Page last updated: 2024-08-17

nadp and Cardiomyopathies

nadp has been researched along with Cardiomyopathies in 14 studies

Research

Studies (14)

TimeframeStudies, this research(%)All Research%
pre-19903 (21.43)18.7374
1990's0 (0.00)18.2507
2000's3 (21.43)29.6817
2010's8 (57.14)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
de Souza Gomes, R; Monteiro, MC; Monteiro, VVS; Navegantes, KC; Reis, JF1
Benjamin, IJ; Bernstein, SI; Cammarato, A; Golic, KG; Lin, HC; Rajasekaran, NS; Suggs, JA; Xie, HB; Zhang, H1
Amaral, AU; Busanello, EN; Gasparotto, J; Gelain, DP; Gregersen, N; Tonin, AM; Wajner, M1
Li, F; Meng, R; Sun, X; Wang, J; Yang, Z; Zhang, A1
Amaral, AU; Cecatto, C; Hickmann, FH; Rodrigues, MD; Wajner, M1
Amaral, AU; Cecatto, C; da Silva, JC; Godoy, KDS; Wajner, M1
De Mello, WC1
Cai, L; Hein, DW; Li, X; Marshall, JP; Prabhu, SD; Xiang, X; Zhou, G1
Brown, BH; Galvao, TF; Gupte, SA; Hecker, PA; Henderson, R; O'Shea, KM; Riggle, H; Stanley, WC1
Filho, CS; Grings, M; Knebel, LA; Leipnitz, G; Moura, AP; Tonin, AM; Wajner, M; Zanatta, A1
Greenberg, ML; Gu, Z; Ma, L; Vaz, FM; Wanders, RJ1
Chekman, IS1
Fedosenko, NG; Klering, PG; Krasovitskiĭ, AI1
Burch, GE; Ferrans, VJ; Hibbs, RG; Walsh, JJ; Weilbaecher, DG; Weily, HS1

Reviews

1 review(s) available for nadp and Cardiomyopathies

ArticleYear
Dual Behavior of Exosomes in Septic Cardiomyopathy.
    Advances in experimental medicine and biology, 2017, Volume: 998

    Topics: Animals; Cardiomyopathies; Exosomes; Host-Pathogen Interactions; Humans; MicroRNAs; Myocardium; NADP; Sepsis; Signal Transduction

2017

Other Studies

13 other study(ies) available for nadp and Cardiomyopathies

ArticleYear
The NADPH metabolic network regulates human αB-crystallin cardiomyopathy and reductive stress in Drosophila melanogaster.
    PLoS genetics, 2013, Volume: 9, Issue:6

    Topics: alpha-Crystallin B Chain; Animals; Cardiomyopathies; Cataract; Drosophila melanogaster; Glucosephosphate Dehydrogenase; Humans; Isocitrate Dehydrogenase; Malate Dehydrogenase; Metabolic Networks and Pathways; Mice; Muscular Diseases; Mutation, Missense; NADP; Phosphogluconate Dehydrogenase

2013
Mitochondrial bioenergetics deregulation caused by long-chain 3-hydroxy fatty acids accumulating in LCHAD and MTP deficiencies in rat brain: a possible role of mPTP opening as a pathomechanism in these disorders?
    Biochimica et biophysica acta, 2014, Volume: 1842, Issue:9

    Topics: 3-Hydroxyacyl CoA Dehydrogenases; Acyl-CoA Dehydrogenase, Long-Chain; Adenosine Triphosphate; Animals; Calcium; Cardiomyopathies; Cerebral Cortex; Cytochromes c; Energy Metabolism; Homeostasis; Hydrogen Peroxide; Lauric Acids; Lipid Metabolism, Inborn Errors; Membrane Potential, Mitochondrial; Mitochondria; Mitochondrial Membrane Transport Proteins; Mitochondrial Myopathies; Mitochondrial Permeability Transition Pore; Mitochondrial Swelling; Mitochondrial Trifunctional Protein; Myristic Acids; NADP; Nervous System Diseases; Oxidants; Palmitic Acids; Rats; Rats, Wistar; Rhabdomyolysis

2014
[The changes of LCHAD in preeclampsia with different clinical features and the correlation with NADPH P47-phox, p38MAPK-α, COX-2 and serum FFA and TG].
    Zhonghua fu chan ke za zhi, 2015, Volume: 50, Issue:2

    Topics: 3-Hydroxyacyl CoA Dehydrogenases; Cardiomyopathies; Cohort Studies; Cyclooxygenase 2; Fatty Acids; Female; Humans; Hypertension; Lipid Metabolism, Inborn Errors; Mitochondrial Myopathies; Mitochondrial Trifunctional Protein; NADP; NADPH Oxidases; Nervous System Diseases; Oxidation-Reduction; Oxidative Stress; p38 Mitogen-Activated Protein Kinases; Placenta; Pre-Eclampsia; Pregnancy; Pregnancy Trimester, Third; Prospective Studies; Rhabdomyolysis; RNA, Messenger; Triglycerides

2015
Deregulation of mitochondrial functions provoked by long-chain fatty acid accumulating in long-chain 3-hydroxyacyl-CoA dehydrogenase and mitochondrial permeability transition deficiencies in rat heart--mitochondrial permeability transition pore opening as
    The FEBS journal, 2015, Volume: 282, Issue:24

