1, resulting in increased ERK1/2 phosphorylation, cell proliferation, and cyst growth in vivo."7.78Cyclic AMP/PKA-dependent paradoxical activation of Raf/MEK/ERK signaling in polycystin-2 defective mice treated with sorafenib. ( Fabris, L; Ferrero, C; Fiorotto, R; Kim, AK; Locatelli, L; Morell, CM; Okolicsanyi, S; Spirli, C; Strazzabosco, M, 2012)
"In PC2-defective cells, sorafenib inhibits B-Raf but paradoxically activates Raf-1, resulting in increased ERK1/2 phosphorylation, cell proliferation, and cyst growth in vivo."3.78Cyclic AMP/PKA-dependent paradoxical activation of Raf/MEK/ERK signaling in polycystin-2 defective mice treated with sorafenib. ( Fabris, L; Ferrero, C; Fiorotto, R; Kim, AK; Locatelli, L; Morell, CM; Okolicsanyi, S; Spirli, C; Strazzabosco, M, 2012)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Spirli, C1
Morell, CM1
Locatelli, L1
Okolicsanyi, S1
Ferrero, C1
Kim, AK1
Fabris, L1
Fiorotto, R1
Strazzabosco, M1

Other Studies

1 other study available for niacinamide and Cyst

ArticleYear
Cyclic AMP/PKA-dependent paradoxical activation of Raf/MEK/ERK signaling in polycystin-2 defective mice treated with sorafenib.
    Hepatology (Baltimore, Md.), 2012, Volume: 56, Issue:6

    Topics: Animals; Antineoplastic Agents, Hormonal; Benzenesulfonates; Bile Ducts; Caspase 3; Cell Proliferati

2012