nafenopin has been researched along with Carcinoma, Hepatocellular in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (33.33) | 18.7374 |
| 1990's | 3 (50.00) | 18.2507 |
| 2000's | 1 (16.67) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chabicovsky, M; Grasl-Kraupp, B; Peter, B; Rossmanith, W; Schausberger, E; Schulte-Hermann, R | 1 |
| Ashby, J; James, NH; Roberts, RA | 1 |
| Bursch, W; Grasl-Kraupp, B; Huber, W; Müllauer, L; Ruttkay-Nedecky, B; Schulte-Hermann, R; Taper, H | 1 |
| Chevalier, S; Roberts, RA | 1 |
| Moody, DE; Rao, S; Reddy, JK | 1 |
| Rao, MS; Reddy, JK | 1 |
6 other study(ies) available for nafenopin and Carcinoma, Hepatocellular
| Article | Year |
|---|---|
Follistatin overexpression in rodent liver tumors: a possible mechanism to overcome activin growth control.
Topics: Activins; Adenoma; Alternative Splicing; Animals; Blotting, Western; Carcinoma, Hepatocellular; Cell Division; Diethylnitrosamine; Follistatin; Gene Expression Regulation, Neoplastic; Humans; Liver Neoplasms; Male; Mice; Mice, Inbred Strains; Nafenopin; Phenobarbital; Rats; Reference Values; RNA, Messenger; Transforming Growth Factor alpha; Tumor Cells, Cultured | 2002 |
Enhanced hepatocyte colony growth in soft agar after in vivo treatment with a genotoxic carcinogen: a potential assay for hepatocarcinogens?
Topics: Agar; Animals; Carcinogenicity Tests; Carcinoma, Hepatocellular; Cells, Cultured; Contact Inhibition; Diethylnitrosamine; Dose-Response Relationship, Drug; Drug Synergism; Epidermal Growth Factor; gamma-Glutamyltransferase; Gene Expression Regulation, Neoplastic; Liver Neoplasms; Male; Nafenopin; Rats; Rats, Wistar; Tumor Stem Cell Assay | 1995 |
Inherent increase of apoptosis in liver tumors: implications for carcinogenesis and tumor regression.
Topics: Adenoma, Liver Cell; Animals; Apoptosis; Carcinogens; Carcinoma, Hepatocellular; Cell Division; DNA, Neoplasm; Humans; Liver; Liver Neoplasms; Liver Neoplasms, Experimental; Male; Nafenopin; Precancerous Conditions; Rats; Rats, Wistar; Signal Transduction | 1997 |
G1-arrested FaO cells re-enter the cell cycle upon stimulation with the rodent non-genotoxic hepatocarcinogen nafenopin.
Topics: Animals; Butyrates; Carcinogens; Carcinoma, Hepatocellular; CDC2-CDC28 Kinases; Cell Cycle Proteins; Cyclin D1; Cyclin E; Cyclin-Dependent Kinase 2; Cyclin-Dependent Kinase 4; Cyclin-Dependent Kinase Inhibitor p27; Cyclin-Dependent Kinases; Down-Regulation; Enzyme Inhibitors; Epidermal Growth Factor; G1 Phase; Microtubule-Associated Proteins; Nafenopin; Phosphorylation; Protein Serine-Threonine Kinases; Proto-Oncogene Proteins; Rats; Time Factors; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha; Tumor Suppressor Proteins | 1999 |
Hepatocellular carcinomas in acatalasemic mice treated with nafenopin, a hypolipidemic peroxisome proliferator.
Topics: Acatalasia; Animals; Carcinoma, Hepatocellular; DNA, Neoplasm; Female; Liver Neoplasms; Lung Neoplasms; Male; Mice; Microbodies; Nafenopin; Neoplasm Metastasis; Neoplasms, Experimental; Propionates | 1976 |
Malignant tumors in rats fed nafenopin, a hepatic peroxisome proliferator.
Topics: Animals; Carcinogens; Carcinoma, Hepatocellular; Liver; Liver Neoplasms; Male; Microbodies; Nafenopin; Neoplasms, Experimental; Pancreatic Neoplasms; Propionates; Rats; Rats, Inbred F344; Testicular Neoplasms | 1977 |