n-acetylglucosaminylasparagine has been researched along with Disease Models, Animal in 6 studies
*Disease Models, Animal: Naturally-occurring or experimentally-induced animal diseases with pathological processes analogous to human diseases. [MeSH]
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 4 (66.67) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (16.67) | 24.3611 |
2020's | 1 (16.67) | 2.80 |
Authors | Studies |
---|---|
Asahina, M; Fujinawa, R; Hirayama, H; Kajii, Y; Suzuki, T; Tozawa, R | 1 |
Dunder, U; Kelo, E; Mononen, I; Valtonen, P | 1 |
Gonzalez-Gomez, I; Groffen, J; Heisterkamp, N; Kaartinen, V; Mononen, I; Noronkoski, T; Voncken, JW | 1 |
Autti, T; Ginns, EI; Ikonen, S; Jalanko, A; Joensuu, R; LaMarca, ME; Manninen, T; McKinney, CE; Peltonen, L; Rapola, J; Riekkinen, P; Sipilä, I; Tenhunen, K | 1 |
Gonzalez-Gomez, I; Groffen, J; Heisterkamp, N; Kaartinen, V; Mononen, I; Noronkoski, T | 1 |
Aronson, NN | 1 |
1 review(s) available for n-acetylglucosaminylasparagine and Disease Models, Animal
Article | Year |
---|---|
Aspartylglycosaminuria: biochemistry and molecular biology.
Topics: Acetylglucosamine; Amino Acid Sequence; Animals; Aspartylglucosylaminase; Base Sequence; Disease Models, Animal; DNA, Complementary; Evolution, Molecular; Finland; Gene Deletion; Gene Rearrangement; Glycoproteins; Humans; Lysosomal Storage Diseases; Lysosomes; Molecular Sequence Data; Mutation; Mutation, Missense | 1999 |
5 other study(ies) available for n-acetylglucosaminylasparagine and Disease Models, Animal
Article | Year |
---|---|
Reversibility of motor dysfunction in the rat model of NGLY1 deficiency.
Topics: Acetylglucosamine; Animals; Congenital Disorders of Glycosylation; Disease Models, Animal; Genetic Therapy; Genetic Vectors; Gliosis; Humans; Inflammation; Injections, Intraventricular; Motor Activity; Neurons; Peptide-N4-(N-acetyl-beta-glucosaminyl) Asparagine Amidase; Rats; Rats, Transgenic; Transgenes | 2021 |
Early initiation of enzyme replacement therapy improves metabolic correction in the brain tissue of aspartylglycosaminuria mice.
Topics: Acetylglucosamine; Age Factors; Animals; Animals, Newborn; Aspartylglucosaminuria; Aspartylglucosylaminase; Biomarkers; Brain; Disease Models, Animal; Dose-Response Relationship, Drug; Drug Administration Schedule; Enzyme Replacement Therapy; Humans; Injections, Intraperitoneal; Injections, Intravenous; Mice; Mice, Knockout; NIH 3T3 Cells; Recombinant Proteins; Time Factors; Transfection | 2010 |
A mouse model for the human lysosomal disease aspartylglycosaminuria.
Topics: Acetylglucosamine; Age Factors; Animals; Aspartylglucosaminuria; Aspartylglucosylaminase; Central Nervous System; Disease Models, Animal; Female; Humans; Lysosomal Storage Diseases; Lysosomes; Male; Mice; Mice, Knockout; Psychomotor Performance; RNA, Messenger; Stem Cells; Urinary Bladder | 1996 |
Mice with an aspartylglucosaminuria mutation similar to humans replicate the pathophysiology in patients.
Topics: Acetylglucosamine; Animals; Aspartylglucosaminuria; Aspartylglucosylaminase; Brain; Disease Models, Animal; Disease Progression; Gene Targeting; Genes, Recessive; Humans; Intellectual Disability; Liver; Lysosomal Storage Diseases; Magnetic Resonance Imaging; Maze Learning; Mice; Mice, Knockout; Microscopy, Electron; Phenotype | 1998 |
Phenotypic characterization of mice with targeted disruption of glycosylasparaginase gene: a mouse model for aspartylglycosaminuria.
Topics: Acetylglucosamine; Animals; Aspartylglucosylaminase; Disease Models, Animal; Gene Targeting; Lysosomal Storage Diseases; Mice; Phenotype | 1998 |