n-methylaspartate has been researched along with Motor Neuron Disease in 6 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (16.67) | 18.2507 |
2000's | 5 (83.33) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Brunet, N; Calderó, J; Esquerda, JE; Tarabal, O | 1 |
Calderó, J; Casanovas, A; Casas, C; Ciutat, D; Esquerda, JE; Lladó, J; Tarabal, O | 1 |
Anderton, BH; Leigh, PN; Meldrum, BS; Nunn, PB; Willis, CL | 1 |
Akaike, A; Honda, K; Inoue, R; Kihara, T; Nakamizo, T; Sawada, H; Shimohama, S; Urushitani, M | 1 |
Andreassen, OA; Beal, MF; Bogdanov, MB; Dedeoglu, A; Ferrante, KL; Jenkins, BG; Kaddurah-Daouk, R | 1 |
Calderó, J; Esquerda, JE; Lladó, J; Oppenheim, RW; Tarabal, O | 1 |
6 other study(ies) available for n-methylaspartate and Motor Neuron Disease
Article | Year |
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Excitotoxic motoneuron degeneration induced by glutamate receptor agonists and mitochondrial toxins in organotypic cultures of chick embryo spinal cord.
Topics: Animals; Calcium Signaling; Chick Embryo; Disease Models, Animal; Dose-Response Relationship, Drug; Excitatory Amino Acid Agonists; Glutamic Acid; Kainic Acid; Malonates; Mitochondria; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Nerve Degeneration; Neuroprotective Agents; Neurotoxins; Nitro Compounds; Organ Culture Techniques; Propionates; Riluzole; Spinal Cord | 2009 |
Excitotoxic motoneuron disease in chick embryo evolves with autophagic neurodegeneration and deregulation of neuromuscular innervation.
Topics: Age Factors; Animals; Autophagy; Calcitonin Gene-Related Peptide; Calcium; Chick Embryo; Curare; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Interactions; Excitatory Amino Acid Agonists; Gene Expression Regulation, Developmental; Microscopy, Electron, Transmission; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Nerve Degeneration; Neuromuscular Junction; Neuromuscular Nondepolarizing Agents; Receptors, Nicotinic; Spinal Cord; Tubulin | 2007 |
Neuroprotective effect of free radical scavengers on beta-N-oxalylamino-L-alanine (BOAA)-induced neuronal damage in rat hippocampus.
Topics: alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Dose-Response Relationship, Drug; Free Radical Scavengers; Hippocampus; Kainic Acid; Male; Motor Neuron Disease; N-Methylaspartate; Neurons; Rats; Rats, Wistar | 1994 |
N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx.
Topics: 2,4-Dinitrophenol; 6-Cyano-7-nitroquinoxaline-2,3-dione; alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Calcium; Calcium Signaling; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone; Cells, Cultured; Cyclosporine; Dibucaine; Dizocilpine Maleate; Enzyme Inhibitors; Excitatory Amino Acid Agonists; Excitatory Amino Acid Antagonists; Fluoresceins; Fluorescent Dyes; Glutamic Acid; Heterocyclic Compounds, 3-Ring; Imidazoles; Kainic Acid; Mitochondria; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Nerve Tissue Proteins; Neurons; Neurotoxins; Nitric Oxide Synthase; Nitric Oxide Synthase Type I; Oxidative Stress; Rats; Rats, Wistar; Reactive Oxygen Species; Receptors, AMPA; Receptors, Kainic Acid; Receptors, N-Methyl-D-Aspartate; Rhodamines; Spinal Cord; Superoxide Dismutase; Uncoupling Agents | 2001 |
Increases in cortical glutamate concentrations in transgenic amyotrophic lateral sclerosis mice are attenuated by creatine supplementation.
Topics: Amino Acid Transport System X-AG; Animals; ATP-Binding Cassette Transporters; Biological Transport; Body Weight; Brain Chemistry; Creatine; Dicarboxylic Acids; Disease Models, Animal; Excitatory Amino Acid Agonists; Glutamic Acid; Glutamine; Magnetic Resonance Spectroscopy; Male; Mice; Mice, Transgenic; Microdialysis; Motor Activity; Motor Neuron Disease; N-Methylaspartate; Neurotransmitter Uptake Inhibitors; Oxidative Stress; Psychomotor Performance; Pyrrolidines; Superoxide Dismutase | 2001 |
Long-lasting aberrant tubulovesicular membrane inclusions accumulate in developing motoneurons after a sublethal excitotoxic insult: a possible model for neuronal pathology in neurodegenerative disease.
Topics: Animals; Brefeldin A; Cell Compartmentation; Chick Embryo; Disease Models, Animal; Endoplasmic Reticulum; Endosomes; Golgi Apparatus; Inclusion Bodies; Intracellular Membranes; Lysosomes; Membrane Proteins; Microscopy, Electron; Microtubules; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Neuromuscular Junction; Presenilin-1; Protein Synthesis Inhibitors; Spinal Cord; trans-Golgi Network; Vacuoles | 2001 |