nadp has been researched along with Cardiomyopathies in 14 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 3 (21.43) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (21.43) | 29.6817 |
| 2010's | 8 (57.14) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| de Souza Gomes, R; Monteiro, MC; Monteiro, VVS; Navegantes, KC; Reis, JF | 1 |
| Benjamin, IJ; Bernstein, SI; Cammarato, A; Golic, KG; Lin, HC; Rajasekaran, NS; Suggs, JA; Xie, HB; Zhang, H | 1 |
| Amaral, AU; Busanello, EN; Gasparotto, J; Gelain, DP; Gregersen, N; Tonin, AM; Wajner, M | 1 |
| Li, F; Meng, R; Sun, X; Wang, J; Yang, Z; Zhang, A | 1 |
| Amaral, AU; Cecatto, C; Hickmann, FH; Rodrigues, MD; Wajner, M | 1 |
| Amaral, AU; Cecatto, C; da Silva, JC; Godoy, KDS; Wajner, M | 1 |
| De Mello, WC | 1 |
| Cai, L; Hein, DW; Li, X; Marshall, JP; Prabhu, SD; Xiang, X; Zhou, G | 1 |
| Brown, BH; Galvao, TF; Gupte, SA; Hecker, PA; Henderson, R; O'Shea, KM; Riggle, H; Stanley, WC | 1 |
| Filho, CS; Grings, M; Knebel, LA; Leipnitz, G; Moura, AP; Tonin, AM; Wajner, M; Zanatta, A | 1 |
| Greenberg, ML; Gu, Z; Ma, L; Vaz, FM; Wanders, RJ | 1 |
| Chekman, IS | 1 |
| Fedosenko, NG; Klering, PG; Krasovitskiĭ, AI | 1 |
| Burch, GE; Ferrans, VJ; Hibbs, RG; Walsh, JJ; Weilbaecher, DG; Weily, HS | 1 |
1 review(s) available for nadp and Cardiomyopathies
| Article | Year |
|---|---|
Dual Behavior of Exosomes in Septic Cardiomyopathy.
Topics: Animals; Cardiomyopathies; Exosomes; Host-Pathogen Interactions; Humans; MicroRNAs; Myocardium; NADP; Sepsis; Signal Transduction | 2017 |
13 other study(ies) available for nadp and Cardiomyopathies
| Article | Year |
|---|---|
The NADPH metabolic network regulates human αB-crystallin cardiomyopathy and reductive stress in Drosophila melanogaster.
Topics: alpha-Crystallin B Chain; Animals; Cardiomyopathies; Cataract; Drosophila melanogaster; Glucosephosphate Dehydrogenase; Humans; Isocitrate Dehydrogenase; Malate Dehydrogenase; Metabolic Networks and Pathways; Mice; Muscular Diseases; Mutation, Missense; NADP; Phosphogluconate Dehydrogenase | 2013 |
Mitochondrial bioenergetics deregulation caused by long-chain 3-hydroxy fatty acids accumulating in LCHAD and MTP deficiencies in rat brain: a possible role of mPTP opening as a pathomechanism in these disorders?
Topics: 3-Hydroxyacyl CoA Dehydrogenases; Acyl-CoA Dehydrogenase, Long-Chain; Adenosine Triphosphate; Animals; Calcium; Cardiomyopathies; Cerebral Cortex; Cytochromes c; Energy Metabolism; Homeostasis; Hydrogen Peroxide; Lauric Acids; Lipid Metabolism, Inborn Errors; Membrane Potential, Mitochondrial; Mitochondria; Mitochondrial Membrane Transport Proteins; Mitochondrial Myopathies; Mitochondrial Permeability Transition Pore; Mitochondrial Swelling; Mitochondrial Trifunctional Protein; Myristic Acids; NADP; Nervous System Diseases; Oxidants; Palmitic Acids; Rats; Rats, Wistar; Rhabdomyolysis | 2014 |
[The changes of LCHAD in preeclampsia with different clinical features and the correlation with NADPH P47-phox, p38MAPK-α, COX-2 and serum FFA and TG].
Topics: 3-Hydroxyacyl CoA Dehydrogenases; Cardiomyopathies; Cohort Studies; Cyclooxygenase 2; Fatty Acids; Female; Humans; Hypertension; Lipid Metabolism, Inborn Errors; Mitochondrial Myopathies; Mitochondrial Trifunctional Protein; NADP; NADPH Oxidases; Nervous System Diseases; Oxidation-Reduction; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Placenta; Pre-Eclampsia; Pregnancy; Pregnancy Trimester, Third; Prospective Studies; Rhabdomyolysis; RNA, Messenger; Triglycerides | 2015 |
Deregulation of mitochondrial functions provoked by long-chain fatty acid accumulating in long-chain 3-hydroxyacyl-CoA dehydrogenase and mitochondrial permeability transition deficiencies in rat heart--mitochondrial permeability transition pore opening as
Topics: Adenosine Triphosphate; Animals; Calcium Channel Blockers; Calcium Signaling; Cardiomyopathies; Cell Membrane Permeability; Enzyme Inhibitors; Humans; Lipid Metabolism, Inborn Errors; Long-Chain-3-Hydroxyacyl-CoA Dehydrogenase; Membrane Potential, Mitochondrial; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Membranes; Mitochondrial Myopathies; Mitochondrial Permeability Transition Pore; Mitochondrial Swelling; Mitochondrial Trifunctional Protein; Myristic Acids; NADP; Nervous System Diseases; Organ Specificity; Oxidative Phosphorylation; Palmitic Acids; Rats, Wistar; Rhabdomyolysis | 2015 |
Disturbance of mitochondrial functions provoked by the major long-chain 3-hydroxylated fatty acids accumulating in MTP and LCHAD deficiencies in skeletal muscle.
