nad has been researched along with Injury, Myocardial Reperfusion in 76 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 18 (23.68) | 18.2507 |
2000's | 23 (30.26) | 29.6817 |
2010's | 33 (43.42) | 24.3611 |
2020's | 2 (2.63) | 2.80 |
Authors | Studies |
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Camara, AKS; Elorbany, R; Riess, ML; Stowe, DF; Weihrauch, D | 1 |
Aksentijević, D; Bridges, HR; Burger, N; Grba, DN; Hirst, J; James, AM; Krieg, T; Kula-Alwar, D; Mottahedin, A; Murphy, MP; Prag, HA; Viscomi, C; Yin, Z | 1 |
Jaimes, R; Kay, MW; Kuzmiak-Glancy, S; Moreno, A | 1 |
Liu, L; Wang, P; Wang, Q; Wu, Q; Zhao, B | 1 |
Xu, MJ; Zhang, QL; Zhang, WY | 1 |
Seif, AA | 1 |
Adachi, T; Bessho, M; Kusuhara, M; Nishizawa, K; Ohsuzu, F; Takayama, E; Yamagishi, T; Yanagida, S | 1 |
Chen, D; Fang, N; Li, H; Li, L; Yao, Y; Zhou, C | 1 |
Liu, T; O'Rourke, B | 1 |
Gottlieb, RA; Mentzer, RM; Perry, CN; Wider, J | 1 |
Karamanlidis, G; Kuroda, J; Lee, CF; Matsushima, S; Sadoshima, J; Tian, R; Wang, W; Yu, Q | 1 |
Galagudza, M; Papayan, G; Petrishchev, N | 1 |
Acsai, K; Ágoston, S; Kormos, A; Levijoki, J; Nagy, N; Papp, JG; Pollesello, P; Szentandrássy, N; Tóth, A; Váczi, K; Varró, A | 1 |
Hu, SM; Liu, LX; Rebecchi, M; Wang, C; Xie, H; Zhang, J; Zhu, J | 1 |
Banke, NH; Lewandowski, ED | 1 |
Du, Q; Jovanović, A; Jovanović, S; Mohammed Abdul, KS; Sukhodub, A | 1 |
Albiero, M; Avogaro, A; Bova, S; Cattelan, A; Ceolotto, G; de Kreutzenberg, SV; De Martin, S; Fadini, GP; Kuppusamy, M; Semplicini, A | 1 |
Liu, LF; Qian, ZH; Qin, Q; Shi, M; Tao, XM; Zhang, H; Zhu, WP | 1 |
Korge, P; Ping, P; Weiss, JN | 1 |
Aldakkak, M; Camara, AK; Heisner, JS; Spence, M; Stowe, DF | 1 |
Kavadze, IK; Sukoyan, GV | 1 |
Chance, B; Gorman Iii, JH; Gorman, RC; Hinmon, RH; Jaggard, DL; Leshnower, BG; Matsubara, M; Ranji, M | 1 |
Colantuono, G; Di Venosa, N; Fiore, T; Moro, N; Paradies, G; Petrosillo, G; Ruggiero, FM; Tiravanti, E | 1 |
Cecchini, G; Honbo, N; Jin, ZQ; Karliner, JS; Kotlyar, AB; Randazzo, A | 1 |
Ha, SH; Han, J; Kim, HK; Kim, N; Oh, GT; Park, WS; Thu, VT; Yoo, JY | 1 |
Aponte, A; Deschamps, A; Lagranha, CJ; Murphy, E; Steenbergen, C | 1 |
Brink, PR; Glass, PS; Liu, L; Rebecchi, MJ; Tan, M; Zhu, J | 1 |
Brookes, PS; Nadtochiy, SM; Rahman, I; Redman, E | 1 |
Colantuono, G; Di Venosa, N; Federici, A; Moro, N; Paradies, G; Paradies, V; Petrosillo, G; Ruggiero, FM; Tiravanti, E | 1 |
Brink, PR; Glass, PS; Liu, L; Rebecchi, MJ; Zhu, J | 2 |
Adachi, T; Amo, T; Fukuda, K; Ishiwata, K; Katsumata, Y; Nakashima-Kamimura, N; Ohta, S; Sano, M; Shinmura, K; Suematsu, M; Tamaki, K; Wolf, AM | 1 |
Brookes, PS; Gu, W; Guarente, L; McBurney, MW; Nadtochiy, SM; Rahman, I; Yao, H | 1 |
Aldakkak, M; Camara, AK; Heisner, JS; Rhodes, SS; Riess, ML; Stowe, DF | 1 |
Agarwal, B; Bosnjak, ZJ; Camara, AK; Dash, RK; Stowe, DF | 1 |
Ikeda, T; Komeda, M; Marui, A; Minakata, K; Miwa, S; Oriyanhan, W; Sakata, R; Takaba, K; Tanaka, S; Toyokuni, S; Ueda, K; Yamazaki, K | 1 |
Baliutyte, G; Baniene, R; Borutaite, V; Gendviliene, V; Martisiene, I; Toleikis, A; Trumbeckaite, S | 1 |
Akhmedov, A; Bauer, I; Bertolotto, M; Braunersreuther, V; Bruzzone, S; Dallegri, F; Galan, K; Garuti, A; Gayet-Ageron, A; Lenglet, S; Lerch, R; Lüscher, TF; Mach, F; Mage, J; Mannino, E; Montecucco, F; Montessuit, C; Mostoslavsky, R; Nencioni, A; Patrone, F; Pelli, G; Pellieux, C; Poggi, A; Sebastian, C; Speer, T; Vuilleumier, N | 1 |
Chaté, V; Demaison, L; Leverve, X; Peltier, S; Richardson, M; Suranadi, IW | 1 |
He, D; Liang, C; Liu, L; Liu, X; Wang, P; Yu, Y | 1 |
Hellige, G; Neckel, M; Ochs, M; Schmidt, G; Schrader, C; Vetterlein, F; Volkmann, R | 1 |
Budas, GR; Crawford, RM; Davies, AM; Jovanović, A; Jovanović, S; Lad, H; Ranki, HJ; Robertson, KA; Roy, DJ; Wenger, RH | 1 |
