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ng-nitroarginine methyl ester and Motor Neuron Disease

ng-nitroarginine methyl ester has been researched along with Motor Neuron Disease in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (66.67)18.2507
2000's1 (33.33)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Angelini, DF; Carrì, MT; Casciati, A; Cozzolino, M; Ferri, A; Longone, P; Nencini, M; Rotilio, G; Spalloni, A1
Ikeda, K; Iwasaki, Y; Kinoshita, M1
Kruman, II; Mattson, MP; Pedersen, WA; Springer, JE1

Other Studies

3 other study(ies) available for ng-nitroarginine methyl ester and Motor Neuron Disease

ArticleYear
Cell death in amyotrophic lateral sclerosis: interplay between neuronal and glial cells.
    FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 2004, Volume: 18, Issue:11

    Topics: Animals; Apoptosis; Caspase 1; Catalase; Cell Communication; Coculture Techniques; Cytokines; Enzyme Activation; Enzyme Inhibitors; Glioblastoma; Humans; Interferon-gamma; Lipopolysaccharides; Mice; Mice, Transgenic; Motor Neuron Disease; Motor Neurons; Neuroblastoma; Neuroglia; Neurons; NG-Nitroarginine Methyl Ester; Nitric Oxide Donors; Nitric Oxide Synthase; Nitroso Compounds; Oxidative Stress; Reactive Oxygen Species; Spinal Cord; Superoxide Dismutase; Superoxide Dismutase-1; Transfection

2004
Neuronal nitric oxide synthase inhibitor, 7-nitroindazole, delays motor dysfunction and spinal motoneuron degeneration in the wobbler mouse.
    Journal of the neurological sciences, 1998, Sep-18, Volume: 160, Issue:1

    Topics: Amyotrophic Lateral Sclerosis; Animals; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Evaluation, Preclinical; Enzyme Inhibitors; Hand Strength; Indazoles; Mice; Mice, Neurologic Mutants; Motor Neuron Disease; Motor Neurons; Muscle Denervation; Muscle, Skeletal; Nerve Tissue Proteins; NG-Nitroarginine Methyl Ester; Nitric Oxide Synthase; Nitric Oxide Synthase Type I; Organ Size; Vacuoles

1998
ALS-linked Cu/Zn-SOD mutation increases vulnerability of motor neurons to excitotoxicity by a mechanism involving increased oxidative stress and perturbed calcium homeostasis.
    Experimental neurology, 1999, Volume: 160, Issue:1

    Topics: 6-Cyano-7-nitroquinoxaline-2,3-dione; alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Antioxidants; Calcium; Cells, Cultured; Cyclic N-Oxides; Estradiol; Excitatory Amino Acid Agonists; Excitatory Amino Acid Antagonists; Fluorescent Dyes; Free Radicals; Glutamic Acid; Homeostasis; Humans; Imidazoles; Lipid Peroxidation; Mice; Mice, Transgenic; Mitochondria; Motor Neuron Disease; Motor Neurons; Neurotoxins; NG-Nitroarginine Methyl Ester; Nitrates; Oxidative Stress; Rhodamine 123; Spinal Cord; Superoxide Dismutase; Superoxides; Vitamin E

1999