Page last updated: 2024-10-22

n-acetyltryptophan and Dyskinesia, Drug-Induced

n-acetyltryptophan has been researched along with Dyskinesia, Drug-Induced in 1 studies

N-acetyltryptophan : An N-acetylamino acid that is the N-acetyl derivative of tryptophan.

Dyskinesia, Drug-Induced: Abnormal movements, including HYPERKINESIS; HYPOKINESIA; TREMOR; and DYSTONIA, associated with the use of certain medications or drugs. Muscles of the face, trunk, neck, and extremities are most commonly affected. Tardive dyskinesia refers to abnormal hyperkinetic movements of the muscles of the face, tongue, and neck associated with the use of neuroleptic agents (see ANTIPSYCHOTIC AGENTS). (Adams et al., Principles of Neurology, 6th ed, p1199)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Thornton, E1
Hassall, MM1
Corrigan, F1
Vink, R1

Other Studies

1 other study available for n-acetyltryptophan and Dyskinesia, Drug-Induced

ArticleYear
The NK1 receptor antagonist N-acetyl-L-tryptophan reduces dyskinesia in a hemi-parkinsonian rodent model.
    Parkinsonism & related disorders, 2014, Volume: 20, Issue:5

    Topics: Adrenergic Agents; Analysis of Variance; Animals; Antiparkinson Agents; Dose-Response Relationship,

2014