    Topics: Adenosine Triphosphate; Animals; Calcium Channel Blockers; Calcium Signaling; Cardiomyopathies; Cell Membrane Permeability; Enzyme Inhibitors; Humans; Lipid Metabolism, Inborn Errors; Long-Chain-3-Hydroxyacyl-CoA Dehydrogenase; Membrane Potential, Mitochondrial; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Membranes; Mitochondrial Myopathies; Mitochondrial Permeability Transition Pore; Mitochondrial Swelling; Mitochondrial Trifunctional Protein; Myristic Acids; NADP; Nervous System Diseases; Organ Specificity; Oxidative Phosphorylation; Palmitic Acids; Rats, Wistar; Rhabdomyolysis

2015
Disturbance of mitochondrial functions provoked by the major long-chain 3-hydroxylated fatty acids accumulating in MTP and LCHAD deficiencies in skeletal muscle.
    Toxicology in vitro : an international journal published in association with BIBRA, 2016, Volume: 36

    Topics: Animals; Calcium; Cardiomyopathies; Lipid Metabolism, Inborn Errors; Membrane Potential, Mitochondrial; Mitochondria, Muscle; Mitochondrial Myopathies; Mitochondrial Trifunctional Protein; Muscle, Skeletal; Myristic Acids; NADP; Nervous System Diseases; Oxygen Consumption; Palmitic Acids; Rats, Wistar; Rhabdomyolysis

2016
Metallothionein reverses the harmful effects of angiotensin II on the diabetic heart.
    Journal of the American College of Cardiology, 2008, Aug-19, Volume: 52, Issue:8

    Topics: Angiotensin II; Cardiomyopathies; Diabetic Angiopathies; Humans; Metallothionein; NADP; Oxidative Stress

2008
Metallothionein suppresses angiotensin II-induced nicotinamide adenine dinucleotide phosphate oxidase activation, nitrosative stress, apoptosis, and pathological remodeling in the diabetic heart.
    Journal of the American College of Cardiology, 2008, Aug-19, Volume: 52, Issue:8

    Topics: Angiotensin II; Animals; Apoptosis; Cardiomyopathies; Diabetes Mellitus, Experimental; Diabetic Angiopathies; Fibrosis; Hypertrophy; Metallothionein; Mice; Mice, Transgenic; Myocardium; Myocytes, Cardiac; NADP; Oxidative Stress; Ventricular Remodeling

2008
High-sugar intake does not exacerbate metabolic abnormalities or cardiac dysfunction in genetic cardiomyopathy.
    Nutrition (Burbank, Los Angeles County, Calif.), 2012, Volume: 28, Issue:5

    Topics: Animals; Cardiomyopathies; Cricetinae; Dietary Sucrose; Echocardiography; Energy Intake; Fructose; Glucosephosphate Dehydrogenase; Glutathione; Heart; Humans; Lipid Peroxidation; Lipids; Male; NADP; Oxidative Stress; Reactive Oxygen Species; Sarcoglycans; Starch

2012
Phytanic acid disturbs mitochondrial homeostasis in heart of young rats: a possible pathomechanism of cardiomyopathy in Refsum disease.
    Molecular and cellular biochemistry, 2012, Volume: 366, Issue:1-2

    Topics: Animals; Antioxidants; Cardiomyopathies; Chromans; Electron Transport Chain Complex Proteins; Glutathione; Homeostasis; In Vitro Techniques; Male; Membrane Potential, Mitochondrial; Mitochondria, Heart; Myocardium; NADP; NG-Nitroarginine Methyl Ester; Nitric Oxide Synthase; Oxidation-Reduction; Oxidative Stress; Phytanic Acid; Protein Carbonylation; Rats; Rats, Wistar; Refsum Disease; Thiobarbituric Acid Reactive Substances

2012
The human TAZ gene complements mitochondrial dysfunction in the yeast taz1Delta mutant. Implications for Barth syndrome.
    The Journal of biological chemistry, 2004, Oct-22, Volume: 279, Issue:43

    Topics: Acyltransferases; Adenosine Triphosphate; Alamethicin; Alternative Splicing; Cardiolipins; Cardiomyopathies; Cell Membrane; Cloning, Molecular; Cytosol; DNA; Ethanol; Exons; Genetic Complementation Test; Humans; Immunoblotting; Mitochondria; Mutation; NAD; NADP; Open Reading Frames; Oxygen; Oxygen Consumption; Phosphorylation; Proteins; Saccharomyces cerevisiae; Subcellular Fractions; Substrate Specificity; Syndrome; Temperature; Transcription Factors

2004
[Biochemical pharmacology of cardiac glycosides].
    Vestnik Akademii meditsinskikh nauk SSSR, 1982, Issue:5

    Topics: Adenine Nucleotides; Animals; Cardiac Glycosides; Cardiomyopathies; Energy Metabolism; Heart; Ion Channels; Myocardium; NAD; NADP; Rats

1982
[Changes in the myocardium damaged by strophanthin].
    Arkhiv anatomii, gistologii i embriologii, 1989, Volume: 96, Issue:5

    Topics: Adenosine Triphosphate; Animals; Cardiomyopathies; L-Lactate Dehydrogenase; Mitochondria, Heart; Myocardium; NADP; Rats; Sarcoplasmic Reticulum; Strophanthins; Succinate Dehydrogenase

1989
A histochemical and electron microscopic study of epinephrine-induced myocardial necrosis.
    Journal of molecular and cellular cardiology, 1970, Volume: 1, Issue:1

    Topics: Animals; Cardiomyopathies; Dihydrolipoamide Dehydrogenase; Electron Transport Complex IV; Endoplasmic Reticulum; Epinephrine; Glycogen; Heart; Histocytochemistry; Lipids; Male; Microscopy, Electron; Mitochondria, Muscle; Myocardial Infarction; Myocardium; Myofibrils; NAD; NADP; Necrosis; Rats; Succinate Dehydrogenase; Transferases

1970