n-acetylserotonin has been researched along with Encephalopathy, Traumatic in 3 studies
N-acetylserotonin : An N-acylserotonin resulting from the formal condensation of the primary amino group of serotonin with the carboxy group of acetic acid.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (33.33) | 24.3611 |
2020's | 2 (66.67) | 2.80 |
Authors | Studies |
---|---|
Cheng, Y | 2 |
Gao, Y | 2 |
Li, J | 2 |
Rui, T | 3 |
Li, Q | 4 |
Chen, H | 2 |
Jia, B | 2 |
Song, Y | 2 |
Gu, Z | 2 |
Wang, T | 3 |
Gao, C | 2 |
Wang, Y | 2 |
Wang, Z | 3 |
Wang, F | 2 |
Tao, L | 3 |
Luo, C | 3 |
Wang, H | 1 |
Zhang, M | 1 |
Wang, P | 1 |
Huang, C | 1 |
Chen, B | 1 |
Liu, J | 1 |
Zhao, M | 1 |
Zhao, J | 1 |
3 other studies available for n-acetylserotonin and Encephalopathy, Traumatic
Article | Year |
---|---|
TrkB agonist N-acetyl serotonin promotes functional recovery after traumatic brain injury by suppressing ferroptosis via the PI3K/Akt/Nrf2/Ferritin H pathway.
Topics: Animals; Brain Injuries, Traumatic; Ferritins; Iron; Mice; NF-E2-Related Factor 2; Phosphatidylinosi | 2023 |
TrkB agonist N-acetyl serotonin promotes functional recovery after traumatic brain injury by suppressing ferroptosis via the PI3K/Akt/Nrf2/Ferritin H pathway.
Topics: Animals; Brain Injuries, Traumatic; Ferritins; Iron; Mice; NF-E2-Related Factor 2; Phosphatidylinosi | 2023 |
TrkB agonist N-acetyl serotonin promotes functional recovery after traumatic brain injury by suppressing ferroptosis via the PI3K/Akt/Nrf2/Ferritin H pathway.
Topics: Animals; Brain Injuries, Traumatic; Ferritins; Iron; Mice; NF-E2-Related Factor 2; Phosphatidylinosi | 2023 |
TrkB agonist N-acetyl serotonin promotes functional recovery after traumatic brain injury by suppressing ferroptosis via the PI3K/Akt/Nrf2/Ferritin H pathway.
Topics: Animals; Brain Injuries, Traumatic; Ferritins; Iron; Mice; NF-E2-Related Factor 2; Phosphatidylinosi | 2023 |
A TrkB receptor agonist N-acetyl serotonin provides cerebral protection after traumatic brain injury by mitigating apoptotic activation and autophagic dysfunction.
Topics: Animals; Apoptosis; Autophagy; Brain Injuries, Traumatic; Male; Membrane Glycoproteins; Mice; Mice, | 2020 |
N-Acetyl Serotonin Protects Neural Progenitor Cells Against Oxidative Stress-Induced Apoptosis and Improves Neurogenesis in Adult Mouse Hippocampus Following Traumatic Brain Injury.
Topics: Animals; Apoptosis; Brain Injuries, Traumatic; Cells, Cultured; Cyclic AMP; Cyclic AMP Response Elem | 2019 |