n-methyladenosine has been researched along with Hypertrophy, Left Ventricular in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 2 (66.67) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, Y; Cui, J; Cui, Y; Li, M; Tang, X; Wang, P; Wang, Y; Zhang, T | 1 |
| Chen, XZ; Dong, YH; Gao, XQ; Ju, J; Li, PF; Li, XM; Liu, CY; Liu, F; Ponnusamy, M; Qian, LL; Shan, C; Shan, PP; Wang, K; Wang, M; Wang, T; Wang, Y; Wang, YH; Yu, T; Zhai, M; Zhang, J; Zhang, YH; Zhao, XM; Zhou, LY | 1 |
| Accornero, F; Chen, J; Dorn, LE; Hanna, JH; Hund, TJ; Lasman, L; Medvedovic, M; van Berlo, JH; Xu, X | 1 |
3 other study(ies) available for n-methyladenosine and Hypertrophy, Left Ventricular
| Article | Year |
|---|---|
Cinnamic acid mitigates left ventricular hypertrophy and heart failure in part through modulating FTO-dependent N
Topics: Alpha-Ketoglutarate-Dependent Dioxygenase FTO; Animals; Disease Models, Animal; Heart Failure; Hypertrophy, Left Ventricular; Mice; Mice, Inbred C57BL; Myocytes, Cardiac; Phenylephrine; RNA | 2023 |
The piRNA CHAPIR regulates cardiac hypertrophy by controlling METTL3-dependent N
Topics: Adenosine; Animals; Cells, Cultured; Disease Models, Animal; Gene Expression Regulation, Enzymologic; Glycogen Synthase Kinase 3 beta; Heart Ventricles; Hypertrophy, Left Ventricular; Male; Methylation; Methyltransferases; Mice, Inbred C57BL; Mice, Knockout; Myocytes, Cardiac; NFATC Transcription Factors; Poly(ADP-ribose) Polymerases; Proto-Oncogene Proteins; RNA Stability; RNA, Messenger; RNA, Small Interfering; Signal Transduction; Ventricular Function, Left; Ventricular Remodeling | 2020 |
The N
Topics: Adenosine; Animals; Cells, Cultured; Disease Models, Animal; Female; Hypertrophy, Left Ventricular; Male; Methyltransferases; Mice, Inbred C57BL; Mice, Knockout; Myocytes, Cardiac; Rats; RNA Processing, Post-Transcriptional; RNA, Messenger; Signal Transduction; Ventricular Function, Left; Ventricular Remodeling | 2019 |