Bacci, ML; Bach, FH; Bracco, A; Calise, F; Cozzi, E; D'Alise, G; Di Florio, E; Forni, M; Froio, A; Giovannoni, R; Lavitrano, M; Maccherini, M; Mancini, A; Musumeci, A; Otterbein, LE; Patti, M; Simeone, F; Slominska, E; Smolenski, RT; Stassi, G; Yacoub, MH | 1 |
Bakr, S; Homma, S; Hwang, YC; Ii, S; Itakura, M; Kaneko, M; Lewis, ER; Liao, H; Lu, Y; Oates, PJ; Ramasamy, R; Rui, L; Schmidt, AM; Skopicki, H; Szabolcs, M; Yan, S | 1 |
Komeda, M; Miwa, S; Nishimura, K; Tanaka, S; Toyokuni, S; Ueda, K; Unimonh, O; Yamazaki, K | 1 |
Andriadze, NA; Guchua, EI; Karsanov, NV; Sukoyan, GV | 1 |
Ascah, A; Bélanger, S; Burelle, Y; Deschepper, CF; Marcil, M; Matas, J | 1 |
Anderson, TC; Becker, LB; Chan, KC; Hamann, KJ; Hoang, T; Li, CQ; Shao, ZH; Vanden Hoek, TL | 1 |
Correa, F; Soto, V; Zazueta, C | 1 |
Clarke, SJ; Halestrap, AP; Khaliulin, I; Lin, H; Parker, J; Suleiman, MS | 1 |
Gille, L; Nohl, H; Schönheit, K | 1 |
Crestanello, JA; Kamelgard, J; Whitman, GJ | 1 |
Bernocchi, P; Cargnoni, A; Ceconi, C; Curello, S; Ferrari, R | 2 |
Terada, LS | 1 |
Bünger, R; Mallet, RT | 1 |
Corsi, M; Ferrero, ME; Gaja, G; Parise, M | 1 |
Camacho, SA; Figueredo, VM; Miyamae, M; Weiner, MW | 1 |
Barbieri, JT; Gross, GJ; Hsu, AK; Li, PL; Schultz, JE | 1 |
Chun, YS; Kim, MS; Kwak, SJ; Park, JW; Park, SC; Park, YC | 1 |
Lucas, DT; Szweda, LI | 1 |
Fischer, GM; Gallyas, F; Kispal, G; Sumegi, B; Szabados, E | 1 |
Amorini, AM; Di Pierro, D; Fazzina, G; Galvano, M; Giardina, B; Lazzarino, G; Lupi, A; Tavazzi, B | 1 |
Ramasamy, R; Schaefer, S; Trueblood, NA; Wang, LF | 1 |
Thiemermann, C | 1 |
Bernocchi, P; Boraso, A; Cargnoni, A; Ceconi, C; Curello, S; Ferrari, R; Pepi, P | 1 |
Barile, M; Bernardi, P; Canton, M; Di Lisa, F; Menabò, R | 1 |
Al-Affar, EB; Liaudet, L; Szabó, C; Yang, Z | 1 |
Habon, T; Halmosi, R; Kesmarky, G; Past, T; Sumegi, B; Szabados, E; Toth, K | 1 |
Lawrence, CL; Rodrigo, GC; Standen, NB | 1 |
Caucheteux, D; Hombroeckx, A; Hue, L; Pouleur, H; Veitch, K | 1 |
Daggett, WM; Horvath, KA; Nishioka, NS; Torchiana, DF | 1 |
Clark, JB; Darley-Usmar, VM; Hardy, DL; Smith, DR | 1 |
Cannon, PJ; Keller, AM | 1 |
Caucheteux, D; Hombroeckx, A; Hue, L; Veitch, K | 1 |
Minezaki, KK; Nakazawa, H; Okino, H; Shinozaki, Y; Yamada, Y | 1 |
1 review(s) available for nad and Injury, Myocardial Reperfusion
Article | Year |
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Possible role of defibrotide in endothelial cell protection.
Topics: Adenine Nucleotides; Animals; Cell Adhesion; Endothelium; Epoprostenol; Heart Transplantation; Ischemia; Kidney; Kidney Transplantation; Leukocytes; Myocardial Reperfusion Injury; NAD; Platelet Adhesiveness; Polydeoxyribonucleotides; Rats; Rats, Wistar; Receptors, Purinergic P1; Reperfusion Injury | 1993 |
75 other study(ies) available for nad and Injury, Myocardial Reperfusion
Article | Year |
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PPARγ-Independent Side Effects of Thiazolidinediones on Mitochondrial Redox State in Rat Isolated Hearts.
Topics: Animals; Flavin-Adenine Dinucleotide; Fluorescence; Male; Mitochondria, Heart; Myocardial Reperfusion Injury; NAD; Oxidation-Reduction; PPAR gamma; Rats, Inbred BN; Thiazolidinediones | 2020 |
Structural basis for a complex I mutation that blocks pathological ROS production.
Topics: Amino Acid Substitution; Animals; Cryoelectron Microscopy; Disease Models, Animal; Disease Resistance; DNA, Mitochondrial; Electron Transport; Electron Transport Complex I; Humans; Isolated Heart Preparation; Leucine; Male; Mice; Mice, Transgenic; Mitochondria; Mitochondrial Membranes; Myocardial Reperfusion Injury; NAD; NADH Dehydrogenase; Oxidation-Reduction; Point Mutation; Proline; Reactive Oxygen Species | 2021 |
Enzyme-dependent fluorescence recovery of NADH after photobleaching to assess dehydrogenase activity of isolated perfused hearts.
