Page last updated: 2024-08-22

n-methylaspartate and Motor Neuron Disease

n-methylaspartate has been researched along with Motor Neuron Disease in 6 studies

Research

Studies (6)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (16.67)18.2507
2000's5 (83.33)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Brunet, N; Calderó, J; Esquerda, JE; Tarabal, O1
Calderó, J; Casanovas, A; Casas, C; Ciutat, D; Esquerda, JE; Lladó, J; Tarabal, O1
Anderton, BH; Leigh, PN; Meldrum, BS; Nunn, PB; Willis, CL1
Akaike, A; Honda, K; Inoue, R; Kihara, T; Nakamizo, T; Sawada, H; Shimohama, S; Urushitani, M1
Andreassen, OA; Beal, MF; Bogdanov, MB; Dedeoglu, A; Ferrante, KL; Jenkins, BG; Kaddurah-Daouk, R1
Calderó, J; Esquerda, JE; Lladó, J; Oppenheim, RW; Tarabal, O1

Other Studies

6 other study(ies) available for n-methylaspartate and Motor Neuron Disease

ArticleYear
Excitotoxic motoneuron degeneration induced by glutamate receptor agonists and mitochondrial toxins in organotypic cultures of chick embryo spinal cord.
    The Journal of comparative neurology, 2009, Oct-01, Volume: 516, Issue:4

    Topics: Animals; Calcium Signaling; Chick Embryo; Disease Models, Animal; Dose-Response Relationship, Drug; Excitatory Amino Acid Agonists; Glutamic Acid; Kainic Acid; Malonates; Mitochondria; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Nerve Degeneration; Neuroprotective Agents; Neurotoxins; Nitro Compounds; Organ Culture Techniques; Propionates; Riluzole; Spinal Cord

2009
Excitotoxic motoneuron disease in chick embryo evolves with autophagic neurodegeneration and deregulation of neuromuscular innervation.
    Journal of neuroscience research, 2007, Volume: 85, Issue:12

    Topics: Age Factors; Animals; Autophagy; Calcitonin Gene-Related Peptide; Calcium; Chick Embryo; Curare; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Interactions; Excitatory Amino Acid Agonists; Gene Expression Regulation, Developmental; Microscopy, Electron, Transmission; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Nerve Degeneration; Neuromuscular Junction; Neuromuscular Nondepolarizing Agents; Receptors, Nicotinic; Spinal Cord; Tubulin

2007
Neuroprotective effect of free radical scavengers on beta-N-oxalylamino-L-alanine (BOAA)-induced neuronal damage in rat hippocampus.
    Neuroscience letters, 1994, Dec-05, Volume: 182, Issue:2

    Topics: alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Dose-Response Relationship, Drug; Free Radical Scavengers; Hippocampus; Kainic Acid; Male; Motor Neuron Disease; N-Methylaspartate; Neurons; Rats; Rats, Wistar

1994
N-methyl-D-aspartate receptor-mediated mitochondrial Ca(2+) overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca(2+) influx.
    Journal of neuroscience research, 2001, Mar-01, Volume: 63, Issue:5

    Topics: 2,4-Dinitrophenol; 6-Cyano-7-nitroquinoxaline-2,3-dione; alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Calcium; Calcium Signaling; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone; Cells, Cultured; Cyclosporine; Dibucaine; Dizocilpine Maleate; Enzyme Inhibitors; Excitatory Amino Acid Agonists; Excitatory Amino Acid Antagonists; Fluoresceins; Fluorescent Dyes; Glutamic Acid; Heterocyclic Compounds, 3-Ring; Imidazoles; Kainic Acid; Mitochondria; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Nerve Tissue Proteins; Neurons; Neurotoxins; Nitric Oxide Synthase; Nitric Oxide Synthase Type I; Oxidative Stress; Rats; Rats, Wistar; Reactive Oxygen Species; Receptors, AMPA; Receptors, Kainic Acid; Receptors, N-Methyl-D-Aspartate; Rhodamines; Spinal Cord; Superoxide Dismutase; Uncoupling Agents

2001
Increases in cortical glutamate concentrations in transgenic amyotrophic lateral sclerosis mice are attenuated by creatine supplementation.
    Journal of neurochemistry, 2001, Volume: 77, Issue:2

    Topics: Amino Acid Transport System X-AG; Animals; ATP-Binding Cassette Transporters; Biological Transport; Body Weight; Brain Chemistry; Creatine; Dicarboxylic Acids; Disease Models, Animal; Excitatory Amino Acid Agonists; Glutamic Acid; Glutamine; Magnetic Resonance Spectroscopy; Male; Mice; Mice, Transgenic; Microdialysis; Motor Activity; Motor Neuron Disease; N-Methylaspartate; Neurotransmitter Uptake Inhibitors; Oxidative Stress; Psychomotor Performance; Pyrrolidines; Superoxide Dismutase

2001
Long-lasting aberrant tubulovesicular membrane inclusions accumulate in developing motoneurons after a sublethal excitotoxic insult: a possible model for neuronal pathology in neurodegenerative disease.
    The Journal of neuroscience : the official journal of the Society for Neuroscience, 2001, Oct-15, Volume: 21, Issue:20

    Topics: Animals; Brefeldin A; Cell Compartmentation; Chick Embryo; Disease Models, Animal; Endoplasmic Reticulum; Endosomes; Golgi Apparatus; Inclusion Bodies; Intracellular Membranes; Lysosomes; Membrane Proteins; Microscopy, Electron; Microtubules; Motor Neuron Disease; Motor Neurons; N-Methylaspartate; Neuromuscular Junction; Presenilin-1; Protein Synthesis Inhibitors; Spinal Cord; trans-Golgi Network; Vacuoles

2001