n-acetyltryptophan has been researched along with Disease Models, Animal in 4 studies
N-acetyltryptophan : An N-acetylamino acid that is the N-acetyl derivative of tryptophan.
Disease Models, Animal: Naturally-occurring or experimentally-induced animal diseases with pathological processes analogous to human diseases.
Excerpt | Relevance | Reference |
---|---|---|
"The neuropeptide substance P (SP) is a well-known mediator of neurogenic inflammation following a variety of CNS disorders." | 3.80 | NK1 receptor blockade is ineffective in improving outcome following a balloon compression model of spinal cord injury. ( Leonard, AV; Thornton, E; Vink, R, 2014) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 1 (25.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Fernandes, J | 1 |
Mudgal, J | 1 |
Rao, CM | 1 |
Arora, D | 1 |
Basu Mallik, S | 1 |
Pai, KSR | 1 |
Nampoothiri, M | 1 |
Leonard, AV | 1 |
Thornton, E | 2 |
Vink, R | 3 |
Donkin, JJ | 1 |
Nimmo, AJ | 1 |
Cernak, I | 1 |
Blumbergs, PC | 1 |
Turner, RJ | 1 |
Helps, SC | 1 |
4 other studies available for n-acetyltryptophan and Disease Models, Animal
Article | Year |
---|---|
N-acetyl-L-tryptophan, a substance-P receptor antagonist attenuates aluminum-induced spatial memory deficit in rats.
Topics: Acetylcholinesterase; Aluminum Chloride; Aluminum Compounds; Animals; Apoptosis; Behavior, Animal; B | 2018 |
NK1 receptor blockade is ineffective in improving outcome following a balloon compression model of spinal cord injury.
Topics: Animals; Blood-Brain Barrier; Disease Models, Animal; Inflammation; Neurokinin-1 Receptor Antagonist | 2014 |
Substance P is associated with the development of brain edema and functional deficits after traumatic brain injury.
Topics: Animals; Brain Edema; Brain Injuries; Capillary Permeability; Disease Models, Animal; Enzyme-Linked | 2009 |
A substance P antagonist improves outcome when administered 4 h after onset of ischaemic stroke.
Topics: Animals; Blood-Brain Barrier; Brain Edema; Brain Ischemia; Disease Models, Animal; Drug Administrati | 2011 |