Topics: Animals; Female; Fluorescence Recovery After Photobleaching; Heart; Male; Myocardial Reperfusion Injury; Myocardium; NAD; Oxidoreductases; Rats, Sprague-Dawley | 2017 |
Exogenous nicotinamide adenine dinucleotide administration alleviates ischemia/reperfusion-induced oxidative injury in isolated rat hearts via Sirt5-SDH-succinate pathway.
Topics: Animals; Male; Malondialdehyde; Myocardial Reperfusion Injury; Myocardium; NAD; Oxidative Stress; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Signal Transduction; Sirtuins; Succinate Dehydrogenase; Succinic Acid | 2019 |
Effects of propofol on myocardial ischemia reperfusion injury through inhibiting the JAK/STAT pathway.
Topics: Animals; Disease Models, Animal; Gene Expression Regulation; Janus Kinase 1; L-Lactate Dehydrogenase; Myocardial Reperfusion Injury; NAD; Phosphorylation; Propofol; Random Allocation; Rats; Rats, Sprague-Dawley; Signal Transduction; STAT1 Transcription Factor; STAT3 Transcription Factor; Treatment Outcome | 2019 |
Nigella sativa attenuates myocardial ischemic reperfusion injury in rats.
Topics: Animals; Heart Rate; Infusion Pumps; Male; Malondialdehyde; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Reperfusion Injury; Myocardium; NAD; Nigella sativa; Organ Culture Techniques; Plant Preparations; Rats; Rats, Wistar; Rheology; Seeds | 2013 |
Continuous inhibition of poly(ADP-ribose) polymerase does not reduce reperfusion injury in isolated rat heart.
Topics: Animals; Apoptosis; Benzamides; Blotting, Western; Cardiotonic Agents; Enzyme Inhibitors; In Vitro Techniques; Magnetic Resonance Spectroscopy; Male; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD; Oncogene Protein v-akt; Phosphocreatine; Phosphorus; Phosphorylation; Poly(ADP-ribose) Polymerase Inhibitors; Poly(ADP-ribose) Polymerases; Rats; Rats, Wistar; Sodium; Sodium Radioisotopes; Treatment Failure; Ventricular Function, Left | 2013 |
Failure to protect against myocardial ischemia-reperfusion injury with sevoflurane postconditioning in old rats in vivo.
Topics: Aging; Anesthetics, Inhalation; Animals; Blotting, Western; Enzyme Activation; Ischemic Postconditioning; MAP Kinase Signaling System; Methyl Ethers; Mitochondria; Mitogen-Activated Protein Kinases; Myocardial Reperfusion Injury; Myocardium; NAD; Permeability; Phosphorylation; Proto-Oncogene Proteins c-akt; Rats; Rats, Sprague-Dawley; Sevoflurane; Ventricular Function, Left | 2013 |
Regulation of the Na+/Ca2+ exchanger by pyridine nucleotide redox potential in ventricular myocytes.
Topics: Animals; Calcium; Catalase; Cells, Cultured; Enzyme Inhibitors; Glycoproteins; Guinea Pigs; Heart Ventricles; Homeostasis; Hydrogen Peroxide; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; NADPH Oxidases; Onium Compounds; Oxidation-Reduction; Sodium-Calcium Exchanger | 2013 |
Reduction of infarct size by the therapeutic protein TAT-Ndi1 in vivo.
Topics: Animals; Disease Models, Animal; Electron Transport Complex I; Female; Gene Products, tat; Mitochondria, Heart; Myocardial Infarction; Myocardial Reperfusion Injury; NAD; Rats; Rats, Sprague-Dawley | 2014 |
Elimination of NADPH oxidase activity promotes reductive stress and sensitizes the heart to ischemic injury.
Topics: Animals; Disease Models, Animal; Energy Metabolism; Glutathione; Mice; Mice, Knockout; Mitochondria, Heart; Myocardial Contraction; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD; NADPH Oxidase 4; NADPH Oxidases; Oxidation-Reduction; Oxidative Stress; Reactive Oxygen Species; Time Factors | 2014 |
Autofluorescence spectroscopy for NADH and flavoproteins redox state monitoring in the isolated rat heart subjected to ischemia-reperfusion.
Topics: Animals; Flavoproteins; In Vitro Techniques; Male; Myocardial Reperfusion Injury; Myocardium; NAD; Oxidation-Reduction; Rats; Rats, Wistar; Spectrometry, Fluorescence | 2014 |
Efficacy of selective NCX inhibition by ORM-10103 during simulated ischemia/reperfusion.
Topics: Action Potentials; Animals; Benzopyrans; Calcium; Cell Survival; Dogs; Female; Male; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; Pyridines; Sodium-Calcium Exchanger | 2014 |
Sevoflurane post-conditioning protects isolated rat hearts against ischemia-reperfusion injury via activation of the ERK1/2 pathway.
Topics: Adenosine Triphosphate; Animals; Apoptosis; Apoptosis Regulatory Proteins; Cardiotonic Agents; Cytoprotection; Disease Models, Animal; Enzyme Activation; Heart Rate; Male; MAP Kinase Signaling System; Methyl Ethers; Mitochondria, Heart; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Myocardial Contraction; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD; Perfusion; Phosphorylation; Protein Kinase Inhibitors; Rats, Sprague-Dawley; Sevoflurane; Time Factors; Ventricular Function, Left | 2014 |
Impaired cytosolic NADH shuttling and elevated UCP3 contribute to inefficient citric acid cycle flux support of postischemic cardiac work in diabetic hearts.
Topics: Animals; Aspartic Acid; Carbon-13 Magnetic Resonance Spectroscopy; Carrier Proteins; Citric Acid Cycle; Cytosol; Diabetes Mellitus, Experimental; Hemodynamics; Ion Channels; Malates; Male; Mice, Inbred C57BL; Mitochondrial Proteins; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; NAD; Oxidation-Reduction; Perfusion; PPAR alpha; Uncoupling Protein 2; Uncoupling Protein 3 | 2015 |
Mild hypoxia in vivo regulates cardioprotective SUR2A: A role for Akt and LDH.
