Page last updated: 2024-10-19

niacin and Encephalitis

niacin has been researched along with Encephalitis in 6 studies

Niacin: A water-soluble vitamin of the B complex occurring in various animal and plant tissues. It is required by the body for the formation of coenzymes NAD and NADP. It has PELLAGRA-curative, vasodilating, and antilipemic properties.
vitamin B3 : Any member of a group of vitamers that belong to the chemical structural class called pyridines that exhibit biological activity against vitamin B3 deficiency. Vitamin B3 deficiency causes a condition known as pellagra whose symptoms include depression, dermatitis and diarrhea. The vitamers include nicotinic acid and nicotinamide (and their ionized and salt forms).
nicotinic acid : A pyridinemonocarboxylic acid that is pyridine in which the hydrogen at position 3 is replaced by a carboxy group.

Encephalitis: Inflammation of the BRAIN due to infection, autoimmune processes, toxins, and other conditions. Viral infections (see ENCEPHALITIS, VIRAL) are a relatively frequent cause of this condition.

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19903 (50.00)18.7374
1990's0 (0.00)18.2507
2000's1 (16.67)29.6817
2010's1 (16.67)24.3611
2020's1 (16.67)2.80

Authors

AuthorsStudies
Ibrahim, WW1
Sayed, RH1
Kandil, EA1
Wadie, W1
Kumar, N1
Penkowa, M1
Poulsen, C1
Carrasco, J1
Hidalgo, J1
HALIKOWSKI, B1
DENYS, P1
HALLET, J1
MITCHELL, RS1

Reviews

1 review available for niacin and Encephalitis

ArticleYear
Acute and subacute encephalopathies: deficiency states (nutritional).
    Seminars in neurology, 2011, Volume: 31, Issue:2

    Topics: Acute Disease; Animals; Disease Management; Encephalitis; Humans; Malnutrition; Niacin; Thiamine Def

2011

Other Studies

5 other studies available for niacin and Encephalitis

ArticleYear
Niacin mitigates blood-brain barrier tight junctional proteins dysregulation and cerebral inflammation in ketamine rat model of psychosis: Role of GPR109A receptor.
    Progress in neuro-psychopharmacology & biological psychiatry, 2022, 12-20, Volume: 119

    Topics: Animals; Blood-Brain Barrier; Disease Models, Animal; Encephalitis; Hypolipidemic Agents; Ketamine;

2022
M-CSF deficiency leads to reduced metallothioneins I and II expression and increased tissue damage in the brain stem after 6-aminonicotinamide treatment.
    Experimental neurology, 2002, Volume: 176, Issue:2

    Topics: 6-Aminonicotinamide; Animals; Antioxidants; Apoptosis; Astrocytes; Brain Stem; Caspases; Encephaliti

2002
[Pathogenesis and course of tuberculous encephalitis and meningitis. I. Clinical types observed during the course of the disease treated with streptomycin and with isonicotinic and hydrazide].
    Gruzlica (Warsaw, Poland : 1926), 1954, Volume: 22, Issue:7

    Topics: Brain; Brain Diseases; Child; Encephalitis; Humans; Infant; Meningitis; Niacin; Nicotinic Acids; Str

1954
[Treatment for tuberculous meningo-encephalitis with the combination of streptomycine, INH and PAS (para-aminosalycilic acid)].
    Acta paediatrica Belgica, 1954, Volume: 8, Issue:3

    Topics: Aminosalicylic Acid; Encephalitis; Niacin; Nicotinic Acids; Streptomycin; Tuberculosis; Tuberculosis

1954
Fatal toxic encephalitis occurring during iproniazid therapy in pulmonary tuberculosis.
    Annals of internal medicine, 1955, Volume: 42, Issue:2

    Topics: Encephalitis; Iproniazid; Neurotoxicity Syndromes; Niacin; Nicotinic Acids; Tuberculosis; Tuberculos

1955