nadifloxacin has been researched along with Dermatitis, Atopic in 1 studies
nadifloxacin: (R)-isomer does not induce chromosomal aberrations, unlike (S)-isomer; structure given in first source
Dermatitis, Atopic: A chronic inflammatory genetically determined disease of the skin marked by increased ability to form reagin (IgE), with increased susceptibility to allergic rhinitis and asthma, and hereditary disposition to a lowered threshold for pruritus. It is manifested by lichenification, excoriation, and crusting, mainly on the flexural surfaces of the elbow and knee. In infants it is known as infantile eczema.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (100.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kimata, H | 1 |
1 trial available for nadifloxacin and Dermatitis, Atopic
| Article | Year |
|---|---|
Effect of nadifloxacin on atopic dermatitis with methicillin-resistant Staphylococcus aureus in young children.
Topics: Administration, Topical; Anti-Infective Agents; Anti-Inflammatory Agents; Bufexamac; Dermatitis, Ato | 1999 |