Topics: Animals; Blotting, Western; Cardiotonic Agents; Cell Hypoxia; Cell Line; Chromones; Enzyme Inhibitors; Female; Hypoxia; L-Lactate Dehydrogenase; Lactates; Male; Mice, Inbred C57BL; Morpholines; Mutation; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Phosphorylation; Proto-Oncogene Proteins c-akt; Rats; Signal Transduction; Sodium Lactate; Sulfonylurea Receptors | 2015 |
NAD(+)-dependent SIRT1 deactivation has a key role on ischemia-reperfusion-induced apoptosis.
Topics: AMP-Activated Protein Kinases; Animals; Apoptosis; Binding Sites; Caspase 3; DNA-Binding Proteins; Enzyme Activation; Forkhead Transcription Factors; Glucose; Heat Shock Transcription Factors; Isolated Heart Preparation; Male; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; Nerve Tissue Proteins; Promoter Regions, Genetic; Rats, Inbred WKY; Resveratrol; Signal Transduction; Sirtuin 1; Sirtuins; Stilbenes; Time Factors; Transcription Factors | 2015 |
Effect of melatonin on oncosis of myocardial cells in the myocardial ischemia/reperfusion injury rat and the role of the mitochondrial permeability transition pore.
Topics: Animals; Male; Melatonin; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD; Rats, Sprague-Dawley; Receptors, Cell Surface | 2015 |
Reactive oxygen species production in energized cardiac mitochondria during hypoxia/reoxygenation: modulation by nitric oxide.
Topics: Aconitate Hydratase; Animals; Cell Hypoxia; Cell Respiration; Electron Transport; Fluoresceins; Glutathione; Hydrogen Peroxide; Iron; Membrane Potential, Mitochondrial; Mitochondria, Heart; Myocardial Reperfusion Injury; Myocardium; NAD; Nitric Oxide; Oxidation-Reduction; Oxygen; Oxygen Consumption; Rabbits; Reactive Oxygen Species; Signal Transduction; Time Factors | 2008 |
Enhanced Na+/H+ exchange during ischemia and reperfusion impairs mitochondrial bioenergetics and myocardial function.
Topics: Animals; Calcium; Flavin-Adenine Dinucleotide; Guanidines; Guinea Pigs; Hydrogen-Ion Concentration; Mitochondria, Heart; Myocardial Reperfusion Injury; NAD; Oxidation-Reduction; Reactive Oxygen Species; Sodium-Hydrogen Exchangers; Spectrometry, Fluorescence; Sulfones; Superoxides | 2008 |
Effect of nadcin on energy supply system and apoptosis in ischemia-reperfusion injury to the myocardium.
Topics: Adenine Nucleotides; Animals; Apoptosis; Dogs; Drug Combinations; Energy Metabolism; Female; Inosine; Male; Myocardial Reperfusion Injury; NAD; Poly(ADP-ribose) Polymerases | 2008 |
Quantifying acute myocardial injury using ratiometric fluorometry.
Topics: Analysis of Variance; Animals; Apoptosis; Biomarkers; Disease Models, Animal; Flavoproteins; Microscopy, Electron, Transmission; Mitochondria, Heart; Myocardial Infarction; Myocardial Reperfusion Injury; NAD; Oxidation-Reduction; Rabbits; Spectrometry, Fluorescence | 2009 |
Melatonin protects against heart ischemia-reperfusion injury by inhibiting mitochondrial permeability transition pore opening.
Topics: Animals; Antioxidants; Calcium; Cardiolipins; Cardiovascular Agents; Cyclosporine; Cytochromes c; Heart Rate; In Vitro Techniques; L-Lactate Dehydrogenase; Lipid Peroxidation; Male; Melatonin; Membrane Potential, Mitochondrial; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD; Necrosis; Perfusion; Rats; Rats, Wistar; Recovery of Function; Time Factors; Ventricular Function, Left; Ventricular Pressure | 2009 |
Cardioprotective activity of a novel and potent competitive inhibitor of lactate dehydrogenase.
Topics: Animals; Cardiotonic Agents; Enzyme Inhibitors; In Vitro Techniques; L-Lactate Dehydrogenase; Mice; Mice, Inbred C57BL; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD | 2010 |
Glutathione peroxidase 1 protects mitochondria against hypoxia/reoxygenation damage in mouse hearts.
Topics: Adenosine Triphosphate; Animals; Blotting, Western; Cell Hypoxia; Cytoprotection; DNA Damage; DNA, Mitochondrial; Electrophoresis, Gel, Two-Dimensional; Glutathione Peroxidase; Glutathione Peroxidase GPX1; Hydrogen Peroxide; Male; Membrane Potential, Mitochondrial; Mice; Mice, Inbred C57BL; Mice, Knockout; Mitochondria, Muscle; Myocardial Reperfusion Injury; Myocardium; NAD; Oxygen Consumption; Perfusion; Protein Interaction Domains and Motifs; Protein Interaction Mapping; Proteomics; Reactive Oxygen Species; Superoxides; Time Factors | 2010 |
Sex differences in the phosphorylation of mitochondrial proteins result in reduced production of reactive oxygen species and cardioprotection in females.
Topics: Aldehyde Dehydrogenase; Aldehyde Dehydrogenase, Mitochondrial; Androstadienes; Animals; Benzamides; Benzodioxoles; Blotting, Western; Disease Models, Animal; Electrophoresis, Gel, Two-Dimensional; Enzyme Activation; Enzyme Activators; Estradiol; Female; Indoles; Ketoglutarate Dehydrogenase Complex; Male; Maleimides; Mitochondria, Heart; Mitochondrial Proteins; Myocardial Infarction; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; Ovariectomy; Oxidative Stress; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Phosphorylation; Protein Kinase C; Protein Kinase Inhibitors; Protein Processing, Post-Translational; Proteomics; Rats; Rats, Sprague-Dawley; Reactive Oxygen Species; Sex Factors; Ventricular Function, Left; Ventricular Pressure; Wortmannin | 2010 |
Age-associated differences in activation of Akt/GSK-3beta signaling pathways and inhibition of mitochondrial permeability transition pore opening in the rat heart.
