n-tert-butylhydroxylamine has been researched along with Neuronal Ceroid-Lipofuscinoses in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bagh, MB; Chandra, G; Moralle, M; Mukherjee, AB; Peng, S; Saha, A; Sarkar, C; Zhang, Z | 1 |
| Chen, JG; Deng, Q; Nie, TL; Shen, ZC; Wang, F; Wang, J; Wu, PF; Xia, ZX; Zhang, SQ | 1 |
| Chandra, G; Liu, A; Mukherjee, AB; Peng, S; Sarkar, C; Zhang, Z | 1 |
3 other study(ies) available for n-tert-butylhydroxylamine and Neuronal Ceroid-Lipofuscinoses
| Article | Year |
|---|---|
Cln1 gene disruption in mice reveals a common pathogenic link between two of the most lethal childhood neurodegenerative lysosomal storage disorders.
Topics: Animals; Brain; Cathepsin D; Child; Disease Models, Animal; Gene Expression Regulation; Humans; Hydroxylamines; Lysosomes; Mice; Mutation; Neuronal Ceroid-Lipofuscinoses; Thiolester Hydrolases | 2015 |
De-palmitoylation by N-(tert-Butyl) hydroxylamine inhibits AMPAR-mediated synaptic transmission via affecting receptor distribution in postsynaptic densities.
Topics: Animals; Biotin; Calcium Channels; Disks Large Homolog 4 Protein; Excitatory Postsynaptic Potentials; Hippocampus; Hydroxylamines; Male; Mice; Mice, Inbred C57BL; Neuronal Ceroid-Lipofuscinoses; Palmitates; Patch-Clamp Techniques; Receptors, AMPA; Synapses; Synaptic Transmission | 2019 |
Neuroprotection and lifespan extension in Ppt1(-/-) mice by NtBuHA: therapeutic implications for INCL.
Topics: Animals; Apoptosis; Carbon Isotopes; Cells, Cultured; Cerebral Cortex; Disease Models, Animal; Dose-Response Relationship, Drug; Gene Expression Regulation; Hydroxylamines; Longevity; Mice; Mice, Inbred C57BL; Mice, Knockout; Neuronal Ceroid-Lipofuscinoses; Neurons; Neuroprotective Agents; Palmitoyl Coenzyme A; Thiolester Hydrolases; Time Factors | 2013 |