n-acetylneuraminic acid has been researched along with Glomerulonephritis, Membranoproliferative in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (50.00) | 18.2507 |
2000's | 1 (50.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Isome, M; Kawasaki, Y; Nozawa, R; Nozawa, Y; Sakai, N; Suzuki, H; Suzuki, J; Tannji, M | 1 |
Endo, M; Fujita, T; Hidaka, M; Ohi, H; Ohsawa, I; Satomura, A | 1 |
2 other study(ies) available for n-acetylneuraminic acid and Glomerulonephritis, Membranoproliferative
Article | Year |
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FB21, a monoclonal antibody that reacts with a sialic-acid-dependent carbohydrate epitope, is a marker for glomerular endothelial cell injury.
Topics: Adult; Age Factors; Antibodies, Monoclonal; Antibody Specificity; Antigens, Surface; Biomarkers; Child; Child, Preschool; E-Selectin; Endothelial Cells; Endothelium, Vascular; Epitopes; Female; Gestational Age; Glomerulonephritis; Glomerulonephritis, IGA; Glomerulonephritis, Membranoproliferative; Glomerulosclerosis, Focal Segmental; Hemolytic-Uremic Syndrome; Humans; IgA Vasculitis; Immunoenzyme Techniques; Immunohistochemistry; Kidney Glomerulus; Male; Microscopy, Immunoelectron; Middle Aged; N-Acetylneuraminic Acid; Streptococcal Infections | 2004 |
Inhibitory effect of free sialic acid on complement activation and its significance in hypocomplementemic glomerulonephritis.
Topics: Animals; Blood Proteins; Chronic Disease; Complement Activation; Complement C3; Complement System Proteins; Glomerulonephritis, Membranoproliferative; Hemolysis; Humans; N-Acetylneuraminic Acid; Protein Binding; Rabbits | 1999 |