Compounds > n-acetylneuraminic acid

n-acetylneuraminic acid and Gasser Syndrome

n-acetylneuraminic acid has been researched along with Gasser Syndrome in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (20.00)18.2507
2000's2 (40.00)29.6817
2010's2 (40.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Blaum, BS; Hannan, JP; Herbert, AP; Kavanagh, D; Stehle, T; Uhrín, D1
Conway, EM1
Isome, M; Kawasaki, Y; Nozawa, R; Nozawa, Y; Sakai, N; Suzuki, H; Suzuki, J; Tannji, M1
Kawasaki, Y1
Hellwege, HH; Poschmann, A; Seitz, RC1

Reviews

1 review(s) available for n-acetylneuraminic acid and Gasser Syndrome

ArticleYear
The role of FB21 as a marker of glomerular endothelial cell injury.
    Fukushima journal of medical science, 2006, Volume: 52, Issue:2

    Topics: Animals; Antibodies, Monoclonal; Antigens, Surface; Biomarkers; Endothelial Cells; Glomerulonephritis; Hemolytic-Uremic Syndrome; Humans; Kidney Glomerulus; N-Acetylneuraminic Acid; Staining and Labeling

2006

Other Studies

4 other study(ies) available for n-acetylneuraminic acid and Gasser Syndrome

ArticleYear
Structural basis for sialic acid-mediated self-recognition by complement factor H.
    Nature chemical biology, 2015, Volume: 11, Issue:1

    Topics: Animals; Binding Sites; Carbohydrate Sequence; Complement C3b; Complement Factor H; Complement Pathway, Alternative; Conserved Sequence; Hemolysis; Hemolytic-Uremic Syndrome; Humans; Mice; Models, Molecular; Molecular Sequence Data; N-Acetylneuraminic Acid; Polysaccharides; Sheep

2015
Sweeteners for factor H.
    Blood, 2016, 06-02, Volume: 127, Issue:22

    Topics: Complement Factor H; Hemolytic-Uremic Syndrome; Humans; N-Acetylneuraminic Acid

2016
FB21, a monoclonal antibody that reacts with a sialic-acid-dependent carbohydrate epitope, is a marker for glomerular endothelial cell injury.
    American journal of kidney diseases : the official journal of the National Kidney Foundation, 2004, Volume: 44, Issue:2

    Topics: Adult; Age Factors; Antibodies, Monoclonal; Antibody Specificity; Antigens, Surface; Biomarkers; Child; Child, Preschool; E-Selectin; Endothelial Cells; Endothelium, Vascular; Epitopes; Female; Gestational Age; Glomerulonephritis; Glomerulonephritis, IGA; Glomerulonephritis, Membranoproliferative; Glomerulosclerosis, Focal Segmental; Hemolytic-Uremic Syndrome; Humans; IgA Vasculitis; Immunoenzyme Techniques; Immunohistochemistry; Kidney Glomerulus; Male; Microscopy, Immunoelectron; Middle Aged; N-Acetylneuraminic Acid; Streptococcal Infections

2004
Monoclonal antibodies for the detection of desialylation of erythrocyte membranes during haemolytic disease and haemolytic uraemic syndrome caused by the in vivo action of microbial neuraminidase.
    Glycoconjugate journal, 1997, Volume: 14, Issue:6

    Topics: Anemia, Hemolytic; Animals; Antibodies, Monoclonal; Binding Sites, Antibody; Carbohydrates; Erythrocyte Membrane; Glycopeptides; Haptens; Hemolytic-Uremic Syndrome; Humans; Mice; Mice, Inbred BALB C; N-Acetylneuraminic Acid; Neuraminidase; Sensitivity and Specificity; Vibrio cholerae

1997