Topics: Animals; Calcium; Cardiomyopathies; Lipid Metabolism, Inborn Errors; Membrane Potential, Mitochondrial; Mitochondria, Muscle; Mitochondrial Myopathies; Mitochondrial Trifunctional Protein; Muscle, Skeletal; Myristic Acids; NADP; Nervous System Diseases; Oxygen Consumption; Palmitic Acids; Rats, Wistar; Rhabdomyolysis | 2016 |
Metallothionein reverses the harmful effects of angiotensin II on the diabetic heart.
Topics: Angiotensin II; Cardiomyopathies; Diabetic Angiopathies; Humans; Metallothionein; NADP; Oxidative Stress | 2008 |
Metallothionein suppresses angiotensin II-induced nicotinamide adenine dinucleotide phosphate oxidase activation, nitrosative stress, apoptosis, and pathological remodeling in the diabetic heart.
Topics: Angiotensin II; Animals; Apoptosis; Cardiomyopathies; Diabetes Mellitus, Experimental; Diabetic Angiopathies; Fibrosis; Hypertrophy; Metallothionein; Mice; Mice, Transgenic; Myocardium; Myocytes, Cardiac; NADP; Oxidative Stress; Ventricular Remodeling | 2008 |
High-sugar intake does not exacerbate metabolic abnormalities or cardiac dysfunction in genetic cardiomyopathy.
Topics: Animals; Cardiomyopathies; Cricetinae; Dietary Sucrose; Echocardiography; Energy Intake; Fructose; Glucosephosphate Dehydrogenase; Glutathione; Heart; Humans; Lipid Peroxidation; Lipids; Male; NADP; Oxidative Stress; Reactive Oxygen Species; Sarcoglycans; Starch | 2012 |
Phytanic acid disturbs mitochondrial homeostasis in heart of young rats: a possible pathomechanism of cardiomyopathy in Refsum disease.
Topics: Animals; Antioxidants; Cardiomyopathies; Chromans; Electron Transport Chain Complex Proteins; Glutathione; Homeostasis; In Vitro Techniques; Male; Membrane Potential, Mitochondrial; Mitochondria, Heart; Myocardium; NADP; NG-Nitroarginine Methyl Ester; Nitric Oxide Synthase; Oxidation-Reduction; Oxidative Stress; Phytanic Acid; Protein Carbonylation; Rats; Rats, Wistar; Refsum Disease; Thiobarbituric Acid Reactive Substances | 2012 |
The human TAZ gene complements mitochondrial dysfunction in the yeast taz1Delta mutant. Implications for Barth syndrome.
Topics: Acyltransferases; Adenosine Triphosphate; Alamethicin; Alternative Splicing; Cardiolipins; Cardiomyopathies; Cell Membrane; Cloning, Molecular; Cytosol; DNA; Ethanol; Exons; Genetic Complementation Test; Humans; Immunoblotting; Mitochondria; Mutation; NAD; NADP; Open Reading Frames; Oxygen; Oxygen Consumption; Phosphorylation; Proteins; Saccharomyces cerevisiae; Subcellular Fractions; Substrate Specificity; Syndrome; Temperature; Transcription Factors | 2004 |
[Biochemical pharmacology of cardiac glycosides].
Topics: Adenine Nucleotides; Animals; Cardiac Glycosides; Cardiomyopathies; Energy Metabolism; Heart; Ion Channels; Myocardium; NAD; NADP; Rats | 1982 |
[Changes in the myocardium damaged by strophanthin].
Topics: Adenosine Triphosphate; Animals; Cardiomyopathies; L-Lactate Dehydrogenase; Mitochondria, Heart; Myocardium; NADP; Rats; Sarcoplasmic Reticulum; Strophanthins; Succinate Dehydrogenase | 1989 |
A histochemical and electron microscopic study of epinephrine-induced myocardial necrosis.
Topics: Animals; Cardiomyopathies; Dihydrolipoamide Dehydrogenase; Electron Transport Complex IV; Endoplasmic Reticulum; Epinephrine; Glycogen; Heart; Histocytochemistry; Lipids; Male; Microscopy, Electron; Mitochondria, Muscle; Myocardial Infarction; Myocardium; Myofibrils; NAD; NADP; Necrosis; Rats; Succinate Dehydrogenase; Transferases | 1970 |