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n-(4-aminobutyl)-5-chloro-2-naphthalenesulfonamide and Iron Overload

n-(4-aminobutyl)-5-chloro-2-naphthalenesulfonamide has been researched along with Iron Overload in 1 studies

N-(4-aminobutyl)-5-chloro-2-naphthalenesulfonamide: calmodulin antagonist; structure given in first source
N-(4-aminobutyl)-5-chloronaphthalene-2-sulfonamide : A sulfonamide that is 5-chloronaphthalene-2-sulfonamide in which one of the hydrogens of the nitrogen atom is substituted by a 4-aminobutyl group.

Iron Overload: An excessive accumulation of iron in the body due to a greater than normal absorption of iron from the gastrointestinal tract or from parenteral injection. This may arise from idiopathic hemochromatosis, excessive iron intake, chronic alcoholism, certain types of refractory anemia, or transfusional hemosiderosis. (From Churchill's Illustrated Medical Dictionary, 1989)

Research Excerpts

ExcerptRelevanceReference
" In this study, we investigated the mechanism underlying iron overload-induced mitochondrial fragmentation in HT-22 hippocampal neuron cells that were incubated with ferric ammonium citrate (FAC)."3.83Iron overload-induced calcium signals modulate mitochondrial fragmentation in HT-22 hippocampal neuron cells. ( Lee, DG; Lee, DS; Lee, HS; Lee, SR; Park, J, 2016)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Lee, DG1
Park, J1
Lee, HS1
Lee, SR1
Lee, DS1

Other Studies

1 other study available for n-(4-aminobutyl)-5-chloro-2-naphthalenesulfonamide and Iron Overload

ArticleYear
Iron overload-induced calcium signals modulate mitochondrial fragmentation in HT-22 hippocampal neuron cells.
    Toxicology, 2016, 07-15, Volume: 365

    Topics: Animals; Calcineurin; Calcium; Calcium Signaling; Calmodulin; Calpain; Cell Death; Cell Line; Cell S

2016