ng-nitroarginine methyl ester has been researched along with Marfan Syndrome in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (50.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Alberca, R; Briones, AM; Campanero, MR; Canelas, LI; De Backer, J; Evangelista, A; Hurlé, MA; Jiménez-Borreguero, LJ; Lozano-Vidal, N; Méndez-Barbero, N; Milewicz, D; Nistal, JF; Oller, J; Redondo, JM; Renard, M; Ruiz, EJ; Salaices, M; Villahoz, S | 1 |
Au Yeung, K; Chung, AW; Cortes, SF; Dietz, HC; Judge, DP; Sandor, GG; van Breemen, C | 1 |
2 other study(ies) available for ng-nitroarginine methyl ester and Marfan Syndrome
Article | Year |
---|---|
Nitric oxide mediates aortic disease in mice deficient in the metalloprotease Adamts1 and in a mouse model of Marfan syndrome.
Topics: ADAMTS1 Protein; Adult; Aged; Animals; Aorta; Aortic Aneurysm; Aortic Aneurysm, Thoracic; Aortic Dissection; Disease Models, Animal; Enzyme Inhibitors; Female; Fibrillin-1; Gene Knockdown Techniques; Haploinsufficiency; Humans; Immunoblotting; Male; Marfan Syndrome; Mice; Middle Aged; NG-Nitroarginine Methyl Ester; Nitric Oxide; Nitric Oxide Synthase Type II; Real-Time Polymerase Chain Reaction | 2017 |
Endothelial dysfunction and compromised eNOS/Akt signaling in the thoracic aorta during the progression of Marfan syndrome.
Topics: Acetylcholine; Age Factors; Animals; Aorta, Abdominal; Aorta, Thoracic; Calcium; Cyclic GMP; Disease Models, Animal; Disease Progression; Endothelium, Vascular; Enzyme Inhibitors; Fibrillin-1; Fibrillins; Marfan Syndrome; Mice; Mice, Inbred C57BL; Mice, Transgenic; Microfilament Proteins; NG-Nitroarginine Methyl Ester; Nitric Oxide; Nitric Oxide Synthase Type II; Nitric Oxide Synthase Type III; Phosphorylation; Proto-Oncogene Proteins c-akt; Signal Transduction; Vasodilation; Vasodilator Agents | 2007 |