Topics: Aging; Anesthetics, Inhalation; Animals; Cardiotonic Agents; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Heart Ventricles; Hemodynamics; Ischemic Preconditioning, Myocardial; Isoflurane; Male; Microscopy, Electron; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD; Phosphorylation; Proto-Oncogene Proteins c-akt; Rats; Rats, Inbred F344; Signal Transduction | 2010 |
Lysine deacetylation in ischaemic preconditioning: the role of SIRT1.
Topics: Acetylation; Animals; Disease Models, Animal; Ischemic Preconditioning, Myocardial; Lysine; Male; Mice; Mice, Inbred C57BL; Myocardial Reperfusion Injury; Myocardium; Myocytes, Cardiac; NAD; Sirtuin 1 | 2011 |
In vivo hyperoxic preconditioning protects against rat-heart ischemia/reperfusion injury by inhibiting mitochondrial permeability transition pore opening and cytochrome c release.
Topics: Animals; Calcium; Cardiolipins; Cytochromes c; Hyperoxia; Male; Mitochondrial Membrane Transport Proteins; Mitochondrial Membranes; Mitochondrial Permeability Transition Pore; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD; Necrosis; Oxygen; Rats; Rats, Wistar | 2011 |
Cardioprotection of the aged rat heart by GSK-3beta inhibitor is attenuated: age-related changes in mitochondrial permeability transition pore modulation.
Topics: Aging; Animals; Glycogen Synthase Kinase 3; Glycogen Synthase Kinase 3 beta; Heart; Indoles; Male; Maleimides; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; Myocytes, Cardiac; NAD; Rats; Rats, Inbred F344; Reactive Oxygen Species | 2011 |
Caloric restriction primes mitochondria for ischemic stress by deacetylating specific mitochondrial proteins of the electron transport chain.
Topics: Acetylation; Animals; Antioxidants; Blotting, Western; Caloric Restriction; Cells, Cultured; Disease Models, Animal; Electron Transport Chain Complex Proteins; Electron Transport Complex III; Humans; Hydrogen Peroxide; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; NADH Dehydrogenase; Niacinamide; Oxidative Stress; Proteomics; Rats; Rats, Inbred F344; Resveratrol; Sirtuins; Stilbenes | 2011 |
Age-associated differences in the inhibition of mitochondrial permeability transition pore opening by cyclosporine A.
Topics: Aging; Animals; Cardiotonic Agents; Cell Separation; Cyclosporine; Hemodynamics; Image Processing, Computer-Assisted; Immunosuppressive Agents; In Vitro Techniques; Male; Microscopy, Confocal; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Infarction; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; Permeability; Rats; Rats, Inbred F344; Reactive Oxygen Species | 2011 |
SIRT1-mediated acute cardioprotection.
Topics: Acetylation; Animals; Animals, Genetically Modified; Blotting, Western; Cytosol; Forkhead Box Protein O1; Forkhead Transcription Factors; Heart; In Vitro Techniques; Ischemic Preconditioning, Myocardial; Lysine; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocardial Reperfusion Injury; NAD; Signal Transduction; Sirtuin 1; Superoxide Dismutase | 2011 |
Reduced mitochondrial Ca2+ loading and improved functional recovery after ischemia-reperfusion injury in old vs. young guinea pig hearts.
Topics: Aging; Animals; Calcium; Cardiotonic Agents; Coronary Circulation; Cytosol; Digoxin; Dopamine; Guinea Pigs; Mitochondria; Myocardial Contraction; Myocardial Infarction; Myocardial Reperfusion Injury; Myocardium; NAD; Recovery of Function | 2012 |
Enhanced charge-independent mitochondrial free Ca(2+) and attenuated ADP-induced NADH oxidation by isoflurane: Implications for cardioprotection.
Topics: Adenosine Diphosphate; Animals; Calcium; Energy Metabolism; Isoflurane; Membrane Potential, Mitochondrial; Mitochondria, Heart; Myocardial Reperfusion Injury; NAD; Oxidation-Reduction; Rats; Rats, Wistar | 2012 |
Protective effect of cardioplegia with poly (ADP-ribose) polymerase-1 inhibitor against myocardial ischemia-reperfusion injury: in vitro study of isolated rat heart model.
Topics: Animals; Apoptosis; Cardioplegic Solutions; Cardiotonic Agents; Enzyme Inhibitors; Heart; Hydrazines; In Vitro Techniques; Male; Myocardial Reperfusion Injury; Myocardium; Myocytes, Cardiac; NAD; Oxidative Stress; Poly (ADP-Ribose) Polymerase-1; Poly(ADP-ribose) Polymerase Inhibitors; Quinazolinones; Rats; Rats, Sprague-Dawley; Ventricular Function, Left | 2013 |
Influence of ethanol extract of Ginkgo biloba leaves on the isolated rat heart work and mitochondria functions.
Topics: Animals; Dose-Response Relationship, Drug; Ethanol; Fluorescence; Ginkgo biloba; Heart Rate; Hydrogen Peroxide; Malates; Male; Mitochondria, Heart; Myocardial Contraction; Myocardial Reperfusion Injury; NAD; Oxidation-Reduction; Plant Extracts; Plant Leaves; Pyruvic Acid; Rats; Rats, Wistar; Time Factors | 2012 |
Inhibition of nicotinamide phosphoribosyltransferase reduces neutrophil-mediated injury in myocardial infarction.
Topics: Acrylamides; Animals; Chemokine CXCL2; Humans; Male; Mice; Myocardial Infarction; Myocardial Reperfusion Injury; NAD; Neutrophil Infiltration; Nicotinamide Phosphoribosyltransferase; Oxidative Stress; Piperidines; Reactive Oxygen Species; Signal Transduction | 2013 |
An increase in the redox state during reperfusion contributes to the cardioprotective effect of GIK solution.
