nadp has been researched along with Cancer of Nasopharynx in 8 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 8 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Hou, H; Lan, F; Li, D; Li, J; Li, Z; Liang, D; Su, Z; Tian, W; Wang, C | 1 |
| Dai, Z; Fang, W; Fu, H; He, Z; Huang, G; Li, B; Qin, D; Tian, L; Wang, H; Yang, H; Zhang, X; Zhu, B | 1 |
| Hou, H; Li, D; Li, J; Mo, C; Mo, Y; Pan, Z; Wang, C | 1 |
| Dong, Y; Wang, M | 1 |
| Chan, AT; Cheng, SH; Cheung, CS; Ho, K; Hong, B; Lau, CP; Lei, KI; Lui, VW; Mita, A; Ng, MH; Tsang, CM; Tsao, SW; Yamasaki, Y | 1 |
| Fu, L; Hu, W; Liang, X | 1 |
| Chan, AT; Cheng, SH; Ho, K; Hong, B; Lam, EK; Lau, CP; Lo, KW; Lui, VW; Mills, GB; Mok, TS; Ng, MH; Ng, PK; Ng, YK; Tsang, CM; Tsao, SW; Tsui, SK; Wong, EY | 1 |
| Lian, YF; Qian, CN; Wu, MS; Ye, HB; Zeng, YX; Zheng, FJ | 1 |
8 other study(ies) available for nadp and Cancer of Nasopharynx
| Article | Year |
|---|---|
A novel Rhein derivative: Activation of Rac1/NADPH pathway enhances sensitivity of nasopharyngeal carcinoma cells to radiotherapy.
Topics: Anthraquinones; Cell Line, Tumor; Cell Proliferation; Cell Survival; Humans; NADP; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; rac1 GTP-Binding Protein; Radiation Tolerance; Radiation-Sensitizing Agents | 2019 |
Deuterium-depleted water (DDW) inhibits the proliferation and migration of nasopharyngeal carcinoma cells in vitro.
Topics: Animals; Carcinoma; Cell Cycle Checkpoints; Cell Line; Cell Line, Tumor; Cell Movement; Cell Proliferation; Deuterium; G1 Phase; Humans; Matrix Metalloproteinase 9; Mice; NAD(P)H Dehydrogenase (Quinone); NADP; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; S Phase; Water | 2013 |
The Enhancement of Radiation Sensitivity in Nasopharyngeal Carcinoma Cells via Activation of the Rac1/NADPH Signaling Pathway.
Topics: Aminoquinolines; Apoptosis; Carcinoma; Cell Line, Tumor; Enzyme Activation; Gene Expression Regulation, Neoplastic; Humans; Intracellular Space; NADP; NADPH Oxidases; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; NF-kappa B; Phosphoproteins; Protein Transport; Pyrimidines; rac1 GTP-Binding Protein; Radiation Tolerance; Reactive Oxygen Species; Signal Transduction; Tetradecanoylphorbol Acetate; X-Rays | 2016 |
Knockdown of TKTL1 additively complements cisplatin-induced cytotoxicity in nasopharyngeal carcinoma cells by regulating the levels of NADPH and ribose-5-phosphate.
Topics: Apoptosis; Carcinoma; Cell Line, Tumor; Cell Survival; Cisplatin; Comet Assay; Gene Expression Regulation; Gene Knockdown Techniques; Humans; NADP; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; Ribosemonophosphates; Transketolase | 2017 |
An RNA-directed nucleoside anti-metabolite, 1-(3-C-ethynyl-beta-d-ribo-pentofuranosyl)cytosine (ECyd), elicits antitumor effect via TP53-induced Glycolysis and Apoptosis Regulator (TIGAR) downregulation.
Topics: Apoptosis; Apoptosis Regulatory Proteins; Cell Cycle; Cell Line, Tumor; Cell Proliferation; Cytidine; Dose-Response Relationship, Drug; Down-Regulation; Humans; Inhibitory Concentration 50; Intracellular Signaling Peptides and Proteins; NADP; Nasopharyngeal Neoplasms; Phosphoric Monoester Hydrolases | 2010 |
Pulse compression in two-photon excitation fluorescence microscopy.
Topics: Cell Line, Tumor; Humans; Imaging, Three-Dimensional; Microscopy, Fluorescence; NADP; Nasopharyngeal Neoplasms; Nonlinear Dynamics; Photons | 2010 |
Inhibition of c-Met downregulates TIGAR expression and reduces NADPH production leading to cell death.
Topics: Apoptosis; Apoptosis Regulatory Proteins; Cell Line, Tumor; Cell Proliferation; Cell Survival; Down-Regulation; Humans; Indoles; Intracellular Signaling Peptides and Proteins; NADP; Nasopharyngeal Neoplasms; Phosphoric Monoester Hydrolases; Phosphorylation; Piperazines; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-met; Pyrimidinones; Quinolines; Signal Transduction; Sulfonamides | 2011 |
Repressing malic enzyme 1 redirects glucose metabolism, unbalances the redox state, and attenuates migratory and invasive abilities in nasopharyngeal carcinoma cell lines.
Topics: Carcinoma; Cell Line, Tumor; Cell Movement; Cell Survival; Glucose; Glycolysis; Humans; Malate Dehydrogenase; NADP; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; Neoplasm Invasiveness; Oxidation-Reduction; Oxidative Phosphorylation; Pentose Phosphate Pathway; Proto-Oncogene Proteins c-akt | 2012 |