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niacinamide and Asphyxia Neonatorum

niacinamide has been researched along with Asphyxia Neonatorum in 5 studies

nicotinamide : A pyridinecarboxamide that is pyridine in which the hydrogen at position 3 is replaced by a carboxamide group.

Asphyxia Neonatorum: Respiratory failure in the newborn. (Dorland, 27th ed)

Research Excerpts

ExcerptRelevanceReference
" After approximately one month in vitro (DIV 25), the cultures were treated for immunocytochemistry to characterise neuronal phenotype with markers against the N-methyl-D-aspartate receptor subunit 1 (NR1), the dopamine pacemaker enzyme tyrosine hydroxylase (TH), and nitric oxide synthase (NOS), the enzyme regulating the bioavailability of NO."1.33Plasticity of the central nervous system (CNS) following perinatal asphyxia: does nicotinamide provide neuroprotection? ( Bustamante, D; Goiny, M; Herrera-Marschitz, M; Klawitter, V; Morales, P, 2006)

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (20.00)29.6817
2010's4 (80.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Lespay-Rebolledo, C1
Tapia-Bustos, A1
Bustamante, D5
Morales, P5
Herrera-Marschitz, M5
Neira-Peña, T1
Rojas-Mancilla, E2
Munoz-Vio, V1
Perez, R1
Gutierrez-Hernandez, M1
Hermoso, MA1
Gebicke-Haerter, P1
Simola, N1
Lisboa, F1
Fiedler, J1
Gebicke-Haerter, PJ2
Morelli, M1
Tasker, RA1
Allende-Castro, C1
Espina-Marchant, P1
Neira, T1
Gutierrez-Hernandez, MA1
Esmar, D1
Valdes, JL1
Klawitter, V1
Goiny, M1

Other Studies

5 other studies available for niacinamide and Asphyxia Neonatorum

ArticleYear
The Long-Term Impairment in Redox Homeostasis Observed in the Hippocampus of Rats Subjected to Global Perinatal Asphyxia (PA) Implies Changes in Glutathione-Dependent Antioxidant Enzymes and TIGAR-Dependent Shift Towards the Pentose Phosphate Pathways: Ef
    Neurotoxicity research, 2019, Volume: 36, Issue:3

    Topics: Animals; Asphyxia Neonatorum; Catalase; Glutathione; Glutathione Peroxidase; Glutathione Reductase;

2019
Perinatal asphyxia leads to PARP-1 overactivity, p65 translocation, IL-1β and TNF-α overexpression, and apoptotic-like cell death in mesencephalon of neonatal rats: prevention by systemic neonatal nicotinamide administration.
    Neurotoxicity research, 2015, Volume: 27, Issue:4

    Topics: Animals; Animals, Newborn; Apoptosis; Asphyxia; Asphyxia Neonatorum; Humans; Inflammation; Interleuk

2015
Nicotinamide prevents the long-term effects of perinatal asphyxia on apoptosis, non-spatial working memory and anxiety in rats.
    Experimental brain research, 2010, Volume: 202, Issue:1

    Topics: Aging; Animals; Animals, Newborn; Anxiety; Apoptosis; Asphyxia Neonatorum; Behavior, Animal; Female;

2010
Further studies on the hypothesis of PARP-1 inhibition as a strategy for lessening the long-term effects produced by perinatal asphyxia: effects of nicotinamide and theophylline on PARP-1 activity in brain and peripheral tissue : nicotinamide and theophyl
    Neurotoxicity research, 2012, Volume: 22, Issue:1

    Topics: Animals; Animals, Newborn; Asphyxia; Asphyxia Neonatorum; Behavior, Animal; Brain; Heart; Humans; In

2012
Plasticity of the central nervous system (CNS) following perinatal asphyxia: does nicotinamide provide neuroprotection?
    Amino acids, 2006, Volume: 31, Issue:4

    Topics: Animals; Asphyxia Neonatorum; Basal Ganglia; Disease Models, Animal; Female; Humans; Infant, Newborn

2006