Topics: Amide Synthases; Animals; Cardiotonic Agents; Glucose; Insulin; Male; Mitochondria, Heart; Myocardial Reperfusion Injury; NAD; Oxidation-Reduction; Potassium; Random Allocation; Rats; Rats, Wistar; Reactive Oxygen Species | 2012 |
Exogenous NAD(+) supplementation protects H9c2 cardiac myoblasts against hypoxia/reoxygenation injury via Sirt1-p53 pathway.
Topics: Animals; Apoptosis; Cell Culture Techniques; Cell Hypoxia; Cell Line; Cell Survival; Dose-Response Relationship, Drug; Myoblasts, Cardiac; Myocardial Reperfusion Injury; NAD; Oxygen; Rats; Signal Transduction; Sirtuin 1; Time Factors; Tumor Suppressor Protein p53 | 2014 |
Extent of damage in ischemic, nonreperfused, and reperfused myocardium of anesthetized rats.
Topics: Anesthesia; Animals; Female; Fluorescence; Microscopy, Electron; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; Myocytes, Cardiac; NAD; Rats; Rats, Wistar; Tetrazolium Salts | 2003 |
Chronic mild hypoxia protects heart-derived H9c2 cells against acute hypoxia/reoxygenation by regulating expression of the SUR2A subunit of the ATP-sensitive K+ channel.
Topics: Acute Disease; Adenosine Triphosphate; Animals; Calcium; Cell Membrane; Cells, Cultured; Chronic Disease; Gene Expression Regulation; Hypoxia; Hypoxia-Inducible Factor 1, alpha Subunit; MAP Kinase Kinase 1; Mitogen-Activated Protein Kinase Kinases; Myocardial Reperfusion Injury; Myocardium; NAD; Oxygen; Phenotype; Potassium Channels, Inwardly Rectifying; Promoter Regions, Genetic; Protein Serine-Threonine Kinases; Rats; Sarcolemma; Signal Transduction; Transcription Factors | 2003 |
Carbon monoxide improves cardiac energetics and safeguards the heart during reperfusion after cardiopulmonary bypass in pigs.
Topics: Adenosine Diphosphate; Adenosine Monophosphate; Adenosine Triphosphate; Animals; Apoptosis; Carbon Monoxide; Cardiopulmonary Bypass; Cardiotonic Agents; Edema; Electric Countershock; Energy Metabolism; Female; Guanosine Triphosphate; Heart; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; NADP; Oxidation-Reduction; Sus scrofa | 2004 |
Central role for aldose reductase pathway in myocardial ischemic injury.
Topics: Adenosine Triphosphate; Aldehyde Reductase; Animals; Coronary Vessels; Cytosol; Enzyme Inhibitors; Glucose; Glycolysis; L-Iditol 2-Dehydrogenase; Ligation; Mice; Mice, Inbred C57BL; Mice, Transgenic; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; NAD; Niacin; Organ Culture Techniques; Oxidation-Reduction; Palmitic Acid; Recombinant Fusion Proteins; Ventricular Function, Left | 2004 |
Prevention of myocardial reperfusion injury by poly(ADP-ribose) synthetase inhibitor, 3-aminobenzamide, in cardioplegic solution: in vitro study of isolated rat heart model.
Topics: Animals; Apoptosis; Benzamides; Cardiac Surgical Procedures; Cardioplegic Solutions; Disease Models, Animal; DNA; Enzyme Inhibitors; Heart; Heart Arrest, Induced; Male; Muscle, Smooth, Vascular; Myocardial Reperfusion Injury; Myocardium; NAD; Oxidative Stress; Poly(ADP-ribose) Polymerase Inhibitors; Rats; Rats, Sprague-Dawley; Time Factors; Ventricular Function, Left | 2004 |
Effect of NAD on recovery of adenine nucleotide pool, phosphorylation potential, and stimulation of apoptosis during late period of reperfusion damage to myocardium.
Topics: Adenine Nucleotides; Adenosine Triphosphate; Animals; Apoptosis; Dogs; Heart Ventricles; Myocardial Reperfusion Injury; Myocardium; NAD; Phosphocreatine; Phosphorylation; Poly(ADP-ribose) Polymerase Inhibitors; Poly(ADP-ribose) Polymerases | 2005 |
Compensated volume overload increases the vulnerability of heart mitochondria without affecting their functions in the absence of stress.
Topics: Animals; Calcium; Electron Transport; Heart Failure; In Vitro Techniques; Male; Membrane Potential, Mitochondrial; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Reperfusion Injury; NAD; Oxidative Phosphorylation; Rats; Rats, Inbred Strains; Ventricular Remodeling | 2006 |
Transient and partial mitochondrial inhibition for the treatment of postresuscitation injury: getting it just right.
Topics: Animals; Anthracenes; Carboxylic Acids; Cardiopulmonary Resuscitation; Chick Embryo; Ditiocarb; Dose-Response Relationship, Drug; Electron Transport; Mitochondria, Heart; Myocardial Reperfusion Injury; Myocytes, Cardiac; NAD; Polyenes; Reactive Oxygen Species; Rotenone | 2006 |
Mitochondrial permeability transition relevance for apoptotic triggering in the post-ischemic heart.
Topics: Animals; Apoptosis; bcl-2-Associated X Protein; Blotting, Western; Calcium; Carbonyl Cyanide m-Chlorophenyl Hydrazone; Cyclosporine; Cytochromes c; Electron Transport Complex IV; Heart; In Situ Nick-End Labeling; In Vitro Techniques; Male; Mitochondria, Heart; Mitochondrial Swelling; Myocardial Reperfusion Injury; Myocardium; NAD; Permeability; Protein Transport; Rats; Rats, Wistar; Ruthenium Compounds; Time Factors; Uncoupling Agents | 2007 |
Temperature preconditioning of isolated rat hearts--a potent cardioprotective mechanism involving a reduction in oxidative stress and inhibition of the mitochondrial permeability transition pore.
