Compounds > n-(3-iodopropen-1-yl)-2-carbomethoxy-3-(4-chlorophenyl)tropane

n-(3-iodopropen-1-yl)-2-carbomethoxy-3-(4-chlorophenyl)tropane and Hyponatremia

n-(3-iodopropen-1-yl)-2-carbomethoxy-3-(4-chlorophenyl)tropane has been researched along with Hyponatremia* in 1 studies

Other Studies

1 other study(ies) available for n-(3-iodopropen-1-yl)-2-carbomethoxy-3-(4-chlorophenyl)tropane and Hyponatremia

Article	Year
Decreased striatal dopamine transporter binding in a patient with extrapontine myelinolysis. Movement disorders : official journal of the Movement Disorder Society, 2003, Volume: 18, Issue:3 We describe the case of a 61-year-old woman who developed extrapontine myelinolysis (EPM) with parkinsonism. Decreased striatal dopamine transporter binding assessed by [(123)I]N-(3-iodopropen-2-yl)-2beta-carbomethoxy-3beta-(4-chlorophenyl) tropane and single photon emission computed tomography ([(123)I]IPT) SPECT) were observed in the patient, suggesting that osmotic injury causes the demyelination of nigrostriatal dopaminergic neurons and that such injury may be involved in the pathogenesis of EPM with parkinsonism. Topics: Antiparkinson Agents; Dopamine Plasma Membrane Transport Proteins; Female; Humans; Hyponatremia; Isotonic Solutions; Levodopa; Membrane Glycoproteins; Membrane Transport Proteins; Middle Aged; Myelinolysis, Central Pontine; Nerve Tissue Proteins; Parkinsonian Disorders; Protein Binding; Radiopharmaceuticals; Tomography, Emission-Computed, Single-Photon; Treatment Outcome; Tropanes	2003

Article

Year

Decreased striatal dopamine transporter binding in a patient with extrapontine myelinolysis.

Movement disorders : official journal of the Movement Disorder Society, 2003, Volume: 18, Issue:3

We describe the case of a 61-year-old woman who developed extrapontine myelinolysis (EPM) with parkinsonism. Decreased striatal dopamine transporter binding assessed by [(123)I]N-(3-iodopropen-2-yl)-2beta-carbomethoxy-3beta-(4-chlorophenyl) tropane and single photon emission computed tomography ([(123)I]IPT) SPECT) were observed in the patient, suggesting that osmotic injury causes the demyelination of nigrostriatal dopaminergic neurons and that such injury may be involved in the pathogenesis of EPM with parkinsonism.

Topics: Antiparkinson Agents; Dopamine Plasma Membrane Transport Proteins; Female; Humans; Hyponatremia; Isotonic Solutions; Levodopa; Membrane Glycoproteins; Membrane Transport Proteins; Middle Aged; Myelinolysis, Central Pontine; Nerve Tissue Proteins; Parkinsonian Disorders; Protein Binding; Radiopharmaceuticals; Tomography, Emission-Computed, Single-Photon; Treatment Outcome; Tropanes

2003