Page last updated: 2024-08-25

n-methylscopolamine and Colonic Neoplasms

n-methylscopolamine has been researched along with Colonic Neoplasms in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's2 (66.67)18.2507
2000's1 (33.33)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Frucht, H; Yang, WL1
Frucht, H; Gazdar, AF; Jensen, RT; Oie, H; Park, JA1
Kopp, R; Mayer, P; Pfeiffer, A1

Other Studies

3 other study(ies) available for n-methylscopolamine and Colonic Neoplasms

ArticleYear
Cholinergic receptor up-regulates COX-2 expression and prostaglandin E(2) production in colon cancer cells.
    Carcinogenesis, 2000, Volume: 21, Issue:10

    Topics: Carbachol; Colon; Colonic Neoplasms; Cyclooxygenase 2; Dinoprostone; Dose-Response Relationship, Drug; Gene Expression Regulation, Neoplastic; HT29 Cells; Humans; Isoenzymes; Membrane Proteins; Muscarinic Agonists; Muscarinic Antagonists; N-Methylscopolamine; Prostaglandin-Endoperoxide Synthases; Receptor, Muscarinic M3; Receptors, Muscarinic; Reference Values; Reverse Transcriptase Polymerase Chain Reaction; RNA, Neoplasm; Up-Regulation

2000
Characterization of functional receptors for gastrointestinal hormones on human colon cancer cells.
    Cancer research, 1992, Mar-01, Volume: 52, Issue:5

    Topics: Bombesin; Calcium; Carbachol; Colonic Neoplasms; Humans; Inositol 1,4,5-Trisphosphate; N-Methylscopolamine; Receptors, Bombesin; Receptors, Gastrointestinal Hormone; Receptors, Neurotransmitter; Scopolamine Derivatives; Tumor Cells, Cultured; Vasoactive Intestinal Peptide

1992
Agonist-induced desensitization of cholinergically stimulated phosphoinositide breakdown is independent of endogenously activated protein kinase C in HT-29 human colon carcinoma cells.
    The Biochemical journal, 1990, Jul-01, Volume: 269, Issue:1

    Topics: Carbachol; Colonic Neoplasms; Enzyme Activation; GTP-Binding Proteins; Humans; Hydrolysis; Inositol; Ionomycin; N-Methylscopolamine; Phosphatidylinositols; Phosphorylation; Protein Kinase C; Receptors, Muscarinic; Scopolamine Derivatives; Sodium Fluoride; Tetradecanoylphorbol Acetate; Tumor Cells, Cultured; Type C Phospholipases

1990