Topics: AMP-Activated Protein Kinases; Animals; Arrhythmias, Cardiac; Coronary Circulation; Hypothermia, Induced; In Vitro Techniques; Ischemic Preconditioning, Myocardial; L-Lactate Dehydrogenase; Male; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Mitochondrial Swelling; Multienzyme Complexes; Myocardial Reperfusion Injury; Myocardium; NAD; Necrosis; Oxidative Stress; Perfusion; Phosphocreatine; Phosphorylation; Protein Carbonylation; Protein Kinase C-epsilon; Protein Serine-Threonine Kinases; Protein Transport; Rats; Rats, Wistar; Reactive Oxygen Species; Rewarming; Signal Transduction; Temperature | 2007 |
Stimulation and modulation of mitochondrial radical generation during reperfusion injury of ischemic hearts.
Topics: 8,11,14-Eicosatrienoic Acid; Animals; Electron Spin Resonance Spectroscopy; Epinephrine; Free Radicals; Kinetics; Microwaves; Mitochondria, Heart; Myocardial Reperfusion Injury; NAD; Rats; Rotenone; Superoxide Dismutase; Superoxides | 1995 |
The cumulative nature of pyruvate's dual mechanism for myocardial protection.
Topics: Adenosine Triphosphate; Animals; Creatine Kinase; Magnetic Resonance Spectroscopy; Male; Myocardial Reperfusion Injury; Myocardium; NAD; Pyruvates; Pyruvic Acid; Rats; Rats, Sprague-Dawley | 1995 |
In vitro administration of ergothioneine failed to protect isolated ischaemic and reperfused rabbit heart.
Topics: Adenine Nucleotides; Animals; Creatine Kinase; Ergothioneine; Glutathione; Glutathione Disulfide; In Vitro Techniques; Lactates; Lactic Acid; Male; Myocardial Contraction; Myocardial Reperfusion Injury; NAD; Oxidative Stress; Rabbits | 1995 |
In vitro ergothioneine administration failed to protect isolated ischaemic and reperfused rabbit heart.
Topics: Adenosine Triphosphate; Animals; Creatine Kinase; Ergothioneine; Glutathione; Glutathione Disulfide; Heart; In Vitro Techniques; Lactates; Myocardial Ischemia; Myocardial Reperfusion; Myocardial Reperfusion Injury; Myocardium; NAD; Phosphocreatine; Rabbits | 1994 |
Suppression of rabbit myocardial xanthine dehydrogenase activity by an endogenous compound.
Topics: Animals; Butanols; In Vitro Techniques; Milk; Molecular Weight; Myocardial Reperfusion Injury; Myocardium; NAD; Rabbits; Xanthine Dehydrogenase; Xanthine Oxidase | 1994 |
Metabolic protection of post-ischemic phosphorylation potential and ventricular performance.
Topics: Adenosine Triphosphate; Animals; Cardiotonic Agents; Coronary Circulation; Guinea Pigs; In Vitro Techniques; Myocardial Reperfusion Injury; NAD; Phosphorylation; Pyruvates; Pyruvic Acid; Ventricular Function, Left | 1993 |
Attenuation of postischemic reperfusion injury is related to prevention of [Ca2+]m overload in rat hearts.
Topics: Animals; Calcium; Cytosol; Endothelium, Vascular; Fluorescence; Hemodynamics; Indoles; Male; Mitochondria, Heart; Myocardial Ischemia; Myocardial Reperfusion Injury; NAD; Osmolar Concentration; Rats; Rats, Sprague-Dawley | 1996 |
Pertussis toxin abolishes the cardioprotective effect of ischemic preconditioning in intact rat heart.
Topics: Acetylcholine; Adenosine; Adenosine Diphosphate Ribose; Animals; Dogs; GTP-Binding Proteins; Heart Rate; Ischemic Preconditioning, Myocardial; Male; Myocardial Infarction; Myocardial Ischemia; Myocardial Reperfusion Injury; NAD; Pertussis Toxin; Rabbits; Rats; Rats, Wistar; Virulence Factors, Bordetella | 1998 |
Metabolic modulation of cellular redox potential can improve cardiac recovery from ischemia-reperfusion injury.
Topics: Animals; Aspartic Acid; Male; Myocardial Contraction; Myocardial Reperfusion Injury; Myocardium; NAD; Oxidation-Reduction; Pyruvic Acid; Rats; Rats, Sprague-Dawley; Xanthines | 1998 |
Declines in mitochondrial respiration during cardiac reperfusion: age-dependent inactivation of alpha-ketoglutarate dehydrogenase.
Topics: Aging; Animals; Cell Respiration; Ketoglutarate Dehydrogenase Complex; Male; Mitochondria, Heart; Myocardial Reperfusion Injury; Myocardium; NAD; Rats; Rats, Inbred F344 | 1999 |
Enhanced ADP-ribosylation and its diminution by lipoamide after ischemia-reperfusion in perfused rat heart.
Topics: Adenosine Diphosphate Ribose; ADP Ribose Transferases; Animals; Antioxidants; DNA Damage; Enzyme Activation; In Vitro Techniques; Lipid Peroxidation; Male; Mitochondria, Heart; Myocardial Reperfusion Injury; NAD; Perfusion; Poly(ADP-ribose) Polymerases; Proteins; Rats; Rats, Wistar; Reactive Oxygen Species; Thioctic Acid | 1999 |
Direct NAD(P)H hydrolysis into ADP-ribose(P) and nicotinamide induced by reactive oxygen species: a new mechanism of oxygen radical toxicity.
Topics: Adenosine Diphosphate Ribose; Animals; Free Radicals; Hydrolysis; In Vitro Techniques; Male; Models, Biological; Myocardial Reperfusion Injury; NAD; NADP; Niacinamide; Rats; Rats, Wistar; Reactive Oxygen Species | 2000 |
Niacin protects the isolated heart from ischemia-reperfusion injury.
