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n,n,n',n'-tetrakis(2-pyridylmethyl)ethylenediamine and Ischemic Attack, Transient

n,n,n',n'-tetrakis(2-pyridylmethyl)ethylenediamine has been researched along with Ischemic Attack, Transient in 1 studies

N,N,N',N'-tetrakis(2-pyridylmethyl)ethylenediamine: a zinc ion chelating agent; structure given in first source
N,N,N',N'-tetrakis(2-pyridylmethyl)ethylenediamine : An N-substituted diamine that is ethylenediamine in which the four amino hydrogens are replaced by 2-pyridylmethyl groups.

Ischemic Attack, Transient: Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Zhao, Y1
Pan, R1
Li, S1
Luo, Y1
Yan, F1
Yin, J1
Qi, Z1
Yan, Y1
Ji, X1
Liu, KJ1

Other Studies

1 other study available for n,n,n',n'-tetrakis(2-pyridylmethyl)ethylenediamine and Ischemic Attack, Transient

ArticleYear
Chelating intracellularly accumulated zinc decreased ischemic brain injury through reducing neuronal apoptotic death.
    Stroke, 2014, Volume: 45, Issue:4

    Topics: Animals; Apoptosis; Brain Ischemia; Chelating Agents; Cytosol; Ethylenediamines; Infarction, Middle

2014