Topics: Animals; Blood Pressure; Creatine Kinase; Fatty Acids, Nonesterified; Heart; Heart Rate; In Vitro Techniques; Lactates; Male; Myocardial Ischemia; Myocardial Reperfusion Injury; NAD; Niacin; Pyruvates; Rats; Rats, Sprague-Dawley; Ventricular Function, Left | 2000 |
Inhibition of the activity of poly (ADP-ribose) polymerase in ischemia-reperfusion injury.
Topics: Animals; DNA Damage; Drug Design; Energy Metabolism; Enzyme Inhibitors; Feedback; Humans; Ischemia; Myocardial Infarction; Myocardial Reperfusion Injury; NAD; Niacinamide; Poly(ADP-ribose) Polymerase Inhibitors; Poly(ADP-ribose) Polymerases; Reperfusion Injury; Solubility | 2000 |
New insights on myocardial pyridine nucleotides and thiol redox state in ischemia and reperfusion damage.
Topics: Adenosine Triphosphate; Animals; Creatine Kinase; Energy Metabolism; Glucose-6-Phosphate; Glutathione; Male; Myocardial Reperfusion Injury; Myocardium; NAD; NADP; Oxidation-Reduction; Perfusion; Phosphocreatine; Rabbits; Sulfhydryl Compounds | 2000 |
Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.
Topics: Animals; Cell Death; Cyclosporine; Cytosol; In Vitro Techniques; Ion Channels; Male; Membrane Proteins; Mitochondria, Heart; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; NAD; NAD+ Nucleosidase; Rats; Rats, Wistar | 2001 |
Myocardial ischemic preconditioning in rodents is dependent on poly (ADP-ribose) synthetase.
Topics: Animals; Benzamides; Enzyme Inhibitors; Immunohistochemistry; Interleukin-10; Interleukin-12; Ischemic Preconditioning, Myocardial; Male; Mice; Mice, Knockout; Myocardial Reperfusion Injury; Myocardium; NAD; Peroxidase; Poly(ADP-ribose) Polymerase Inhibitors; Poly(ADP-ribose) Polymerases; Rats; Rats, Wistar; Tissue Extracts; Tumor Necrosis Factor-alpha; Tyrosine | 2001 |
The effect of carvedilol on enhanced ADP-ribosylation and red blood cell membrane damage caused by free radicals.
Topics: Adenosine Diphosphate Ribose; ADP Ribose Transferases; Adrenergic Antagonists; Animals; Blood Viscosity; Carbazoles; Carvedilol; Erythrocyte Membrane; Hemorheology; Humans; Lipid Peroxidation; Male; Myocardial Reperfusion Injury; NAD; Perfusion; Poly(ADP-ribose) Polymerases; Propanolamines; Rats; Rats, Wistar; Reactive Oxygen Species; Signal Transduction | 2001 |
Dinitrophenol pretreatment of rat ventricular myocytes protects against damage by metabolic inhibition and reperfusion.
Topics: Animals; Cyanides; Dinitrophenols; Iodoacetic Acid; Ischemic Preconditioning, Myocardial; Male; Mitochondria; Muscle Fibers, Skeletal; Myocardial Contraction; Myocardial Reperfusion Injury; Myocardium; NAD; Rats; Rats, Wistar; Uncoupling Agents | 2002 |
Global ischaemia induces a biphasic response of the mitochondrial respiratory chain. Anoxic pre-perfusion protects against ischaemic damage.
Topics: Animals; Buffers; Coronary Disease; Electron Transport Complex II; Electron Transport Complex III; Electron Transport Complex IV; Glutamates; Glutamic Acid; Kinetics; Malates; Male; Mitochondria, Heart; Multienzyme Complexes; Myocardial Reperfusion Injury; NAD; NAD(P)H Dehydrogenase (Quinone); NADH Dehydrogenase; NADH, NADPH Oxidoreductases; Oxidation-Reduction; Oxidoreductases; Oxygen; Oxygen Consumption; Rats; Rats, Inbred Strains; Succinate Dehydrogenase; Time Factors | 1992 |
Monitoring myocardial reperfusion injury with NADH fluorometry.
Topics: Animals; Aorta; Cardiac Output; Coronary Circulation; Coronary Disease; Fluorescence; Fluorometry; Glycogen; Male; Monitoring, Physiologic; Myocardial Reperfusion; Myocardial Reperfusion Injury; Myocardium; NAD; Oxygen; Phosphates; Rats; Rats, Inbred Strains; Ventricular Function | 1992 |
Reoxygenation of the hypoxic myocardium causes a mitochondrial complex I defect.
Topics: Animals; Calcium; Electron Transport; Flavin-Adenine Dinucleotide; In Vitro Techniques; Mitochondria, Heart; Myocardial Reperfusion Injury; NAD; NAD(P)H Dehydrogenase (Quinone); Quinone Reductases; Rats; Ruthenium Red | 1990 |
Effect of graded reductions of coronary pressure and flow on myocardial metabolism and performance: a model of "hibernating" myocardium.
Topics: Adenosine Diphosphate; Adenosine Triphosphate; Animals; Coronary Circulation; Lactates; Lactic Acid; Magnetic Resonance Spectroscopy; Male; Myocardial Contraction; Myocardial Reperfusion Injury; Myocardium; NAD; Oxygen Consumption; Rats; Rats, Inbred Strains; Ventricular Function, Left | 1991 |
Mitochondrial damage during cardiac ischaemia and reperfusion: the role of oxygen.
Topics: Animals; Coronary Disease; Free Radicals; In Vitro Techniques; Mitochondria, Heart; Myocardial Reperfusion Injury; NAD; NAD(P)H Dehydrogenase (Quinone); Oxygen Consumption; Quinone Reductases; Rats | 1990 |
The detection of technical failures in perfused heart with ischemia and reperfusion by epicardial NADH fluorescence.
Topics: Animals; Fluorescence; Male; Myocardial Reperfusion; Myocardial Reperfusion Injury; NAD; Pericardium; Photography; Rats; Rats, Inbred Strains; Ultraviolet Rays | 1990 |